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Journal Articles

Occupancy of human EPCR by protein C induces β-arrestin-2 biased PAR1 signaling by both APC and thrombin

Ram Vinod Roy, Abdolreza Ardeshirylajimi, Peyman Dinarvand, Likui Yang, Alireza R. Rezaie

Blood (2016) 128 (14): 1884–1893.

https://doi.org/10.1182/blood-2016-06-720581

Published: 2016

Includes: Supplemental data

Journal Articles

Protein C Thr315Ala variant results in gain of function but manifests as type II deficiency in diagnostic assays

Qiulan Ding, Likui Yang, Peyman Dinarvand, Xuefeng Wang, Alireza R. Rezaie

Blood (2015) 125 (15): 2428–2434.

https://doi.org/10.1182/blood-2014-12-617274

Published: 2015

Includes: Supplemental data

Journal Articles

Polyphosphate amplifies proinflammatory responses of nuclear proteins through interaction with receptor for advanced glycation end products and P2Y 1 purinergic receptor

Peyman Dinarvand, Seyed Mahdi Hassanian, Shabir H. Qureshi, Chandrashekhara Manithody, Joel C. Eissenberg, Likui Yang, Alireza R. Rezaie

Blood (2014) 123 (6): 935–945.

https://doi.org/10.1182/blood-2013-09-529602

Published: 2014

Includes: Supplemental data

Journal Articles

Factor X/Xa Elicits Protective Signaling Responses In Endothelial Cells Directly Via PAR-2 and Indirectly Via EPCR-Dependent Recruitment of PAR-1

Alireza R. Rezaie, PhD, Jong-Sup Bae, Ph.D., Likui Yang, Ph.D.

Blood (2010) 116 (21): 2115.

https://doi.org/10.1182/blood.V116.21.2115.2115

Published: 2010

Journal Articles

Characterization of Heparin Binding Site Residues on the Catalytic Domain of Factor XIa

Likui Yang, Ph.D., Mao-fu Sun, Chandrashekhara Manithody, Ph.D., David Gailani, MD, Alireza R. Rezaie, PhD

Blood (2008) 112 (11): 1018.

https://doi.org/10.1182/blood.V112.11.1018.1018

Published: 2008

Journal Articles

The ligand occupancy of endothelial protein C receptor switches the protease-activated receptor 1-dependent signaling specificity of thrombin from a permeability-enhancing to a barrier-protective response in endothelial cells

Jong-Sup Bae, Likui Yang, Chandrashekhara Manithody, Alireza R. Rezaie

Blood (2007) 110 (12): 3909–3916.

https://doi.org/10.1182/blood-2007-06-096651

Published: 2007

Journal Articles

The Ligand Occupancy of Endothelial Protein C Receptor Switches the PAR-1 Dependent Signaling Specificity of Thrombin from a Disruptive to a Protective Response in Endothelial Cells.

Alireza R. Rezaie, PhD, Jong-Sup Bae, MS, Likui Yang, Ph.D., Chandrashekhara Manithody, Ph.D.

Blood (2007) 110 (11): 1746.

https://doi.org/10.1182/blood.V110.11.1746.1746

Published: 2007

Journal Articles

Engineering a Disulfide Bond To Stabilize the Calcium Binding Loop of Activated Protein C Eliminates Its Anticoagulant but Not Protective Signaling Properties.

Jong-Sup Bae, MS, Likui Yang, Ph.D., Chandrashekhara Manithody, Ph.D., Alireza R. Rezaie, Ph.D.

Blood (2006) 108 (11): 62.

https://doi.org/10.1182/blood.V108.11.62.62

Published: 2006

Journal Articles

Heparin-activated antithrombin interacts with the autolysis loop of target coagulation proteases

Likui Yang, Chandrashekhara Manithody, Alireza R. Rezaie

Blood (2004) 104 (6): 1753–1759.

https://doi.org/10.1182/blood-2004-03-1092

Published: 2004

NARROW

Occupancy of human EPCR by protein C induces β-arrestin-2 biased PAR1 signaling by both APC and thrombin

Protein C Thr315Ala variant results in gain of function but manifests as type II deficiency in diagnostic assays

Polyphosphate amplifies proinflammatory responses of nuclear proteins through interaction with receptor for advanced glycation end products and P2Y 1 purinergic receptor

Factor X/Xa Elicits Protective Signaling Responses In Endothelial Cells Directly Via PAR-2 and Indirectly Via EPCR-Dependent Recruitment of PAR-1

Characterization of Heparin Binding Site Residues on the Catalytic Domain of Factor XIa

The ligand occupancy of endothelial protein C receptor switches the protease-activated receptor 1-dependent signaling specificity of thrombin from a permeability-enhancing to a barrier-protective response in endothelial cells

The Ligand Occupancy of Endothelial Protein C Receptor Switches the PAR-1 Dependent Signaling Specificity of Thrombin from a Disruptive to a Protective Response in Endothelial Cells.

Engineering a Disulfide Bond To Stabilize the Calcium Binding Loop of Activated Protein C Eliminates Its Anticoagulant but Not Protective Signaling Properties.

Heparin-activated antithrombin interacts with the autolysis loop of target coagulation proteases

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American Society of Hematology

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ASH Publications

American Society of Hematology

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