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Journal Articles

Thrombosis Caused By a Novel Coagulation Factor IX Mutation (FIX Shanghai II)

Wenman WU, Rui Wang, Qin Xu, Guanzheng Wang, Shifeng Jiang, Can Lou, Xi Wu, Yanyan Shao, Yeling Lu, Yu Liu, Guanquan Xu, Qiulan Ding, Xiaobo Hu, Xuefeng Wang, Jing Dai

Blood (2023) 142 (Supplement 1): 1233.

https://doi.org/10.1182/blood-2023-184596

Published: 2023

Journal Articles

F9 mutations causing deletions beyond the serine protease domain confer higher risk for inhibitor development in hemophilia B

Guoqing Liu, Jie Sun, Zekun Li, Zhenping Chen, Wenman Wu, Runhui Wu

Blood (2023) 141 (6): 677–680.

https://doi.org/10.1182/blood.2022017871

Published: 2023

Journal Articles

Single-Cell Sequencing of Immune Cells from Hemophilia a Patients with Inhibitors and Acquired Hemophilia a Patients

Enhao Li, Zekun Li, Jing Dai, Guoqing Liu, Jie Sun, Zhenping Chen, Runhui Wu, Xuefeng Wang, Wenman WU

Blood (2022) 140 (Supplement 1): 8428–8429.

https://doi.org/10.1182/blood-2022-169345

Published: 2022

Journal Articles

The Experimental Gene Therapy on Type 2A/Iic VWD Mouse Model with AAV Vector Delivering Only VWF Propeptide Expression cassette4

Qian Liang, Ziqi Zhang, Xuefeng Wang, Wenman WU

Blood (2022) 140 (Supplement 1): 7782–7783.

https://doi.org/10.1182/blood-2022-169113

Published: 2022

Journal Articles

Structural basis of von Willebrand factor multimerization and tubular storage

Jianwei Zeng, Zimei Shu, Qian Liang, Jing Zhang, Wenman Wu, Xuefeng Wang, Aiwu Zhou

Blood (2022) 139 (22): 3314–3324.

https://doi.org/10.1182/blood.2021014729

Published: 2022

Includes: Multimedia, Supplemental data

Journal Articles

Low factor V level ameliorates bleeding diathesis in patients with combined deficiency of factor V and factor VIII

Yanyan Shao, Wenman Wu, Guanqun Xu, Xuefeng Wang, Qiulan Ding

Blood (2019) 134 (20): 1745–1754.

https://doi.org/10.1182/blood.2018886069

Published: 2019

Includes: Supplemental data

Journal Articles

Prothrombin R541W Mutation Impairs Protein C Activation and Constitutes a New Genetic Risk Factor for Venous Thrombosis

XI WU, M.S., Dai Jing, Fang Li, PhD, Qin Xu, PhD, Changming Chen, Qiulan Ding, Xuefeng Wang, MD, Wenman WU, MD

Blood (2019) 134 (Supplement_1): 442.

https://doi.org/10.1182/blood-2019-127694

Published: 2019

Journal Articles

Decreased Factor V Ameliorates Bleeding Diathesis in Patients with Combined Deficiency of Factor V and Factor VIII

Yanyan Shao, PhD, Wenman WU, MD PhD, Guanqun Xu, B.S, Wang Xuefeng, Ding Qiulan

Blood (2018) 132 (Supplement 1): 2503.

https://doi.org/10.1182/blood-2018-99-118350

Published: 2018

Journal Articles

The kunitz protease inhibitor domain of protease nexin-2 inhibits factor XIa and murine carotid artery and middle cerebral artery thrombosis

Wenman Wu, Hongbo Li, Duraiswamy Navaneetham, Zachary W. Reichenbach, Ronald F. Tuma, Peter N. Walsh

Blood (2012) 120 (3): 671–677.

https://doi.org/10.1182/blood-2012-03-419523

Published: 2012

Journal Articles

Conformational Changes Facilitate FXI Autoactivation to FXIa

Wenman Wu, MD, PhD, Heinrich Roder, PhD, Peter N. Walsh, MD, PhD

Blood (2010) 116 (21): 19.

https://doi.org/10.1182/blood.V116.21.19.19

Published: 2010

Journal Articles

Structural Determinants for Heparin Binding in Human Coagulation Factor XI.

Sergei Shikov, MS, Wenman Wu, MD, PhD, Peter N. Walsh, MD, PhD

Blood (2007) 110 (11): 2705.

https://doi.org/10.1182/blood.V110.11.2705.2705

Published: 2007

Journal Articles

Factor XI Homodimer Formation Is Mediated by Electrostatic (Lys331 with Glu287) and Hydrophobic (Ile290 with Leu284 and Tyr329 with Tyr329) Interactions in the Apple 4 Domain and Is Essential for Normal Proteolytic Activation of Factor XI by Factor XIIa, Thrombin and Factor XIa.

Wenman Wu, MD, PhD, Dipali Sinha, PhD, James D. Lear, PhD, Paul C. Billings, PhD, Peter N. Walsh, MD, PhD

Blood (2007) 110 (11): 2706.

https://doi.org/10.1182/blood.V110.11.2706.2706

Published: 2007

Journal Articles

Dominant factor XI deficiency caused by mutations in the factor XI catalytic domain

Dmitri V. Kravtsov, Wenman Wu, Joost C. M. Meijers, Mao-Fu Sun, Morey A. Blinder, Thao P. Dang, Hongli Wang, David Gailani

Blood (2004) 104 (1): 128–134.

https://doi.org/10.1182/blood-2003-10-3530

Published: 2004

NARROW

Thrombosis Caused By a Novel Coagulation Factor IX Mutation (FIX Shanghai II)

F9 mutations causing deletions beyond the serine protease domain confer higher risk for inhibitor development in hemophilia B

Single-Cell Sequencing of Immune Cells from Hemophilia a Patients with Inhibitors and Acquired Hemophilia a Patients

The Experimental Gene Therapy on Type 2A/Iic VWD Mouse Model with AAV Vector Delivering Only VWF Propeptide Expression cassette4

Structural basis of von Willebrand factor multimerization and tubular storage

Low factor V level ameliorates bleeding diathesis in patients with combined deficiency of factor V and factor VIII

Prothrombin R541W Mutation Impairs Protein C Activation and Constitutes a New Genetic Risk Factor for Venous Thrombosis

Decreased Factor V Ameliorates Bleeding Diathesis in Patients with Combined Deficiency of Factor V and Factor VIII

The kunitz protease inhibitor domain of protease nexin-2 inhibits factor XIa and murine carotid artery and middle cerebral artery thrombosis

Conformational Changes Facilitate FXI Autoactivation to FXIa

Structural Determinants for Heparin Binding in Human Coagulation Factor XI.

Factor XI Homodimer Formation Is Mediated by Electrostatic (Lys331 with Glu287) and Hydrophobic (Ile290 with Leu284 and Tyr329 with Tyr329) Interactions in the Apple 4 Domain and Is Essential for Normal Proteolytic Activation of Factor XI by Factor XIIa, Thrombin and Factor XIa.

Dominant factor XI deficiency caused by mutations in the factor XI catalytic domain

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American Society of Hematology

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American Society of Hematology

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