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Journal Articles

Fibrinogen Supports Colitis-Associated Adenoma Progression through a Mechanism Coupled to Engagement of the Leukocyte Integrin α M β 2 .

Netanel Horowitz, MD, Kris A. Steinbrecher, Ph.D, Maureen A. Shaw, Kelley A. Barney, Matthew Flick, PhD, Eleana Harmel-Laws, David Witte, M.D., Elizabeth A. Blevins, Joseph S. Palumbo, MD

Blood (2009) 114 (22): 334.

https://doi.org/10.1182/blood.V114.22.334.334

Published: 2009

Journal Articles

Hemostatic Factors Support Colitis and Colitis-Associated Colon Cancer

Kris A. Steinbrecher, Netanel Horowitz, Maureen A. Shaw, Kelley A. Barney, Matthew J. Flick, Eleana Harmel-Laws, David P. Witte, Elizabeth A. Blevins, Joseph S. Palumbo, MD

Blood (2008) 112 (11): 3079.

https://doi.org/10.1182/blood.V112.11.3079.3079

Published: 2008

Journal Articles

Factor XIII Supports Metastatic Potential through a Mechanism Coupled to Natural Killer Cell Function.

Joseph S. Palumbo, MD, Kelley A. Barney, Elizabeth A. Williams, Maureen A. Shaw, Anjali Mishra, PhD, Matthew J. Flick, PhD, Kathryn E. Talmage, Jay L. Degen, PhD

Blood (2007) 110 (11): 1753.

https://doi.org/10.1182/blood.V110.11.1753.1753

Published: 2007

Journal Articles

Tumor cell–associated tissue factor and circulating hemostatic factors cooperate to increase metastatic potential through natural killer cell–dependent and–independent mechanisms

Joseph S. Palumbo, Kathryn E. Talmage, Jessica V. Massari, Christine M. La Jeunesse, Matthew J. Flick, Keith W. Kombrinck, Zhiwei Hu, Kelley A. Barney, Jay L. Degen

Blood (2007) 110 (1): 133–141.

https://doi.org/10.1182/blood-2007-01-065995

Published: 2007

Journal Articles

Tumor Cell-Associated Tissue Factor Supports Metastatic Potential through Both NK Cell-Dependent and -Independent Mechanisms.

Joseph Palumbo, MD, Kelley A. Barney, Matthew J. Flick, PhD, Kathryn E. Talmage, Keith W. Kombrinck, Zhiwei Hu, PhD, Christine M. La Jeunesse, Jay L. Degen, PhD

Blood (2006) 108 (11): 66.

https://doi.org/10.1182/blood.V108.11.66.66

Published: 2006

NARROW

Fibrinogen Supports Colitis-Associated Adenoma Progression through a Mechanism Coupled to Engagement of the Leukocyte Integrin α M β 2 .

Hemostatic Factors Support Colitis and Colitis-Associated Colon Cancer

Factor XIII Supports Metastatic Potential through a Mechanism Coupled to Natural Killer Cell Function.

Tumor cell–associated tissue factor and circulating hemostatic factors cooperate to increase metastatic potential through natural killer cell–dependent and–independent mechanisms

Tumor Cell-Associated Tissue Factor Supports Metastatic Potential through Both NK Cell-Dependent and -Independent Mechanisms.

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ASH Publications

American Society of Hematology

Fibrinogen Supports Colitis-Associated Adenoma Progression through a Mechanism Coupled to Engagement of the Leukocyte Integrin α M β 2 .

Hemostatic Factors Support Colitis and Colitis-Associated Colon Cancer

Factor XIII Supports Metastatic Potential through a Mechanism Coupled to Natural Killer Cell Function.

Tumor cell–associated tissue factor and circulating hemostatic factors cooperate to increase metastatic potential through natural killer cell–dependent and–independent mechanisms

Tumor Cell-Associated Tissue Factor Supports Metastatic Potential through Both NK Cell-Dependent and -Independent Mechanisms.

Email alerts

ASH Publications

American Society of Hematology

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