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Journal Articles

BCL6 modulates tonic BCR signaling in diffuse large B-cell lymphomas by repressing the SYK phosphatase, PTPROt

Przemyslaw Juszczynski, Linfeng Chen, Evan O'Donnell, Jose M. Polo, Stella M. Ranuncolo, Riccardo Dalla-Favera, Ari Melnick, Margaret A. Shipp

Blood (2009) 114 (26): 5315–5321.

https://doi.org/10.1182/blood-2009-02-204362

Published: 2009

Includes: Supplemental data

Journal Articles

BCL6 Is Critical for the Development of a Diverse Primary B Cell Repertoire.

Cihangir Duy, J. Jessica Yu, Srividya Swaminathan, Rahul R. Nahar, Soo-mi Kweon, Jose M. Polo, PhD, Ester Valls, Lars Klemm, Leandro Cerchietti, Gregor von Levetzow, Sebastian Herzog, Hassan Jumaa, Ignacio Moreno de Alborán, Ari Melnick, B. Hilda Ye, PhD, Markus Müschen

Blood (2009) 114 (22): 91.

https://doi.org/10.1182/blood.V114.22.91.91

Published: 2009

Journal Articles

The BCL6 transcriptional program features repression of multiple oncogenes in primary B cells and is deregulated in DLBCL

Weimin Ci, Jose M. Polo, Leandro Cerchietti, Rita Shaknovich, Ling Wang, Shao Ning Yang, Kenny Ye, Pedro Farinha, Douglas E. Horsman, Randy D. Gascoyne, Olivier Elemento, Ari Melnick

Blood (2009) 113 (22): 5536–5548.

https://doi.org/10.1182/blood-2008-12-193037

Published: 2009

Includes: Supplemental data

Journal Articles

A peptomimetic inhibitor of BCL6 with potent antilymphoma effects in vitro and in vivo

Leandro C. Cerchietti, Shao Ning Yang, Rita Shaknovich, Katerina Hatzi, Jose M. Polo, Amy Chadburn, Steven F. Dowdy, Ari Melnick

Blood (2009) 113 (15): 3397–3405.

https://doi.org/10.1182/blood-2008-07-168773

Published: 2009

Includes: Supplemental data

Journal Articles

BCL6 Regulates Tonic BCR Signaling in Diffuse Large B-Cell Lymphomas by Repressing the SYK Phosphatase, PTPROt

Przemyslaw JuszczynsKi, Linfeng Chen, Jose M. Polo, Stella M. Ranuncolo, Riccardo Dalla-Favera, Ari Melnick, Margaret A. Shipp

Blood (2008) 112 (11): 802.

https://doi.org/10.1182/blood.V112.11.802.802

Published: 2008

Journal Articles

Reversible disruption of BCL6 repression complexes by CD40 signaling in normal and malignant B cells

Jose M. Polo, Weimin Ci, Jonathan D. Licht, Ari Melnick

Blood (2008) 112 (3): 644–651.

https://doi.org/10.1182/blood-2008-01-131813

Published: 2008

Includes: Supplemental data

Journal Articles

BCL6 Transcriptionally Reprograms the DLBCL Genome by Forming Distinct Biochemically Active Complexes on Genes Corresponding to Different Pathways.

Jose M. Polo, MS, Katerina Chatzi, PhD, Tania Dell’Oso, PhD, Paola Lev, PhD, Ari Melnick, PhD

Blood (2007) 110 (11): 2646.

https://doi.org/10.1182/blood.V110.11.2646.2646

Published: 2007

Journal Articles

Design and Development of Small Molecules for Specific Targeted Therapy of Diffuse Large B-Cell Lymphoma.

Gustavo F. Da Silva, MS, Alex Ghetu, PhD, Leandro Cerchietti, MD, Jose M. Polo, MS, Andrew Coop, PhD, Alexander Mackerell, PhD, Gilbert G. Prive, PhD, Ari Melnick, MD

Blood (2007) 110 (11): 799.

https://doi.org/10.1182/blood.V110.11.799.799

Published: 2007

Journal Articles

A “Survival of the Fittest” Mechanism for Weeding Out Potentially Lymphomagenic B-Cells during Germinal Center B-Cell Differentiation.

Weimin Ci, Jose M. Polo, Stella M. Ranuncolo, Ari Melnick

Blood (2007) 110 (11): 559.

https://doi.org/10.1182/blood.V110.11.559.559

Published: 2007

Journal Articles

BCL6 programs lymphoma cells for survival and differentiation through distinct biochemical mechanisms

Samir Parekh, Jose M. Polo, Rita Shaknovich, Przemyslaw Juszczynski, Paola Lev, Stella M. Ranuncolo, Yingnan Yin, Ulf Klein, Giorgio Cattoretti, Riccardo Dalla Favera, Margaret A. Shipp, Ari Melnick

Blood (2007) 110 (6): 2067–2074.

https://doi.org/10.1182/blood-2007-01-069575

Published: 2007

Includes: Supplemental data

Journal Articles

A Critical BCL6-Related Feedback Loop Explains the Unusual Biological Features of Germinal Center B-Cells and Their Malignant Transformation into B-Cell Lymphomas.

Stella M. Ranuncolo, MD, PhD, Jose M. Polo, MS, Jamil Dierov, PhD, Martin Carroll, MD, Ari M. Melnick, MD

Blood (2006) 108 (11): 224.

https://doi.org/10.1182/blood.V108.11.224.224

Published: 2006

Journal Articles

A BCL6 Target Gene Signature Predicts the Biological Behavior and Classification of Diffuse Large B-Cell Lymphoma.

Jose M. Polo, MS, Przemyslaw Juszczynski, MD, PhD, Leandro Cerchietti, MD, Stefano Monti, MD, Kenny Ye, PhD, Margaret A. Shipp, MD, Ari M. Melnick, MD

Blood (2006) 108 (11): 616.

https://doi.org/10.1182/blood.V108.11.616.616

Published: 2006

Journal Articles

BCL6, p53 and Molecular Targeted Therapy in B-Cell Lymphomas.

Leandro C.A. Cerchietti, MD, Jose M. Polo, MS, Gustavo F. Da Silva, Pharm D, Steve M. Dowdy, PhD, Catoretti M. Giorgio, MD, Shaknovich M. Rita, MD, Ari M. Melnick, MD

Blood (2005) 106 (11): 1484.

https://doi.org/10.1182/blood.V106.11.1484.1484

Published: 2005

Journal Articles

BCL6 Attenuates DNA Damage Sensing in Normal and Malignant B-Cells by Directly Repressing ATR.

Stella M. Ranuncolo, MD, PhD, Jose M. Polo, MS, Jamil Dierov, PhD, Rita Shaknovich, MD, Martin Carroll, MD, Ari M. Melnick, MD

Blood (2005) 106 (11): 157.

https://doi.org/10.1182/blood.V106.11.157.157

Published: 2005

Journal Articles

The BCL6 Oncoprotein Forms Distinct Transcriptional Repression Complexes on Cohorts of Target Genes Involved in Specific Cellular Functions.

Jose M. Polo, MS, Stella Maris Ranuncolo, MD/PhD, Samir Parek, MD, Jose Arcaya, Ari M. Melnick, MD

Blood (2005) 106 (11): 2615.

https://doi.org/10.1182/blood.V106.11.2615.2615

Published: 2005

Journal Articles

Specific Peptide Disruption of the Bcl-6 Repression Complex Reveals Its Transcriptional Mechanism of Action in Normal and Malignant B-Cells and Is a Novel Therapeutic Approach for Diffuse Large B-Cell Lymphoma.

Jose M. Polo, MS, Tania Dell’Oso, PhD, Stella Maris Ranuncolo, MD, PhD, Leandro Cerchietti, MD, Gustavo F. Da Silva, Pharm.D, Gilvert G. Prive, PhD, Jonathan D. Licht, MD, Ari Melnick, MD

Blood (2004) 104 (11): 5.

https://doi.org/10.1182/blood.V104.11.5.5

Published: 2004

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BCL6 modulates tonic BCR signaling in diffuse large B-cell lymphomas by repressing the SYK phosphatase, PTPROt

BCL6 Is Critical for the Development of a Diverse Primary B Cell Repertoire.

The BCL6 transcriptional program features repression of multiple oncogenes in primary B cells and is deregulated in DLBCL

A peptomimetic inhibitor of BCL6 with potent antilymphoma effects in vitro and in vivo

BCL6 Regulates Tonic BCR Signaling in Diffuse Large B-Cell Lymphomas by Repressing the SYK Phosphatase, PTPROt

Reversible disruption of BCL6 repression complexes by CD40 signaling in normal and malignant B cells

BCL6 Transcriptionally Reprograms the DLBCL Genome by Forming Distinct Biochemically Active Complexes on Genes Corresponding to Different Pathways.

Design and Development of Small Molecules for Specific Targeted Therapy of Diffuse Large B-Cell Lymphoma.

A “Survival of the Fittest” Mechanism for Weeding Out Potentially Lymphomagenic B-Cells during Germinal Center B-Cell Differentiation.

BCL6 programs lymphoma cells for survival and differentiation through distinct biochemical mechanisms

A Critical BCL6-Related Feedback Loop Explains the Unusual Biological Features of Germinal Center B-Cells and Their Malignant Transformation into B-Cell Lymphomas.

A BCL6 Target Gene Signature Predicts the Biological Behavior and Classification of Diffuse Large B-Cell Lymphoma.

BCL6, p53 and Molecular Targeted Therapy in B-Cell Lymphomas.

BCL6 Attenuates DNA Damage Sensing in Normal and Malignant B-Cells by Directly Repressing ATR.

The BCL6 Oncoprotein Forms Distinct Transcriptional Repression Complexes on Cohorts of Target Genes Involved in Specific Cellular Functions.

Specific Peptide Disruption of the Bcl-6 Repression Complex Reveals Its Transcriptional Mechanism of Action in Normal and Malignant B-Cells and Is a Novel Therapeutic Approach for Diffuse Large B-Cell Lymphoma.

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