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Journal Articles

Defective nonhomologous end joining blocks B-cell development in FLT3/ITD mice

Li Li, Li Zhang, Jinshui Fan, Kathleen Greenberg, Stephen Desiderio, Feyruz V. Rassool, Donald Small

Blood (2011) 117 (11): 3131–3139.

https://doi.org/10.1182/blood-2010-05-286070

Published: 2011

Includes: Supplemental data

Journal Articles

Cells expressing FLT3/ITD mutations exhibit elevated repair errors generated through alternative NHEJ pathways: implications for genomic instability and therapy

Jinshui Fan, Li Li, Donald Small, Feyruz Rassool

Blood (2010) 116 (24): 5298–5305.

https://doi.org/10.1182/blood-2010-03-272591

Published: 2010

Includes: Supplemental data

Journal Articles

B-Lymphocyte Development Is Impaired by FLT3/ITD Signaling in Knock-in Mice Because of Defective Non-Homologous End-Joining.

Li Li, MD, PhD, Li Zhang, PhD, Jinshui Fan, PhD, Kathleen E Greenberg, BS, Stephen Desiderio, MD, PhD, Feyruz V. Rassool, PhD, Donald Small, MD, PhD

Blood (2009) 114 (22): 184.

https://doi.org/10.1182/blood.V114.22.184.184

Published: 2009

Journal Articles

Cell Lines and “Knock-in” Mice Bearing FLT3/ITD Mutations Exhibit Elevated Repair Errors Generated through Alternative DNA Double Strand Break Repair Pathways: Implications for Genomic Instability.

Jinshui Fan, PhD, Li Li, MD, PhD, Donald Small, MD, PhD, Feyruz V. Rassool

Blood (2009) 114 (22): 3237.

https://doi.org/10.1182/blood.V114.22.3237.3237

Published: 2009

Journal Articles

Internal tandem duplication of FLT3 (FLT3/ITD) induces increased ROS production, DNA damage, and misrepair: implications for poor prognosis in AML

Annahita Sallmyr, Jinshui Fan, Kamal Datta, Kyu-Tae Kim, Dan Grosu, Paul Shapiro, Donald Small, Feyruz Rassool

Blood (2008) 111 (6): 3173–3182.

https://doi.org/10.1182/blood-2007-05-092510

Published: 2008

Includes: Supplemental data

Journal Articles

Internal Tandem Duplications of FLT3 Induces Increased ROS Production, DNA Damage and Misrepair: Implications for Genomic Instability and Disease Resistance in Myeloid Malignancies.

Jinshui Fan, PhD, Annahita Sallmyr, PhD, Kyu-Te Kim, PhD, Kamal Datta, MD, Paul Shapiro, PhD, Donald Small, MD, Feyruz V. Rassool, PhD

Blood (2007) 110 (11): 17.

https://doi.org/10.1182/blood.V110.11.17.17

Published: 2007

NARROW

Defective nonhomologous end joining blocks B-cell development in FLT3/ITD mice

Cells expressing FLT3/ITD mutations exhibit elevated repair errors generated through alternative NHEJ pathways: implications for genomic instability and therapy

B-Lymphocyte Development Is Impaired by FLT3/ITD Signaling in Knock-in Mice Because of Defective Non-Homologous End-Joining.

Cell Lines and “Knock-in” Mice Bearing FLT3/ITD Mutations Exhibit Elevated Repair Errors Generated through Alternative DNA Double Strand Break Repair Pathways: Implications for Genomic Instability.

Internal tandem duplication of FLT3 (FLT3/ITD) induces increased ROS production, DNA damage, and misrepair: implications for poor prognosis in AML

Internal Tandem Duplications of FLT3 Induces Increased ROS Production, DNA Damage and Misrepair: Implications for Genomic Instability and Disease Resistance in Myeloid Malignancies.

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ASH Publications

American Society of Hematology

Defective nonhomologous end joining blocks B-cell development in FLT3/ITD mice

Cells expressing FLT3/ITD mutations exhibit elevated repair errors generated through alternative NHEJ pathways: implications for genomic instability and therapy

B-Lymphocyte Development Is Impaired by FLT3/ITD Signaling in Knock-in Mice Because of Defective Non-Homologous End-Joining.

Cell Lines and “Knock-in” Mice Bearing FLT3/ITD Mutations Exhibit Elevated Repair Errors Generated through Alternative DNA Double Strand Break Repair Pathways: Implications for Genomic Instability.

Internal tandem duplication of FLT3 (FLT3/ITD) induces increased ROS production, DNA damage, and misrepair: implications for poor prognosis in AML

Internal Tandem Duplications of FLT3 Induces Increased ROS Production, DNA Damage and Misrepair: Implications for Genomic Instability and Disease Resistance in Myeloid Malignancies.

Email alerts

ASH Publications

American Society of Hematology

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