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Journal Articles

Soluble Inflammatory Mediators Proteinase-3 and Neutrophil Elastase Are Targeted by PR1-Specific Immunotherapy After Cellular Uptake and Cross Presentation of PR1 Peptide by Breast Cancer Cells

Gheath Alatrash, DO, PhD, Elizabeth Mittendorf, MD, Anna Sergeeva, PhD, Pariya Sukhumalchandra, BS, Na Qiao, PhD, Kathryn Quintanilla, MS, Mao Zhang, MPH, Lu Sijie, PhD, Karen Clise-Dwyer, PhD, Jeffrey J. Molldrem, MD

Blood (2010) 116 (21): 2089.

https://doi.org/10.1182/blood.V116.21.2089.2089

Published: 2010

Journal Articles

Cellular Uptake of Soluble Neutrophil Elastase Increases Cyclin E (CCNE) Isoform Expression and Significantly Augments Susceptibility of Breast Cancer Cells to Cytolysis by CCNE-Specific Cytotoxic T Lymphocytes

Elizabeth Mittendorf, M.D., Gheath Alatrash, Na Qiao, Pariya Sukhumalchandra, Sijie Lu, Kathryn Quintanilla, Karen Clise-Dwyer, PhD, Jeffrey J. Molldrem, MD

Blood (2010) 116 (21): 2090.

https://doi.org/10.1182/blood.V116.21.2090.2090

Published: 2010

Journal Articles

Proteinase 3 Mediates Enzyme-Independent Reversible Inhibition of CD4 and CD8 T Cell Proliferation by Blocking G 1 -S Transition

Tian-Hui Yang, MD., PhD, Gheath Alatrash, DO, PhD, Karen Clise-Dwyer, PhD, Kathryn Quintanilla, MS, Sijie Lu, PhD, Jeffrey J. Molldrem, MD

Blood (2010) 116 (21): 2099.

https://doi.org/10.1182/blood.V116.21.2099.2099

Published: 2010

Journal Articles

Peripheral Tolerance to PR1 Is Maintained After Uptake of Soluble Proteinas-3 and Neutrophil Elastase

Gheath Alatrash, DO, PhD, Yoko Ono, MD, Anna Sergeeva, PhD, Pariya Sukhumalchandra, BS, Mao Zhang, MPH, Kathryn Quintanilla, MS, Paul Armistead, MD, PhD, Hong He, MD, Tania Rodriguez-Cruz, BS, Karen Clise-Dwyer, PhD, Eric Wieder, PhD, Greg Lizee, PhD, Sijie Lu, PhD, Jeffrey J. Molldrem, MD

Blood (2010) 116 (21): 4301.

https://doi.org/10.1182/blood.V116.21.4301.4301

Published: 2010

Journal Articles

Direct Visualization of PR1/HLA-A2 on the Membrane of HLAA2+ CD13+CD33+ Myeloid Leukemia Blasts by a Novel Monoclonal Antibody

Anna Sergeeva, Sijie Lu, James N Wygant, Gheath Alatrash, Kathryn Quintanilla, Jeffrey J. Molldrem, MD

Blood (2008) 112 (11): 2545.

https://doi.org/10.1182/blood.V112.11.2545.2545

Published: 2008

NARROW

Soluble Inflammatory Mediators Proteinase-3 and Neutrophil Elastase Are Targeted by PR1-Specific Immunotherapy After Cellular Uptake and Cross Presentation of PR1 Peptide by Breast Cancer Cells

Cellular Uptake of Soluble Neutrophil Elastase Increases Cyclin E (CCNE) Isoform Expression and Significantly Augments Susceptibility of Breast Cancer Cells to Cytolysis by CCNE-Specific Cytotoxic T Lymphocytes

Proteinase 3 Mediates Enzyme-Independent Reversible Inhibition of CD4 and CD8 T Cell Proliferation by Blocking G 1 -S Transition

Peripheral Tolerance to PR1 Is Maintained After Uptake of Soluble Proteinas-3 and Neutrophil Elastase

Direct Visualization of PR1/HLA-A2 on the Membrane of HLAA2+ CD13+CD33+ Myeloid Leukemia Blasts by a Novel Monoclonal Antibody

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ASH Publications

American Society of Hematology

Soluble Inflammatory Mediators Proteinase-3 and Neutrophil Elastase Are Targeted by PR1-Specific Immunotherapy After Cellular Uptake and Cross Presentation of PR1 Peptide by Breast Cancer Cells

Cellular Uptake of Soluble Neutrophil Elastase Increases Cyclin E (CCNE) Isoform Expression and Significantly Augments Susceptibility of Breast Cancer Cells to Cytolysis by CCNE-Specific Cytotoxic T Lymphocytes

Proteinase 3 Mediates Enzyme-Independent Reversible Inhibition of CD4 and CD8 T Cell Proliferation by Blocking G 1 -S Transition

Peripheral Tolerance to PR1 Is Maintained After Uptake of Soluble Proteinas-3 and Neutrophil Elastase

Direct Visualization of PR1/HLA-A2 on the Membrane of HLAA2+ CD13+CD33+ Myeloid Leukemia Blasts by a Novel Monoclonal Antibody

Email alerts

ASH Publications

American Society of Hematology

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