Page 1 | Search Results | Blood | American Society of Hematology

1-20 of 22

Sort by

Sort Order Select

Journal Articles

Plasminogen Mediates Both Positive and Negative Effects on Disease Severity in a Mouse Model of TNFα-Driven Arthritis

Harini Raghu, Malinda D. Frederick, Alice Jone, Kathryn E. Talmage, Keith W. Kombrinck, Matthew J. Flick, PhD

Blood (2011) 118 (21): 854.

https://doi.org/10.1182/blood.V118.21.854.854

Published: 2011

Journal Articles

Factor XIII Deficiency Improves Motor Function in Mice Challenged with Experimental Autoimmune Encephalomyelitis, An Established Model of Multiple Sclerosis

Eric S. Mullins, MD, Kathryn E. Talmage, Keith W. Kombrinck, Whitney M Miller, Joseph S. Palumbo, MD, Jay L. Degen, PhD

Blood (2011) 118 (21): 858.

https://doi.org/10.1182/blood.V118.21.858.858

Published: 2011

Journal Articles

Thrombomodulin is a determinant of metastasis through a mechanism linked to the thrombin binding domain but not the lectin-like domain

Netanel A. Horowitz, Elizabeth A. Blevins, Whitney M. Miller, Ashley R. Perry, Kathryn E. Talmage, Eric S. Mullins, Matthew J. Flick, Karla C. S. Queiroz, Kun Shi, C. Arnold Spek, Edward M. Conway, Brett P. Monia, Hartmut Weiler, Jay L. Degen, Joseph S. Palumbo

Blood (2011) 118 (10): 2889–2895.

https://doi.org/10.1182/blood-2011-03-341222

Published: 2011

Includes: Supplemental data

Journal Articles

The development of inflammatory joint disease is attenuated in mice expressing the anticoagulant prothrombin mutant W215A/E217A

Matthew J. Flick, Anil K. Chauhan, Malinda Frederick, Kathryn E. Talmage, Keith W. Kombrinck, Whitney Miller, Eric S. Mullins, Joseph S. Palumbo, Xunzhen Zheng, Naomi L. Esmon, Charles T. Esmon, Sherry Thornton, Ann Becker, Leslie A. Pelc, Enrico Di Cera, Denisa D. Wagner, Jay L. Degen

Blood (2011) 117 (23): 6326–6337.

https://doi.org/10.1182/blood-2010-08-304915

Published: 2011

Journal Articles

Host Prothrombin and Fibrinogen Support S. Aureus Virulence In the Context of Acute Bacteremia

Matthew J. Flick, PhD, Xinli Du, PhD, Leslie Fausnacht, Harini Raghu, Kathryn E Talmage, Keith W Kombrinck, Elizabeth A. Blevins, Eric S. Mullins, MD, Joseph S. Palumbo, MD, Jay L. Degen, PhD

Blood (2010) 116 (21): 821.

https://doi.org/10.1182/blood.V116.21.821.821

Published: 2010

Journal Articles

Thrombin-Thrombomodulin Interactions Are An Important Determinant of Metastatic Potential

Netanel A Horowitz, MD, Elizabeth A. Blevins, Whitney M Miller, Ashely R Perry, Kathryn E Talmage, Eric S. Mullins, MD, Brett P. Monia, PhD, Jay L. Degen, PhD, Joseph S. Palumbo, MD

Blood (2010) 116 (21): 822.

https://doi.org/10.1182/blood.V116.21.822.822

Published: 2010

Journal Articles

Host Fibrinogen and the S. Aureus -Encoded Procoagulant Vwbp Are Context-Dependent Determinants of Bacterial Virulence.

Joni M Prasad, Xinli Du, MD, PhD, Eric S. Mullins, MD, Senad Divanovic, PhD, Kathryn E Talmage, Keith W Kombrinck, Alice G Cheng, Olaf Schneewind, MD, PhD, Matthew J. Flick, PhD, Jay L. Degen, PhD

Blood (2010) 116 (21): 1152.

https://doi.org/10.1182/blood.V116.21.1152.1152

Published: 2010

Journal Articles

The Virulence of S. Aureus Is Supported by Host Fibrinogen and Its Bacterial Receptor ClfA in the Context of Bacteremia.

Matthew J Flick, PhD, Xinli Du, Kathryn E. Talmage, Keith W. Kombrink, Elizabeth A. Blevins, Joseph S. Palumbo, MD, Jay L Degen, PhD

Blood (2009) 114 (22): 3188.

https://doi.org/10.1182/blood.V114.22.3188.3188

Published: 2009

Journal Articles

Transfusion Related Acute Lung Injury (TRALI) Is Ameliorated by Genetic Elimination of Fibrin(ogen) in Mice.

Eric S. Mullins, MD, Kathryn E. Talmage, Matthew Flick, PhD, Keith W. Kombrinck, Richard T Strait, MD, Jay L Degen, PhD

Blood (2009) 114 (22): 3189.

https://doi.org/10.1182/blood.V114.22.3189.3189

Published: 2009

Journal Articles

Genetic elimination of prothrombin in adult mice is not compatible with survival and results in spontaneous hemorrhagic events in both heart and brain

Eric S. Mullins, Keith W. Kombrinck, Kathryn E. Talmage, Maureen A. Shaw, David P. Witte, Joni M. Ullman, Sandra J. Degen, William Sun, Matthew J. Flick, Jay L. Degen

Blood (2009) 113 (3): 696–704.

https://doi.org/10.1182/blood-2008-07-169003

Published: 2009

Includes: Supplemental data

Journal Articles

Loss of Clotting Function in Mice Expressing a Mutant Form of Fibrinogen Lacking the α Chain Thrombin Cleavage Site

Matthew Flick, PhD, Joni M. Ullman, Joseph S. Palumbo, MD, Eric S. Mullins, MD, Keith W Kombrink, Kathryn E. Talmage, Maureen A. Shaw, Susan T. Lord, Ph.D., Jay Degen, Oleg V Gorkun

Blood (2008) 112 (11): 394.

https://doi.org/10.1182/blood.V112.11.394.394

Published: 2008

Journal Articles

Genetic Elimination of Prothrombin in Adult Mice Results in Fatal Spontaneous Hemorrhage in the Heart and Central Nervous System

Eric S. Mullins, MD, Keith W. Kombrinck, Kathryn E. Talmage, Maureen A. Shaw, David P. Witte, Joni M. Ullman, Sandra J. Degen, William Sun, Matthew Flick, PhD, Jay Degen

Blood (2008) 112 (11): 395.

https://doi.org/10.1182/blood.V112.11.395.395

Published: 2008

Journal Articles

Mice Engineered To Express a Form of Thrombin Favoring Protein C Are Resistant to S. aureus -Induced Sepsis.

Matthew J. Flick, PhD, Anil K. Chauhan, PhD, Sara Welch, BS, Maureen A. Shaw, BS, Kathryn E. Talmage, BS, Eric S. Mullins, MD, Joseph S. Palumbo, MD, Sherry Thornton, PhD, Denisa D. Wagner, PhD, Jay L. Degen, PhD

Blood (2007) 110 (11): 267.

https://doi.org/10.1182/blood.V110.11.267.267

Published: 2007

Journal Articles

Factor XIII Supports Metastatic Potential through a Mechanism Coupled to Natural Killer Cell Function.

Joseph S. Palumbo, MD, Kelley A. Barney, Elizabeth A. Williams, Maureen A. Shaw, Anjali Mishra, PhD, Matthew J. Flick, PhD, Kathryn E. Talmage, Jay L. Degen, PhD

Blood (2007) 110 (11): 1753.

https://doi.org/10.1182/blood.V110.11.1753.1753

Published: 2007

Journal Articles

Tumor cell–associated tissue factor and circulating hemostatic factors cooperate to increase metastatic potential through natural killer cell–dependent and–independent mechanisms

Joseph S. Palumbo, Kathryn E. Talmage, Jessica V. Massari, Christine M. La Jeunesse, Matthew J. Flick, Keith W. Kombrinck, Zhiwei Hu, Kelley A. Barney, Jay L. Degen

Blood (2007) 110 (1): 133–141.

https://doi.org/10.1182/blood-2007-01-065995

Published: 2007

Journal Articles

Fibrin(ogen) Exacerbates Inflammatory Joint Disease Via a Mechanism Linked to Its αMβ2 Binading Motif.

Matthew J. Flick, PhD, Christine M. La Jeunesse, Kathryn E. Talmage, David P. Witte, MD, Joseph Palumbo, MD, Malinda D. Pinkerton, Sherry Thornton, PhD, Jay L. Degen, PhD

Blood (2006) 108 (11): 64.

https://doi.org/10.1182/blood.V108.11.64.64

Published: 2006

Journal Articles

Tumor Cell-Associated Tissue Factor Supports Metastatic Potential through Both NK Cell-Dependent and -Independent Mechanisms.

Joseph Palumbo, MD, Kelley A. Barney, Matthew J. Flick, PhD, Kathryn E. Talmage, Keith W. Kombrinck, Zhiwei Hu, PhD, Christine M. La Jeunesse, Jay L. Degen, PhD

Blood (2006) 108 (11): 66.

https://doi.org/10.1182/blood.V108.11.66.66

Published: 2006

Journal Articles

Hemostatic Factors Regulate Immune Surveillance and Contribute to the Rapid Clearance of Bacterial Pathogens In Vivo.

Matthew J. Flick, PhD, Xinli Du, PhD, Kathryn E. Talmage, Christine M. LaJeunesse, David P. Witte, MD, Joseph S. Palumbo, MD, Jay L. Degen, PhD

Blood (2005) 106 (11): 2132.

https://doi.org/10.1182/blood.V106.11.2132.2132

Published: 2005

Journal Articles

Interplay between Tumor Cell-Associated and Circulating Coagulation Factors in Establishing Metastatic Potential.

Joseph S. Palumbo, MD, Jessica V. Massari, Christine M. La Jeunesse, Matthew J. Flick, Zhiwei Hu, PhD, Kathryn E. Talmage, Jay L. Degen, PhD

Blood (2005) 106 (11): 686.

https://doi.org/10.1182/blood.V106.11.686.686

Published: 2005

Journal Articles

Platelets and fibrin(ogen) increase metastatic potential by impeding natural killer cell–mediated elimination of tumor cells

Joseph S. Palumbo, Kathryn E. Talmage, Jessica V. Massari, Christine M. La Jeunesse, Matthew J. Flick, Keith W. Kombrinck, Markéta Jirousková, Jay L. Degen

Blood (2005) 105 (1): 178–185.

https://doi.org/10.1182/blood-2004-06-2272

Published: 2005

NARROW

Plasminogen Mediates Both Positive and Negative Effects on Disease Severity in a Mouse Model of TNFα-Driven Arthritis

Factor XIII Deficiency Improves Motor Function in Mice Challenged with Experimental Autoimmune Encephalomyelitis, An Established Model of Multiple Sclerosis

Thrombomodulin is a determinant of metastasis through a mechanism linked to the thrombin binding domain but not the lectin-like domain

The development of inflammatory joint disease is attenuated in mice expressing the anticoagulant prothrombin mutant W215A/E217A

Host Prothrombin and Fibrinogen Support S. Aureus Virulence In the Context of Acute Bacteremia

Thrombin-Thrombomodulin Interactions Are An Important Determinant of Metastatic Potential

Host Fibrinogen and the S. Aureus -Encoded Procoagulant Vwbp Are Context-Dependent Determinants of Bacterial Virulence.

The Virulence of S. Aureus Is Supported by Host Fibrinogen and Its Bacterial Receptor ClfA in the Context of Bacteremia.

Transfusion Related Acute Lung Injury (TRALI) Is Ameliorated by Genetic Elimination of Fibrin(ogen) in Mice.

Genetic elimination of prothrombin in adult mice is not compatible with survival and results in spontaneous hemorrhagic events in both heart and brain

Loss of Clotting Function in Mice Expressing a Mutant Form of Fibrinogen Lacking the α Chain Thrombin Cleavage Site

Genetic Elimination of Prothrombin in Adult Mice Results in Fatal Spontaneous Hemorrhage in the Heart and Central Nervous System

Mice Engineered To Express a Form of Thrombin Favoring Protein C Are Resistant to S. aureus -Induced Sepsis.

Factor XIII Supports Metastatic Potential through a Mechanism Coupled to Natural Killer Cell Function.

Tumor cell–associated tissue factor and circulating hemostatic factors cooperate to increase metastatic potential through natural killer cell–dependent and–independent mechanisms

Fibrin(ogen) Exacerbates Inflammatory Joint Disease Via a Mechanism Linked to Its αMβ2 Binading Motif.

Tumor Cell-Associated Tissue Factor Supports Metastatic Potential through Both NK Cell-Dependent and -Independent Mechanisms.

Hemostatic Factors Regulate Immune Surveillance and Contribute to the Rapid Clearance of Bacterial Pathogens In Vivo.

Interplay between Tumor Cell-Associated and Circulating Coagulation Factors in Establishing Metastatic Potential.

Platelets and fibrin(ogen) increase metastatic potential by impeding natural killer cell–mediated elimination of tumor cells

Email alerts

ASH Publications

American Society of Hematology

Email alerts

ASH Publications

American Society of Hematology

This Feature Is Available To Subscribers Only