Issue Archive
Table of Contents
BLOOD COMMENTARIES
So you know how to treat iron deficiency anemia
Clinical Trials & Observations
CLINICAL TRIALS AND OBSERVATIONS
Oral iron supplements increase hepcidin and decrease iron absorption from daily or twice-daily doses in iron-depleted young women
Clinical Trials & Observations
LYMPHOID NEOPLASIA
Prediction of high- and low-risk multiple myeloma based on gene expression and the International Staging System
Clinical Trials & Observations
MYELOID NEOPLASIA
Overexpression and knockout of miR-126 both promote leukemogenesis
PHAGOCYTES, GRANULOCYTES, AND MYELOPOIESIS
Identification and characterization of VEGF-A–responsive neutrophils expressing CD49d, VEGFR1, and CXCR4 in mice and humans
PLATELETS AND THROMBOPOIESIS
Paris-Trousseau thrombocytopenia is phenocopied by the autosomal recessive inheritance of a DNA-binding domain mutation in FLI1
Brief Report
RED CELLS, IRON, AND ERYTHROPOIESIS
THROMBOSIS AND HEMOSTASIS
Hemostatic efficacy, safety, and pharmacokinetics of a recombinant von Willebrand factor in severe von Willebrand disease
Clinical Trials & Observations
Mice expressing a mutant form of fibrinogen that cannot support fibrin formation exhibit compromised antimicrobial host defense
TRANSFUSION MEDICINE
No evidence of transmission of chronic lymphocytic leukemia through blood transfusion
Clinical Trials & Observations
Brief Report
TRANSPLANTATION
Outcome of patients with distinct molecular genotypes and cytogenetically normal AML after allogeneic transplantation
Clinical Trials & Observations
BLOOD WORK
ERRATA
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Cover Image
Cover Image
Extravascular fibrin polymer is deposited in a wound field following a liver puncture injury. The image is a scanning electron micrograph of a wound field 5 minutes after a 2 mm deep puncture to the right medial hepatic lobe of a wild-type mouse (original magnification ×10 000). Fibrin polymer, with associated entrapped red blood cells, is formed in the damaged zone. The contribution of soluble fibrinogen vs fibrin polymer to hemostasis, thrombosis, wound healing, and disease processes may now be determined utilizing FibAEK mice, which carry normal levels of a mutant fibrinogen that cannot be converted to fibrin polymer by thrombin. See the article by Prasad et al on page 2047.
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