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Journal Articles

Aberrant Intracellular Retention of Mutated Receptor Tyrosine Kinase FLT3 in Human Leukemic Cells as a Potential Mechanism Supporting the Pathogenesis of Acute Myeloid Leukaemia.

Sina Koch, Martin Ryser, Angela Jacobi, Sebastian Brenner, Martin Bornhaeuser, Gerhard Ehninger, Christian Thiede

Blood (2008) 112 (11): 2245.

https://doi.org/10.1182/blood.V112.11.2245.2245

Published: 2008

Journal Articles

Expression and Functional Analysis of Autotaxin, a Relevant Motility and Survival Factor, in FLT3-ITD Positive Acute Myeloid Leukemia and Primary Hematopoietic Stem Cells.

Claudia Ortlepp, Christine Steudel, Sina Koch, Angela Jacobi, Satu Kyttaelae, Martin Ryser, Sebastian Brenner, Martin Bornhaeuser, Gerhard Ehninger, Christian Thiede

Blood (2008) 112 (11): 1206.

https://doi.org/10.1182/blood.V112.11.1206.1206

Published: 2008

Journal Articles

Differences in the Localization of Receptor Tyrosine Kinase FLT3 in Human Hematopoietic Stem and Progenitor Cells and Leukemic Blasts.

Sina Koch, Martin Ryser, MD, Martin Bornhaeuser, Gerhard Ehninger, Christian Thiede

Blood (2006) 108 (11): 1910.

https://doi.org/10.1182/blood.V108.11.1910.1910

Published: 2006

Journal Articles

High Expression of ENPP2/Autotaxin in Patients with FLT3-ITD Positive Acute Myeloid Leukemia.

Claudia Ortlepp, Christine Steudel, PhD, Sina Koch, Martin Ryser, MD, Gerhard Ehninger, Christian Thiede

Blood (2006) 108 (11): 1911.

https://doi.org/10.1182/blood.V108.11.1911.1911

Published: 2006

Journal Articles

Haploinsufficiency, rather than the effect of an excessive production of soluble CD95 (CD95ΔTM), is the basis for ALPS Ia in a family with duplicated 3′ splice site AG in CD95 intron 5 on one allele

Joachim Roesler, Jose-Maria Izquierdo, Martin Ryser, Angela Rösen-Wolff, Manfred Gahr, Juan Valcarcel, Michael J. Lenardo, Lixin Zheng

Blood (2005) 106 (5): 1652–1659.

https://doi.org/10.1182/blood-2004-08-3104

Published: 2005

Includes: Supplemental data

NARROW

Aberrant Intracellular Retention of Mutated Receptor Tyrosine Kinase FLT3 in Human Leukemic Cells as a Potential Mechanism Supporting the Pathogenesis of Acute Myeloid Leukaemia.

Expression and Functional Analysis of Autotaxin, a Relevant Motility and Survival Factor, in FLT3-ITD Positive Acute Myeloid Leukemia and Primary Hematopoietic Stem Cells.

Differences in the Localization of Receptor Tyrosine Kinase FLT3 in Human Hematopoietic Stem and Progenitor Cells and Leukemic Blasts.

High Expression of ENPP2/Autotaxin in Patients with FLT3-ITD Positive Acute Myeloid Leukemia.

Haploinsufficiency, rather than the effect of an excessive production of soluble CD95 (CD95ΔTM), is the basis for ALPS Ia in a family with duplicated 3′ splice site AG in CD95 intron 5 on one allele

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ASH Publications

American Society of Hematology

Aberrant Intracellular Retention of Mutated Receptor Tyrosine Kinase FLT3 in Human Leukemic Cells as a Potential Mechanism Supporting the Pathogenesis of Acute Myeloid Leukaemia.

Expression and Functional Analysis of Autotaxin, a Relevant Motility and Survival Factor, in FLT3-ITD Positive Acute Myeloid Leukemia and Primary Hematopoietic Stem Cells.

Differences in the Localization of Receptor Tyrosine Kinase FLT3 in Human Hematopoietic Stem and Progenitor Cells and Leukemic Blasts.

High Expression of ENPP2/Autotaxin in Patients with FLT3-ITD Positive Acute Myeloid Leukemia.

Haploinsufficiency, rather than the effect of an excessive production of soluble CD95 (CD95ΔTM), is the basis for ALPS Ia in a family with duplicated 3′ splice site AG in CD95 intron 5 on one allele

Email alerts

ASH Publications

American Society of Hematology

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