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Journal Articles

Jun N-terminal kinase promotes proliferation of immature erythroid cells and erythropoietin-dependent cell lines

Sarah M. Jacobs-Helber, Stephen T. Sawyer

Blood (2004) 104 (3): 696–703.

https://doi.org/10.1182/blood-2003-05-1754

Published: 2004

Journal Articles

Tumor necrosis factor-alpha expressed constitutively in erythroid cells or induced by erythropoietin has negative and stimulatory roles in normal erythropoiesis and erythroleukemia

Sarah M. Jacobs-Helber, Kwan-ho Roh, Daniel Bailey, Emmanuel N. Dessypris, John J. Ryan, Jingchun Chen, Amittha Wickrema, Dwayne L. Barber, Paul Dent, Stephen T. Sawyer

Blood (2003) 101 (2): 524–531.

https://doi.org/10.1182/blood-2001-11-0084

Published: 2003

Journal Articles

Phosphatase inhibition promotes antiapoptotic but not proliferative signaling pathways in erythropoietin-dependent HCD57 cells

Amy E. Lawson, Haifeng Bao, Amittha Wickrema, Sarah M. Jacobs-Helber, Stephen T. Sawyer

Blood (2000) 96 (6): 2084–2092.

https://doi.org/10.1182/blood.V96.6.2084

Published: 2000

Journal Articles

JNK and p38 are activated by erythropoietin (EPO) but are not induced in apoptosis following EPO withdrawal in EPO-dependent HCD57 cells

Sarah M. Jacobs-Helber, John J. Ryan, Stephen T. Sawyer

Blood (2000) 96 (3): 933–940.

https://doi.org/10.1182/blood.V96.3.933

Published: 2000

Journal Articles

Protein Kinase B (c-Akt), Phosphatidylinositol 3-Kinase, and STAT5 Are Activated by Erythropoietin (EPO) in HCD57 Erythroid Cells But Are Constitutively Active in an EPO-Independent, Apoptosis-Resistant Subclone (HCD57-SREI Cells)

Haifeng Bao, Sarah M. Jacobs-Helber, Amy E. Lawson, Kalyani Penta, Amittha Wickrema, Stephen T. Sawyer

Blood (1999) 93 (11): 3757–3773.

https://doi.org/10.1182/blood.V93.11.3757

Published: 1999

Journal Articles

Polycythemia Vera V. Enhanced Proliferation and Phosphorylation Due to Vanadate Are Diminished in Polycythemia Vera Erythroid Progenitor Cells: A Possible Defect of Phosphatase Activity in Polycythemia Vera

Chun-Hua Dai, Sanford B. Krantz, Stephen T. Sawyer

Blood (1997) 89 (10): 3574–3581.

https://doi.org/10.1182/blood.V89.10.3574

Published: 1997

NARROW

Jun N-terminal kinase promotes proliferation of immature erythroid cells and erythropoietin-dependent cell lines

Tumor necrosis factor-alpha expressed constitutively in erythroid cells or induced by erythropoietin has negative and stimulatory roles in normal erythropoiesis and erythroleukemia

Phosphatase inhibition promotes antiapoptotic but not proliferative signaling pathways in erythropoietin-dependent HCD57 cells

JNK and p38 are activated by erythropoietin (EPO) but are not induced in apoptosis following EPO withdrawal in EPO-dependent HCD57 cells

Protein Kinase B (c-Akt), Phosphatidylinositol 3-Kinase, and STAT5 Are Activated by Erythropoietin (EPO) in HCD57 Erythroid Cells But Are Constitutively Active in an EPO-Independent, Apoptosis-Resistant Subclone (HCD57-SREI Cells)

Polycythemia Vera V. Enhanced Proliferation and Phosphorylation Due to Vanadate Are Diminished in Polycythemia Vera Erythroid Progenitor Cells: A Possible Defect of Phosphatase Activity in Polycythemia Vera

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ASH Publications

American Society of Hematology

Jun N-terminal kinase promotes proliferation of immature erythroid cells and erythropoietin-dependent cell lines

Tumor necrosis factor-alpha expressed constitutively in erythroid cells or induced by erythropoietin has negative and stimulatory roles in normal erythropoiesis and erythroleukemia

Phosphatase inhibition promotes antiapoptotic but not proliferative signaling pathways in erythropoietin-dependent HCD57 cells

JNK and p38 are activated by erythropoietin (EPO) but are not induced in apoptosis following EPO withdrawal in EPO-dependent HCD57 cells

Protein Kinase B (c-Akt), Phosphatidylinositol 3-Kinase, and STAT5 Are Activated by Erythropoietin (EPO) in HCD57 Erythroid Cells But Are Constitutively Active in an EPO-Independent, Apoptosis-Resistant Subclone (HCD57-SREI Cells)

Polycythemia Vera V. Enhanced Proliferation and Phosphorylation Due to Vanadate Are Diminished in Polycythemia Vera Erythroid Progenitor Cells: A Possible Defect of Phosphatase Activity in Polycythemia Vera

Email alerts

ASH Publications

American Society of Hematology

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