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Journal Articles

Transcriptome sequencing reveals a profile that corresponds to genomic variants in Waldenström macroglobulinemia

Zachary R. Hunter, Lian Xu, Guang Yang, Nicholas Tsakmaklis, Josephine M. Vos, Xia Liu, Jie Chen, Robert J. Manning, Jiaji G. Chen, Philip Brodsky, Christopher J. Patterson, Joshua Gustine, Toni Dubeau, Jorge J. Castillo, Kenneth C. Anderson, Nikhil M. Munshi, Steven P. Treon

Blood (2016) 128 (6): 827–838.

https://doi.org/10.1182/blood-2016-03-708263

Published: 2016

Includes: Supplemental data

Journal Articles

The genomic landscape of Waldenström macroglobulinemia is characterized by highly recurring MYD88 and WHIM-like CXCR4 mutations, and small somatic deletions associated with B-cell lymphomagenesis

Zachary R. Hunter, Lian Xu, Guang Yang, Yangsheng Zhou, Xia Liu, Yang Cao, Robert J. Manning, Christina Tripsas, Christopher J. Patterson, Patricia Sheehy, Steven P. Treon

Blood (2014) 123 (11): 1637–1646.

https://doi.org/10.1182/blood-2013-09-525808

Published: 2014

Includes: Supplemental data

Journal Articles

A mutation in MYD88 (L265P) supports the survival of lymphoplasmacytic cells by activation of Bruton tyrosine kinase in Waldenström macroglobulinemia

Guang Yang, Yangsheng Zhou, Xia Liu, Lian Xu, Yang Cao, Robert J. Manning, Christopher J. Patterson, Sara J. Buhrlage, Nathanael Gray, Yu-Tzu Tai, Kenneth C. Anderson, Zachary R. Hunter, Steven P. Treon

Blood (2013) 122 (7): 1222–1232.

https://doi.org/10.1182/blood-2012-12-475111

Published: 2013

Journal Articles

MYD88 L265P in Waldenström macroglobulinemia, immunoglobulin M monoclonal gammopathy, and other B-cell lymphoproliferative disorders using conventional and quantitative allele-specific polymerase chain reaction

Blood (2013) 121 (11): 2051–2058.

https://doi.org/10.1182/blood-2012-09-454355

Published: 2013

Journal Articles

Endoplasmic reticulum stress is a target for therapy in Waldenstrom macroglobulinemia

Blood (2009) 113 (3): 626–634.

https://doi.org/10.1182/blood-2007-10-116848

Published: 2009

Journal Articles

CD27-CD70 interactions in the pathogenesis of Waldenström macroglobulinemia

Allen W. Ho, Evdoxia Hatjiharissi, Bryan T. Ciccarelli, Andrew R. Branagan, Zachary R. Hunter, Xavier Leleu, Olivier Tournilhac, Lian Xu, Kelly O'Connor, Robert J. Manning, Daniel Ditzel Santos, Mariana Chemaly, Christopher J. Patterson, Jacob D. Soumerai, Nikhil C. Munshi, Julie A. McEarchern, Che-Leung Law, Iqbal S. Grewal, Steven P. Treon

Blood (2008) 112 (12): 4683–4689.

https://doi.org/10.1182/blood-2007-04-084525

Published: 2008

Journal Articles

Biological Sequelae of TRAF2 Downregulation in Waldenstrom Macroglobulinemia Cells.

Xavier Leleu, MD, Lian Xu, Zachary R. Hunter, Sophia Adamia, PhD, Evdoxia Hatjiharissi, MD, Anne-Sophie Moreau, MD, Aldo Roccaro, MD, Antonio Sacco, MD, Brian Ciccarelli, Robert J. Manning, Christopher J. Patterson, Irene M. Ghobrial, MD, Steven P. Treon, MD

Blood (2007) 110 (11): 3526.

https://doi.org/10.1182/blood.V110.11.3526.3526

Published: 2007

Journal Articles

Increased natural killer cell expression of CD16, augmented binding and ADCC activity to rituximab among individuals expressing the FcγRIIIa-158 V/V and V/F polymorphism

Evdoxia Hatjiharissi, Lian Xu, Daniel Ditzel Santos, Zachary R. Hunter, Bryan T. Ciccarelli, Sigitas Verselis, Michael Modica, Yang Cao, Robert J. Manning, Xavier Leleu, Elizabeth A. Dimmock, Alexandros Kortsaris, Constantine Mitsiades, Kenneth C. Anderson, Edward A. Fox, Steven P. Treon

Blood (2007) 110 (7): 2561–2564.

https://doi.org/10.1182/blood-2007-01-070656

Published: 2007

Journal Articles

Bone Marrow Mast Cells Are Significantly Increased in Patients with Waldenstrom’s Macroglobulinemia, and Their Number Following Therapeutic Intervention Is Dependent on Extent of Response.

Daniel Ditzel Santos, MD, Mariana Z.A. Chemaly, MD, Olivier Tournilhac, MD, Kelly E. OConnor, Evdoxia Hatjiharissi, MD, Xavier Leleu, MD, Lian Xu, Andrew R. Branagan, Robert J. Manning, Christopher Patterson, Allen W. Ho, Zachary R. Hunter, James M. You, MD. Phd, Yu-Tzu Tai, Phd, Jeffery L. Kutok, Kenneth C. Anderson, Nikhil Munshi, Steven P. Treon

Blood (2005) 106 (11): 980.

https://doi.org/10.1182/blood.V106.11.980.980

Published: 2005

Journal Articles

The Unfolded Protein Response Is a Determinant of Disease Actvity in Waldenstrom’s Macroglobulinemia (WM).

Xavier Leleu, MD, Lian Xu, Daniel D. Santos, MD, Allen W. Ho, Zachary R. Hunter, Kelly O’Connor, Robert J. Manning, Olivier Tournilhac, MD, Mariana Chemaly, Andrew R. Branagan, Christopher J. Patterson, Kenneth C. Anderson, MD, Steven Treon, MD

Blood (2005) 106 (11): 996.

https://doi.org/10.1182/blood.V106.11.996.996

Published: 2005

Journal Articles

Abnormal Expression of the Plasma Cell Differentiation Factor X-Box Protein 1 (Xbp-1) in Waldenstrom’s Macroglobulinemia.

Lian Xu, Xavier Leleu, MD, Zachary R. Hunter, Daniel D. Santos, MD, Allen W. Ho, Evdoxia Hatjiharissi, MD, Kelly O’Connor, Robert J. Manning, Mariana Chemaly, Andrew R. Branagan, Christopher J. Patterson, Sigitas Verselis, Edward Fox, Kenneth C. Anderson, MD, Steven Treon, MD

Blood (2005) 106 (11): 1003.

https://doi.org/10.1182/blood.V106.11.1003.1003

Published: 2005

Journal Articles

High Levels of Soluble Immunoregulatory Receptors in Patients with WaldenströM’s Macroglobulinemia.

Zachary R. Hunter, Andrew R. Branagan, Daniel Ditzel Santos, Olivier Tournilhac, Evdoxia Hatjiharissi, Lian Xu, Robert J. Manning, Steven P. Treon

Blood (2004) 104 (11): 4881.

https://doi.org/10.1182/blood.V104.11.4881.4881

Published: 2004

NARROW

Transcriptome sequencing reveals a profile that corresponds to genomic variants in Waldenström macroglobulinemia

The genomic landscape of Waldenström macroglobulinemia is characterized by highly recurring MYD88 and WHIM-like CXCR4 mutations, and small somatic deletions associated with B-cell lymphomagenesis

A mutation in MYD88 (L265P) supports the survival of lymphoplasmacytic cells by activation of Bruton tyrosine kinase in Waldenström macroglobulinemia

MYD88 L265P in Waldenström macroglobulinemia, immunoglobulin M monoclonal gammopathy, and other B-cell lymphoproliferative disorders using conventional and quantitative allele-specific polymerase chain reaction

Endoplasmic reticulum stress is a target for therapy in Waldenstrom macroglobulinemia

CD27-CD70 interactions in the pathogenesis of Waldenström macroglobulinemia

Biological Sequelae of TRAF2 Downregulation in Waldenstrom Macroglobulinemia Cells.

Increased natural killer cell expression of CD16, augmented binding and ADCC activity to rituximab among individuals expressing the FcγRIIIa-158 V/V and V/F polymorphism

Bone Marrow Mast Cells Are Significantly Increased in Patients with Waldenstrom’s Macroglobulinemia, and Their Number Following Therapeutic Intervention Is Dependent on Extent of Response.

The Unfolded Protein Response Is a Determinant of Disease Actvity in Waldenstrom’s Macroglobulinemia (WM).

Abnormal Expression of the Plasma Cell Differentiation Factor X-Box Protein 1 (Xbp-1) in Waldenstrom’s Macroglobulinemia.

High Levels of Soluble Immunoregulatory Receptors in Patients with WaldenströM’s Macroglobulinemia.

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American Society of Hematology

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ASH Publications

American Society of Hematology

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