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Journal Articles

An Allosteric Mechanism for Epigenetic Activation of the V(D)J Recombinase

Meiling May, BA, Stephen Desiderio, MD PhD, John Thomas Bettridge, BA

Blood (2018) 132 (Supplement 1): 512.

https://doi.org/10.1182/blood-2018-99-116675

Published: 2018

Journal Articles

Smoothened signaling in the mouse osteoblastoid lineage is required for efficient B lymphopoiesis

Wenyan Lu, Dominic Dordai, David Huso, Stephen Desiderio

Blood (2018) 131 (3): 323–327.

https://doi.org/10.1182/blood-2017-06-793539

Published: 2018

Includes: Supplemental data

Journal Articles

Allosteric Regulation of V(D)J Recombination By Active Chromatin

John T Bettridge, Chan-Hyun Na, PhD, Akhilesh Pandey, MD PhD, Stephen Desiderio, MD PhD

Blood (2016) 128 (22): 3707.

https://doi.org/10.1182/blood.V128.22.3707.3707

Published: 2016

Journal Articles

Non–cell-autonomous hedgehog signaling promotes murine B lymphopoiesis from hematopoietic progenitors

Christopher L. Cooper, Richard R. Hardy, Michael Reth, Stephen Desiderio

Blood (2012) 119 (23): 5438–5448.

https://doi.org/10.1182/blood-2011-12-397976

Published: 2012

Includes: Supplemental data

Journal Articles

Knock-in of a FLT3/ITD mutation cooperates with a NUP98-HOXD13 fusion to generate acute myeloid leukemia in a mouse model

Sarah Greenblatt, Li Li, Christopher Slape, Bao Nguyen, Rachel Novak, Amy Duffield, David Huso, Stephen Desiderio, Michael J. Borowitz, Peter Aplan, Donald Small

Blood (2012) 119 (12): 2883–2894.

https://doi.org/10.1182/blood-2011-10-382283

Published: 2012

Includes: Supplemental data

Journal Articles

Defective nonhomologous end joining blocks B-cell development in FLT3/ITD mice

Li Li, Li Zhang, Jinshui Fan, Kathleen Greenberg, Stephen Desiderio, Feyruz V. Rassool, Donald Small

Blood (2011) 117 (11): 3131–3139.

https://doi.org/10.1182/blood-2010-05-286070

Published: 2011

Includes: Supplemental data

Journal Articles

B-Lymphocyte Development Is Impaired by FLT3/ITD Signaling in Knock-in Mice Because of Defective Non-Homologous End-Joining.

Li Li, MD, PhD, Li Zhang, PhD, Jinshui Fan, PhD, Kathleen E Greenberg, BS, Stephen Desiderio, MD, PhD, Feyruz V. Rassool, PhD, Donald Small, MD, PhD

Blood (2009) 114 (22): 184.

https://doi.org/10.1182/blood.V114.22.184.184

Published: 2009

NARROW

An Allosteric Mechanism for Epigenetic Activation of the V(D)J Recombinase

Smoothened signaling in the mouse osteoblastoid lineage is required for efficient B lymphopoiesis

Allosteric Regulation of V(D)J Recombination By Active Chromatin

Non–cell-autonomous hedgehog signaling promotes murine B lymphopoiesis from hematopoietic progenitors

Knock-in of a FLT3/ITD mutation cooperates with a NUP98-HOXD13 fusion to generate acute myeloid leukemia in a mouse model

Defective nonhomologous end joining blocks B-cell development in FLT3/ITD mice

B-Lymphocyte Development Is Impaired by FLT3/ITD Signaling in Knock-in Mice Because of Defective Non-Homologous End-Joining.

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ASH Publications

American Society of Hematology

An Allosteric Mechanism for Epigenetic Activation of the V(D)J Recombinase

Smoothened signaling in the mouse osteoblastoid lineage is required for efficient B lymphopoiesis

Allosteric Regulation of V(D)J Recombination By Active Chromatin

Non–cell-autonomous hedgehog signaling promotes murine B lymphopoiesis from hematopoietic progenitors

Knock-in of a FLT3/ITD mutation cooperates with a NUP98-HOXD13 fusion to generate acute myeloid leukemia in a mouse model

Defective nonhomologous end joining blocks B-cell development in FLT3/ITD mice

B-Lymphocyte Development Is Impaired by FLT3/ITD Signaling in Knock-in Mice Because of Defective Non-Homologous End-Joining.

Email alerts

ASH Publications

American Society of Hematology

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