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Journal Articles

Genome-Wide Analysis by High-Resolution SNP Array Identifies Novel Genomic Alterations in Acute Promyelocytic Leukemia (APL).

Blood (2009) 114 (22): 167.

https://doi.org/10.1182/blood.V114.22.167.167

Published: 2009

Journal Articles

PAX5 Wild-Type without IKZF1 (Ikaros) Deletion Is Associated with Prolonged Disease-Free Survival and Low Rate of Cumulative Incidence of Relapse in Adult BCR-ABL1 -Positive Acute Lymphoblastic Leukemia (ALL): On Behalf of GIMEMA AL Working Party.

Blood (2009) 114 (22): 12.

https://doi.org/10.1182/blood.V114.22.12.12

Published: 2009

Journal Articles

The Oncogenic Kinase Bcr-Abl Directly Regulates Splicing of BclX through a Quaternary Complex Coordinated by Nck-Beta and Sam-68 Adapter Proteins.

Bracco Enrico, Valentina Rosso, Mussino Stefano, Francesca Arruga, Sonia Carturan, PhD, Francesca Messa, PhD, Cristina Panuzzo, PhD, Marco Lo Iacono, Paolo Nicoli, MD, Emanuela Messa, MD, Antonia Rotolo, Monica Pradotto, PhD, Alessandro Morotti, MD, Alessandra Balbo, Ilaria Iacobucci, PhD, Giovanni Martinelli, MD, Daniela Cilloni, MD, PhD, Giuseppe Saglio, MD

Blood (2009) 114 (22): 2171.

https://doi.org/10.1182/blood.V114.22.2171.2171

Published: 2009

Journal Articles

Genome-Wide Screening for Dominant Modifiers in Drosophila Identified New Cluster of Genes Involved in BCR-ABL Signalling and CML Progression.

Francesca Messa, PhD, Monica Pradotto, PhD, Francesca Arruga, PhD, Roberto Bernardoni, PhD, Enrico Bracco, PhD, Chiara Maffè, PhD, Emanuela Messa, MD, Sonia Carturan, PhD, Elisabetta Signorino, PhD, Elisabetta Greco, MD, Antonia Rotolo, MD, Ilaria Iacobucci, PhD, Giovanni Martinelli, MD, Giuseppe Saglio, MD, Daniela Cilloni, MD, PhD

Blood (2009) 114 (22): 2186.

https://doi.org/10.1182/blood.V114.22.2186.2186

Published: 2009

Journal Articles

IKZF1 (IKAROS) Deletions Are Independent On BCR-ABL1 Rearrangement and Are Associated with a Peculiar Gene Expression Signature and Poor Prognosis in Adult B-Progenitor Acute Lymphoblastic Leukemia (ALL) Patients.

Blood (2009) 114 (22): 912.

https://doi.org/10.1182/blood.V114.22.912.912

Published: 2009

Journal Articles

Identification of Rab5 as a Gene Involved in Chronic Myeloid Leukemia (CML) Progression.

Daniela Cilloni, MD, PhD, Monica Pradotto, PhD, Francesca Messa, PhD, Francesca Arruga, PhD, Enrico Bracco, PhD, Sonia Carturan, PhD, Chiara Maffè, PhD, Emanuela Messa, MD, Antonia Rotolo, MD, Elisabetta Greco, MD, Ilaria Iacobucci, PhD, Thea Kalebic, MD, Roberto Bernardoni, Fabrizio Pane, MD, Giovanni Martinelli, MD, Michele Baccarani, M.D., Giuseppe Saglio, MD

Blood (2009) 114 (22): 3470.

https://doi.org/10.1182/blood.V114.22.3470.3470

Published: 2009

Journal Articles

Whole Transcriptome Sequencing of a Philadelphia-Positive Acute Lymphoblastic Leukemia (ALL) with “Next Generation Sequencing” Technology Revealed Novel Point Mutations Associated with Disease-Progression.

Blood (2009) 114 (22): 3074.

https://doi.org/10.1182/blood.V114.22.3074.3074

Published: 2009

Journal Articles

Chronic Eosinophilic Leukaemia (CEL) with FIP1L1-PDGFRalpha Rearrangement (F/P): The Response to Imatinib (IM) Is Durable. A Report of 33 Patients with A Follow –up of 30 to 92 Months.

Michela Rondoni, Emanuela Ottaviani, Daniela Cilloni, MD, PhD, Pier Paolo Piccaluga, Stefania Paolini, Ilaria Iacobucci, PhD, Simona Soverini, PhD, Serena Merante, Francesca Messa, PhD, Francesco Buccisano, M.D., Mario Tiribelli, Fabrizio Pane, Giuseppe Saglio, Francesco Lauria, MD, Michele Baccarani, MD, Giovanni Martinelli, MD

Blood (2009) 114 (22): 3894.

https://doi.org/10.1182/blood.V114.22.3894.3894

Published: 2009

Journal Articles

Disabled Gene Is Involved in CML Progression and Its Expression Level at Diagnosis Can Predict Major Molecular Response (MMR) to Imatinib Therapy.

Daniela Cilloni, MD, PhD, Francesca Arruga, PhD, Francesca Messa, PhD, Monica Pradotto, PhD, Enrico Bracco, PhD, Sonia Carturan, PhD, Chiara Maffè, PhD, Emanuela Messa, MD, Antonia Rotolo, MD, Elisabetta Greco, MD, Ilaria Iacobucci, PhD, Roberto Bernardoni, Fabrizio Pane, MD, Thea Kalebic, MD, Giovanni Martinelli, Michele Baccarani, MD, Giuseppe Saglio, MD

Blood (2009) 114 (22): 3964.

https://doi.org/10.1182/blood.V114.22.3964.3964

Published: 2009

Journal Articles

Innovative Phase I Study of Concomitant and Consecutive Treatment with Dasatinib and MK-0457 in Refractory Ph+ CML and ALL Patients.

Giovanni Martinelli, MD, Cristina Papayannidis, Ilaria Iacobucci, Simona Soverini, PhD, Stefania Paolini, Sandra Santucci, Francesco De Rosa, Daniela Cilloni, MD, Francesca Messa, Fabrizio Pane, MD, Vittorio Meneghini, Panagiota Giannoulia, Emanuela Ottaviani, Nicoletta Testoni, Barbara Lama, Michele Baccarani

Blood (2009) 114 (22): 4277.

https://doi.org/10.1182/blood.V114.22.4277.4277

Published: 2009

Journal Articles

Foxo Transcription Factor Activity Is Retained in Quiescent Chronic Myeloid Leukaemia Stem Cells and Activated by Tyrosine Kinase Inhibitors to Mediate “induced-quiescence” in More Mature progenitors.

Daniela Cilloni, MD, PhD, Francesca Pellicano, PhD, Vignir G Helgason, PhD, Cristina Panuzzo, PhD, Francesca Messa, PhD, Brian J Huntly, MD, Elaine Allan, PhD, Francesca Arruga, PhD, Enrico Bracco, PhD, Giuseppe Saglio, MD, Tessa L Holyoake, MD

Blood (2009) 114 (22): 187.

https://doi.org/10.1182/blood.V114.22.187.187

Published: 2009

Journal Articles

Identification and molecular characterization of recurrent genomic deletions on 7p12 in the IKZF1 gene in a large cohort of BCR-ABL1 –positive acute lymphoblastic leukemia patients: on behalf of Gruppo Italiano Malattie Ematologiche dell'Adulto Acute Leukemia Working Party (GIMEMA AL WP)

Blood (2009) 114 (10): 2159–2167.

https://doi.org/10.1182/blood-2008-08-173963

Published: 2009

Includes: Supplemental data

Journal Articles

The Non Receptor Transmembrane Tyrosine Kinase STYK1 Represents a Novel Lymphocyte Marker, Whose Expression Is Deregulated in Leukemia, and a Potential Molecular Druggable Target

Blood (2008) 112 (11): 5334.

https://doi.org/10.1182/blood.V112.11.5334.5334

Published: 2008

Journal Articles

Deferasirox Is the Only Iron Chelator Acting as a Potent NF-KB Inhibitor in Myelodysplastic Syndromes

Emanuela Messa, MD, Ilaria Defilippi, PhD, Antonella Roetto, PhD, Francesca Messa, PhD, Francesca Arruga, PhD, Sonia Carturan, PhD, Valentina Rosso, PhD, Enrico Bracco, PhD, Daniela Cilloni, Giuseppe Saglio, MD

Blood (2008) 112 (11): 2671.

https://doi.org/10.1182/blood.V112.11.2671.2671

Published: 2008

Journal Articles

Different Isoforms of the B-Cell Mutator Activation-Induced Cytidine Deaminase (AID) Are Aberrantly Over-Expressed in BCR-ABL1 -Positive Acute Lymphoblastic Leukemia (ALL) Patients and Promote Genetic Instability.

Blood (2008) 112 (11): 1497.

https://doi.org/10.1182/blood.V112.11.1497.1497

Published: 2008

Journal Articles

Single Base Pair Mutation Detection by PNA Direct PCR Clamping: Application to Bcr-Abl T315I Mutation

Blood (2008) 112 (11): 4224.

https://doi.org/10.1182/blood.V112.11.4224.4224

Published: 2008

Journal Articles

Identification and Molecular Characterization of Two Recurrent Genomic Deletions (Type A and Type B) on 7p12 in IKZF1 Gene in a Large Cohort of BCR-ABL1 -Positive Acute Lymphoblastic Leukemia (ALL): on Behalf of the GIMEMA ALL Working Party

Blood (2008) 112 (11): 428.

https://doi.org/10.1182/blood.V112.11.428.428

Published: 2008

Journal Articles

Imatinib Induced Re-Activation of FoxO3 Transcription Factor in CML Is Responsible for the Induction of a Quiescent Status of CD34 Leukaemic Progenitor Cells.

Daniela Cilloni, MD, PhD, Cristina Panuzzo, PhD, Francesca Messa, PhD, Francesca Arruga, PhD, Enrico Bracco, PhD, Roberto Bernardoni, PhD, Sonia Carturan, PhD, Marisa Pautasso, MD, Emanuela Messa, MD, Alessandro Morotti, MD, Monica Pradotto, PhD, Ilaria Iacobucci, Thea Kalebic, MD, Giovanni Martinelli, MD, Giuseppe Saglio, MD

Blood (2008) 112 (11): 1090.

https://doi.org/10.1182/blood.V112.11.1090.1090

Published: 2008

Journal Articles

Identification of Genes Sustaining Bcr-Abl Oncogenic Signalling and CML Progression through a Genetic Tool Based on Human Bcr-Abl Transgenic Drosophila Melanogaster.

Daniela Cilloni, Francesca Messa, PhD, Roberto Bernardoni, PhD, Francesca Arruga, PhD, Monica Pradotto, PhD, Enrico Bracco, PhD, Cristina Panuzzo, PhD, Chiara Maffè, PhD, Emanuela Messa, MD, Ilaria Iacobucci, Thea Kalebic, MD, Giovanni Martinelli, MD, Michele Baccarani, MD, Giuseppe Saglio, MD

Blood (2008) 112 (11): 1091.

https://doi.org/10.1182/blood.V112.11.1091.1091

Published: 2008

Journal Articles

EphA3 Kinase Is Constitutively Activated in Chronic Myeloid Leukaemia during Progression to Accelerated and Blast Crisis and It Could Represent a New Molecular Target.

Daniela Cilloni, MD, PhD, Francesca Arruga, PhD, Sonia Carturan, PhD, Francesca Messa, PhD, Monica Pradotto, PhD, Ilaria Defilippi, PhD, Marisa Pautasso, MD, Ilaria Iacobucci, PhD, Giovanni Martinelli, MD, Enrico Bracco, PhD, Thea Kalebic, MD, Martin Lackmann, PhD, Giuseppe Saglio, MD

Blood (2008) 112 (11): 1092.

https://doi.org/10.1182/blood.V112.11.1092.1092

Published: 2008

NARROW

Genome-Wide Analysis by High-Resolution SNP Array Identifies Novel Genomic Alterations in Acute Promyelocytic Leukemia (APL).

PAX5 Wild-Type without IKZF1 (Ikaros) Deletion Is Associated with Prolonged Disease-Free Survival and Low Rate of Cumulative Incidence of Relapse in Adult BCR-ABL1 -Positive Acute Lymphoblastic Leukemia (ALL): On Behalf of GIMEMA AL Working Party.

The Oncogenic Kinase Bcr-Abl Directly Regulates Splicing of BclX through a Quaternary Complex Coordinated by Nck-Beta and Sam-68 Adapter Proteins.

Genome-Wide Screening for Dominant Modifiers in Drosophila Identified New Cluster of Genes Involved in BCR-ABL Signalling and CML Progression.

IKZF1 (IKAROS) Deletions Are Independent On BCR-ABL1 Rearrangement and Are Associated with a Peculiar Gene Expression Signature and Poor Prognosis in Adult B-Progenitor Acute Lymphoblastic Leukemia (ALL) Patients.

Identification of Rab5 as a Gene Involved in Chronic Myeloid Leukemia (CML) Progression.

Whole Transcriptome Sequencing of a Philadelphia-Positive Acute Lymphoblastic Leukemia (ALL) with “Next Generation Sequencing” Technology Revealed Novel Point Mutations Associated with Disease-Progression.

Chronic Eosinophilic Leukaemia (CEL) with FIP1L1-PDGFRalpha Rearrangement (F/P): The Response to Imatinib (IM) Is Durable. A Report of 33 Patients with A Follow –up of 30 to 92 Months.

Disabled Gene Is Involved in CML Progression and Its Expression Level at Diagnosis Can Predict Major Molecular Response (MMR) to Imatinib Therapy.

Innovative Phase I Study of Concomitant and Consecutive Treatment with Dasatinib and MK-0457 in Refractory Ph+ CML and ALL Patients.

Foxo Transcription Factor Activity Is Retained in Quiescent Chronic Myeloid Leukaemia Stem Cells and Activated by Tyrosine Kinase Inhibitors to Mediate “induced-quiescence” in More Mature progenitors.

Identification and molecular characterization of recurrent genomic deletions on 7p12 in the IKZF1 gene in a large cohort of BCR-ABL1 –positive acute lymphoblastic leukemia patients: on behalf of Gruppo Italiano Malattie Ematologiche dell'Adulto Acute Leukemia Working Party (GIMEMA AL WP)

The Non Receptor Transmembrane Tyrosine Kinase STYK1 Represents a Novel Lymphocyte Marker, Whose Expression Is Deregulated in Leukemia, and a Potential Molecular Druggable Target

Deferasirox Is the Only Iron Chelator Acting as a Potent NF-KB Inhibitor in Myelodysplastic Syndromes

Different Isoforms of the B-Cell Mutator Activation-Induced Cytidine Deaminase (AID) Are Aberrantly Over-Expressed in BCR-ABL1 -Positive Acute Lymphoblastic Leukemia (ALL) Patients and Promote Genetic Instability.

Single Base Pair Mutation Detection by PNA Direct PCR Clamping: Application to Bcr-Abl T315I Mutation

Identification and Molecular Characterization of Two Recurrent Genomic Deletions (Type A and Type B) on 7p12 in IKZF1 Gene in a Large Cohort of BCR-ABL1 -Positive Acute Lymphoblastic Leukemia (ALL): on Behalf of the GIMEMA ALL Working Party

Imatinib Induced Re-Activation of FoxO3 Transcription Factor in CML Is Responsible for the Induction of a Quiescent Status of CD34 Leukaemic Progenitor Cells.

Identification of Genes Sustaining Bcr-Abl Oncogenic Signalling and CML Progression through a Genetic Tool Based on Human Bcr-Abl Transgenic Drosophila Melanogaster.

EphA3 Kinase Is Constitutively Activated in Chronic Myeloid Leukaemia during Progression to Accelerated and Blast Crisis and It Could Represent a New Molecular Target.

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American Society of Hematology

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American Society of Hematology

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