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Journal Articles

The Second Signal in Antigen-Presenting Cells: Complementary JAK-1 Directs Pro- and Anti-Inflammatory Cytokine Production Induced by CD40L.

Michael Conzelmann, Andreas Wagner, Anke Hildebrandt, Elena Rodionova, Michael Hess, Annika Zota, Thomas Giese, Christine Falk, Stephen Breit, Markus Hecker, Anthony D. Ho, Peter Dreger, Thomas Luft

Blood (2009) 114 (22): 1348.

https://doi.org/10.1182/blood.V114.22.1348.1348

Published: 2009

Journal Articles

The Favorable Effect of Activating NOTCH1 Receptor Mutations On Long Term Outcome in T-ALL Is Treatment Related and Can Be Separated From NOTCH Pathway Activation by FBXW7 Loss of Function.

Corinne Kox, Martin Zimmermann, PhD, Martin Stanulla, MD, Martin Schrappe, MD, Wolf-Dieter Ludwig, Rolf Koehler, PhD, Svenja Leible, Gabriele Tolle, Obul Reddy Bandapalli, Stephen Breit, Martina Ulrike Muckenthaler, Andreas Kulozik, MD, PhD

Blood (2009) 114 (22): 908.

https://doi.org/10.1182/blood.V114.22.908.908

Published: 2009

Journal Articles

High-resolution genomic profiling of childhood T-ALL reveals frequent copy-number alterations affecting the TGF-β and PI3K-AKT pathways and deletions at 6q15-16.1 as a genomic marker for unfavorable early treatment response

Blood (2009) 114 (5): 1053–1062.

https://doi.org/10.1182/blood-2008-10-186536

Published: 2009

Includes: Supplemental data

Journal Articles

Notch Pathway Activation by a Combination of Activating NOTCH1 -Receptor and Inactivating FBXW7 Mutations Robustly Predicts Excellent Early Treatment Response in Pediatric T-ALL.

Corinne Kox, Svenja Leible, Stephen Breit, Martin Stanulla, MD, Martin Schrappe, MD, Martina Ulrike Muckenthaler, Andreas Kulozik

Blood (2008) 112 (11): 1493.

https://doi.org/10.1182/blood.V112.11.1493.1493

Published: 2008

Journal Articles

The Second Signal in Antigen-Presenting Cells: Complementary JAK1/JAK2 and CD40 Signalling Mediate Pro-Inflammatory Activation.

Michael Conzelmann, Elena Rodionova, Michael Hess, Annika Zota, Thomas Giese, Andreas Wagner, Stephen Breit, Markus Hecker, Anthony D. Ho, Peter Dreger, Thomas Luft

Blood (2008) 112 (11): 1261.

https://doi.org/10.1182/blood.V112.11.1261.1261

Published: 2008

Journal Articles

The Early Treatment Response of the Clinically Challenging Group of Childhood T-ALL without NOTCH1 Mutations Is Signified by a Specific mRNA Gene Profile.

Andreas Kulozik, Stephen Breit, Stefan Pfister, Shangyou Liu, Martin Stanulla, Martin Schrappe, Wolf-Dieter Ludwig, Ulrike Köhl, Arndt Borkhardt, Renate Kirschner-Schwabe, Martina U. Muckenthaler

Blood (2007) 110 (11): 2789.

https://doi.org/10.1182/blood.V110.11.2789.2789

Published: 2007

Journal Articles

Activating NOTCH1 mutations predict favorable early treatment response and long-term outcome in childhood precursor T-cell lymphoblastic leukemia

Stephen Breit, Martin Stanulla, Thomas Flohr, Martin Schrappe, Wolf-Dieter Ludwig, Gabriele Tolle, Margit Happich, Martina U. Muckenthaler, Andreas E. Kulozik

Blood (2006) 108 (4): 1151–1157.

https://doi.org/10.1182/blood-2005-12-4956

Published: 2006

Includes: Supplemental data

Journal Articles

Activating NOTCH1 Mutations Are Associated with Good Treatment Response in Childhood T-Cell Acute Lymphoblastic Leukemia.

Stephen Breit, PhD, Martin Stanulla, MD, Thomas Flohr, PhD, Martin Schrappe, MD, Wolf-Dieter Ludwig, MD, Gabriele Tolle, Margit Happich, Martina M. Muckenthaler, PhD, Andreas E. Kulozik, MD, PhD

Blood (2005) 106 (11): 1444.

https://doi.org/10.1182/blood.V106.11.1444.1444

Published: 2005

NARROW

The Second Signal in Antigen-Presenting Cells: Complementary JAK-1 Directs Pro- and Anti-Inflammatory Cytokine Production Induced by CD40L.

The Favorable Effect of Activating NOTCH1 Receptor Mutations On Long Term Outcome in T-ALL Is Treatment Related and Can Be Separated From NOTCH Pathway Activation by FBXW7 Loss of Function.

High-resolution genomic profiling of childhood T-ALL reveals frequent copy-number alterations affecting the TGF-β and PI3K-AKT pathways and deletions at 6q15-16.1 as a genomic marker for unfavorable early treatment response

Notch Pathway Activation by a Combination of Activating NOTCH1 -Receptor and Inactivating FBXW7 Mutations Robustly Predicts Excellent Early Treatment Response in Pediatric T-ALL.

The Second Signal in Antigen-Presenting Cells: Complementary JAK1/JAK2 and CD40 Signalling Mediate Pro-Inflammatory Activation.

The Early Treatment Response of the Clinically Challenging Group of Childhood T-ALL without NOTCH1 Mutations Is Signified by a Specific mRNA Gene Profile.

Activating NOTCH1 mutations predict favorable early treatment response and long-term outcome in childhood precursor T-cell lymphoblastic leukemia

Activating NOTCH1 Mutations Are Associated with Good Treatment Response in Childhood T-Cell Acute Lymphoblastic Leukemia.

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American Society of Hematology

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ASH Publications

American Society of Hematology

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