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Journal Articles

Ligandless receptors find a role

Terry J. Hamblin

Blood (2008) 112 (1): 2–3.

https://doi.org/10.1182/blood-2008-03-144113

Published: 2008

Journal Articles

CD38 expression in chronic lymphocytic leukemia is regulated by the tumor microenvironment

Piers E. M. Patten, Andrea G. S. Buggins, Julie Richards, Andrew Wotherspoon, Jon Salisbury, Ghulam J. Mufti, Terry J. Hamblin, Stephen Devereux

Blood (2008) 111 (10): 5173–5181.

https://doi.org/10.1182/blood-2007-08-108605

Published: 2008

Journal Articles

The Combination of Fludarabine and Cyclophosphamide Has a Beneficial Effect on the Incidence of Hemolytic Anemia in Chronic Lymphocytic Leukemia. Results from the UK LRF CLL4 Trial.

Claire E. Dearden, MD, Rachel L. Wade, MSc, Monica Else, MSc, Sue M. Richards, DPhil, Don W. Milligan, MD, Terry J. Hamblin, MD, Daniel Catovsky, DSc

Blood (2007) 110 (11): 2044.

https://doi.org/10.1182/blood.V110.11.2044.2044

Published: 2007

Journal Articles

CD38: what is it there for?

Terry J. Hamblin

Blood (2003) 102 (6): 1939–1940.

https://doi.org/10.1182/blood-2003-07-2332

Published: 2003

Journal Articles

ZAP-70 expression identifies a chronic lymphocytic leukemia subtype with unmutated immunoglobulin genes, inferior clinical outcome, and distinct gene expression profile

Adrian Wiestner, Andreas Rosenwald, Todd S. Barry, George Wright, R. Eric Davis, Sarah E. Henrickson, Hong Zhao, Rachel E. Ibbotson, Jenny A. Orchard, Zadie Davis, Maryalice Stetler-Stevenson, Mark Raffeld, Diane C. Arthur, Gerald E. Marti, Wyndham H. Wilson, Terry J. Hamblin, David G. Oscier, Louis M. Staudt

Blood (2003) 101 (12): 4944–4951.

https://doi.org/10.1182/blood-2002-10-3306

Published: 2003

Journal Articles

The alternative transcript of CD79b is overexpressed in B-CLL and inhibits signaling for apoptosis

Mark S. Cragg, H. T. Claude Chan, Mathew D. Fox, Alison Tutt, Aimée Smith, David G. Oscier, Terry J. Hamblin, Martin J. Glennie

Blood (2002) 100 (9): 3068–3076.

https://doi.org/10.1182/blood.V100.9.3068

Published: 2002

Journal Articles

Multivariate analysis of prognostic factors in CLL: clinical stage, IGVH gene mutational status, and loss or mutation of the p53 gene are independent prognostic factors

David G. Oscier, Anne C. Gardiner, Sarah J. Mould, Sharron Glide, Zadie A. Davis, Rachel E. Ibbotson, Martin M. Corcoran, Robert M. Chapman, Peter W. Thomas, J. Adrian Copplestone, Jenny A. Orchard, Terry J. Hamblin

Blood (2002) 100 (4): 1177–1184.

https://doi.org/10.1182/blood.V100.4.1177.h81602001177_1177_1184

Published: 2002

Journal Articles

Typical Waldenstrom macroglobulinemia is derived from a B-cell arrested after cessation of somatic mutation but prior to isotype switch events

Surinder S. Sahota, Francesco Forconi, Christian H. Ottensmeier, Drew Provan, David G. Oscier, Terry J. Hamblin, Freda K. Stevenson

Blood (2002) 100 (4): 1505–1507.

https://doi.org/10.1182/blood.V100.4.1505.h81602001505_1505_1507

Published: 2002

Journal Articles

Acquisition of potential N-glycosylation sites in the immunoglobulin variable region by somatic mutation is a distinctive feature of follicular lymphoma

Delin Zhu, Helen McCarthy, Christian H. Ottensmeier, Peter Johnson, Terry J. Hamblin, Freda K. Stevenson

Blood (2002) 99 (7): 2562–2568.

https://doi.org/10.1182/blood.V99.7.2562

Published: 2002

Journal Articles

CD38 expression and immunoglobulin variable region mutations are independent prognostic variables in chronic lymphocytic leukemia, but CD38 expression may vary during the course of the disease

Terry J. Hamblin, Jenny A. Orchard, Rachel E. Ibbotson, Zadie Davis, Peter W. Thomas, Freda K. Stevenson, David G. Oscier

Blood (2002) 99 (3): 1023–1029.

https://doi.org/10.1182/blood.V99.3.1023

Published: 2002

Journal Articles

Somatic mutation of bcl-6 genes can occur in the absence of V H mutations in chronic lymphocytic leukemia

Surinder S. Sahota, Zadie Davis, Terry J. Hamblin, Freda K. Stevenson

Blood (2000) 95 (11): 3534–3540.

https://doi.org/10.1182/blood.V95.11.3534

Published: 2000

Journal Articles

Unmutated Ig V H Genes Are Associated With a More Aggressive Form of Chronic Lymphocytic Leukemia

Terry J. Hamblin, Zadie Davis, Anne Gardiner, David G. Oscier, Freda K. Stevenson

Blood (1999) 94 (6): 1848–1854.

https://doi.org/10.1182/blood.V94.6.1848

Published: 1999

Journal Articles

Myeloma V L and V H Gene Sequences Reveal a Complementary Imprint of Antigen Selection in Tumor Cells

Surinder S. Sahota, Regine Leo, Terry J. Hamblin, Freda K. Stevenson

Blood (1997) 89 (1): 219–226.

https://doi.org/10.1182/blood.V89.1.219

Published: 1997

NARROW

Ligandless receptors find a role

CD38 expression in chronic lymphocytic leukemia is regulated by the tumor microenvironment

The Combination of Fludarabine and Cyclophosphamide Has a Beneficial Effect on the Incidence of Hemolytic Anemia in Chronic Lymphocytic Leukemia. Results from the UK LRF CLL4 Trial.

CD38: what is it there for?

ZAP-70 expression identifies a chronic lymphocytic leukemia subtype with unmutated immunoglobulin genes, inferior clinical outcome, and distinct gene expression profile

The alternative transcript of CD79b is overexpressed in B-CLL and inhibits signaling for apoptosis

Multivariate analysis of prognostic factors in CLL: clinical stage, IGVH gene mutational status, and loss or mutation of the p53 gene are independent prognostic factors

Typical Waldenstrom macroglobulinemia is derived from a B-cell arrested after cessation of somatic mutation but prior to isotype switch events

Acquisition of potential N-glycosylation sites in the immunoglobulin variable region by somatic mutation is a distinctive feature of follicular lymphoma

CD38 expression and immunoglobulin variable region mutations are independent prognostic variables in chronic lymphocytic leukemia, but CD38 expression may vary during the course of the disease

Somatic mutation of bcl-6 genes can occur in the absence of V H mutations in chronic lymphocytic leukemia

Unmutated Ig V H Genes Are Associated With a More Aggressive Form of Chronic Lymphocytic Leukemia

Myeloma V L and V H Gene Sequences Reveal a Complementary Imprint of Antigen Selection in Tumor Cells

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American Society of Hematology

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ASH Publications

American Society of Hematology

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