Page 1 | Search Results | Blood | American Society of Hematology

1-6 of 6

Sort by

Sort Order Select

Journal Articles

Recurrent genetic fusions redefine MLL germ line acute lymphoblastic leukemia in infants

Blood (2021) 137 (14): 1980–1984.

https://doi.org/10.1182/blood.2020009032

Published: 2021

Includes: Supplemental data

Journal Articles

Monozygotic Twins with Childhood ALL Demonstrate Prenatal Origin of the Philadelphia Chromosome and Post-Natal Ikaros Deletion.

Giovanni Cazzaniga, Luca Lo Nigro, Joannah Score, Silvia Bungaro, Fred van Delft, Ilaria Iacobucci, Lilia Corral, Elena Mirabile, Anthony Ford, Giovanni Martinelli, Nicholas C.P. Cross, Mel Greaves, Andrea Biondi

Blood (2009) 114 (22): 87.

https://doi.org/10.1182/blood.V114.22.87.87

Published: 2009

Journal Articles

In Infant ALL with t(4;11)/ MLL-AF4 Multiple and Phenotypically Distinct CD19+ BM Subsets Initiate Leukemia in NOD/SCID Mice..

Michela Bardini, Lilian Wittmann, Lilia Corral, Luca Lo Nigro, Zhi Ma, Rakesh Singh, Giuseppe Basso, Giulio De Rossi, Mel Greaves, Andrea Biondi, Giovanni Cazzaniga, Sten Eirik Jacobsen

Blood (2009) 114 (22): 1432.

https://doi.org/10.1182/blood.V114.22.1432.1432

Published: 2009

Journal Articles

Infant ALL Patients Carrying t(4;11) Have a Different Genotypic Profile Than Older ALL Children.

Michela Bardini, PhD,Fellow, Lilia Corral, Eleonora Mangano, PhD, fellow, Roberta Spinelli, Grazia Fazio, Ingrid Cifola, PhD, Andrea Biondi, MD, Cristina Battaglia, Giovanni Cazzaniga, PhD

Blood (2007) 110 (11): 1436.

https://doi.org/10.1182/blood.V110.11.1436.1436

Published: 2007

Journal Articles

PTPN11 and RAS Gene Mutation Pattern Identifies an Unique Feature of Upregulated RAS Function in Infant ALL.

Giovanni Cazzaniga, PhD, Simone Martinelli, PhD, Monique L. den Boer, PhD, Lilia Corral, BSc, Monica Spinelli, PhD, Giuseppe Basso, MD, Giulio De Rossi, MD, Rob Pieters, MD, PhD, Marco Tartaglia, MD, PhD, Andrea Biondi, MD

Blood (2004) 104 (11): 996.

https://doi.org/10.1182/blood.V104.11.996.996

Published: 2004

Journal Articles

High Clonal Evolution Rate in Relapsing Infant ALL Patients.

Giovanni Cazzaniga, PhD, Lilia Corral, BSc, Matteo Luciani, MD, Simona Songia, Bsc, Paola De Lorenzo, PhD, Monica Spinelli, PhD, Luca Lo Nigro, MD, Giuseppe Basso, MD, Giulio De Rossi, MD, Andrea Biondi, MD

Blood (2004) 104 (11): 164.

https://doi.org/10.1182/blood.V104.11.164.164

Published: 2004

NARROW

Recurrent genetic fusions redefine MLL germ line acute lymphoblastic leukemia in infants

Monozygotic Twins with Childhood ALL Demonstrate Prenatal Origin of the Philadelphia Chromosome and Post-Natal Ikaros Deletion.

In Infant ALL with t(4;11)/ MLL-AF4 Multiple and Phenotypically Distinct CD19+ BM Subsets Initiate Leukemia in NOD/SCID Mice..

Infant ALL Patients Carrying t(4;11) Have a Different Genotypic Profile Than Older ALL Children.

PTPN11 and RAS Gene Mutation Pattern Identifies an Unique Feature of Upregulated RAS Function in Infant ALL.

High Clonal Evolution Rate in Relapsing Infant ALL Patients.

Email alerts

ASH Publications

American Society of Hematology

Recurrent genetic fusions redefine MLL germ line acute lymphoblastic leukemia in infants

Monozygotic Twins with Childhood ALL Demonstrate Prenatal Origin of the Philadelphia Chromosome and Post-Natal Ikaros Deletion.

In Infant ALL with t(4;11)/ MLL-AF4 Multiple and Phenotypically Distinct CD19+ BM Subsets Initiate Leukemia in NOD/SCID Mice..

Infant ALL Patients Carrying t(4;11) Have a Different Genotypic Profile Than Older ALL Children.

PTPN11 and RAS Gene Mutation Pattern Identifies an Unique Feature of Upregulated RAS Function in Infant ALL.

High Clonal Evolution Rate in Relapsing Infant ALL Patients.

Email alerts

ASH Publications

American Society of Hematology

This Feature Is Available To Subscribers Only