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Journal Articles

Polyphosphate amplifies proinflammatory responses of nuclear proteins through interaction with receptor for advanced glycation end products and P2Y 1 purinergic receptor

Peyman Dinarvand, Seyed Mahdi Hassanian, Shabir H. Qureshi, Chandrashekhara Manithody, Joel C. Eissenberg, Likui Yang, Alireza R. Rezaie

Blood (2014) 123 (6): 935–945.

https://doi.org/10.1182/blood-2013-09-529602

Published: 2014

Includes: Supplemental data

Journal Articles

Characterization of Heparin Binding Site Residues on the Catalytic Domain of Factor XIa

Likui Yang, Ph.D., Mao-fu Sun, Chandrashekhara Manithody, Ph.D., David Gailani, MD, Alireza R. Rezaie, PhD

Blood (2008) 112 (11): 1018.

https://doi.org/10.1182/blood.V112.11.1018.1018

Published: 2008

Journal Articles

The ligand occupancy of endothelial protein C receptor switches the protease-activated receptor 1-dependent signaling specificity of thrombin from a permeability-enhancing to a barrier-protective response in endothelial cells

Jong-Sup Bae, Likui Yang, Chandrashekhara Manithody, Alireza R. Rezaie

Blood (2007) 110 (12): 3909–3916.

https://doi.org/10.1182/blood-2007-06-096651

Published: 2007

Journal Articles

The Ligand Occupancy of Endothelial Protein C Receptor Switches the PAR-1 Dependent Signaling Specificity of Thrombin from a Disruptive to a Protective Response in Endothelial Cells.

Alireza R. Rezaie, PhD, Jong-Sup Bae, MS, Likui Yang, Ph.D., Chandrashekhara Manithody, Ph.D.

Blood (2007) 110 (11): 1746.

https://doi.org/10.1182/blood.V110.11.1746.1746

Published: 2007

Journal Articles

Engineering a Disulfide Bond To Stabilize the Calcium Binding Loop of Activated Protein C Eliminates Its Anticoagulant but Not Protective Signaling Properties.

Jong-Sup Bae, MS, Likui Yang, Ph.D., Chandrashekhara Manithody, Ph.D., Alireza R. Rezaie, Ph.D.

Blood (2006) 108 (11): 62.

https://doi.org/10.1182/blood.V108.11.62.62

Published: 2006

Journal Articles

Heparin-activated antithrombin interacts with the autolysis loop of target coagulation proteases

Likui Yang, Chandrashekhara Manithody, Alireza R. Rezaie

Blood (2004) 104 (6): 1753–1759.

https://doi.org/10.1182/blood-2004-03-1092

Published: 2004

Journal Articles

Exosite-dependent regulation of factor VIIIa by activated protein C

Chandrashekhara Manithody, Philip J. Fay, Alireza R. Rezaie

Blood (2003) 101 (12): 4802–4807.

https://doi.org/10.1182/blood-2003-01-0126

Published: 2003

NARROW

Polyphosphate amplifies proinflammatory responses of nuclear proteins through interaction with receptor for advanced glycation end products and P2Y 1 purinergic receptor

Characterization of Heparin Binding Site Residues on the Catalytic Domain of Factor XIa

The ligand occupancy of endothelial protein C receptor switches the protease-activated receptor 1-dependent signaling specificity of thrombin from a permeability-enhancing to a barrier-protective response in endothelial cells

The Ligand Occupancy of Endothelial Protein C Receptor Switches the PAR-1 Dependent Signaling Specificity of Thrombin from a Disruptive to a Protective Response in Endothelial Cells.

Engineering a Disulfide Bond To Stabilize the Calcium Binding Loop of Activated Protein C Eliminates Its Anticoagulant but Not Protective Signaling Properties.

Heparin-activated antithrombin interacts with the autolysis loop of target coagulation proteases

Exosite-dependent regulation of factor VIIIa by activated protein C

Email alerts

ASH Publications

American Society of Hematology

Polyphosphate amplifies proinflammatory responses of nuclear proteins through interaction with receptor for advanced glycation end products and P2Y 1 purinergic receptor

Characterization of Heparin Binding Site Residues on the Catalytic Domain of Factor XIa

The ligand occupancy of endothelial protein C receptor switches the protease-activated receptor 1-dependent signaling specificity of thrombin from a permeability-enhancing to a barrier-protective response in endothelial cells

The Ligand Occupancy of Endothelial Protein C Receptor Switches the PAR-1 Dependent Signaling Specificity of Thrombin from a Disruptive to a Protective Response in Endothelial Cells.

Engineering a Disulfide Bond To Stabilize the Calcium Binding Loop of Activated Protein C Eliminates Its Anticoagulant but Not Protective Signaling Properties.

Heparin-activated antithrombin interacts with the autolysis loop of target coagulation proteases

Exosite-dependent regulation of factor VIIIa by activated protein C

Email alerts

ASH Publications

American Society of Hematology

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