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Journal Articles

The HDAC inhibitors trichostatin A and suberoylanilide hydroxamic acid exhibit multiple modalities of benefit for the vascular pathobiology of sickle transgenic mice

Robert P. Hebbel, Gregory M. Vercellotti, Betty S. Pace, Anna N. Solovey, Rahn Kollander, Chine F. Abanonu, Julia Nguyen, Julie V. Vineyard, John D. Belcher, Fuad Abdulla, Shadé Osifuye, John W. Eaton, Robert J. Kelm, Jr, Arne Slungaard

Blood (2010) 115 (12): 2483–2490.

https://doi.org/10.1182/blood-2009-02-204990

Published: 2010

Journal Articles

Heme Oxygenase-1 Gene Therapy in a Murine Model of Sickle Cell Disease.

Belcher D. Belcher, Ph.D., Julie V. Vineyard, BS, Carol M Bruzzone, MS, Chunsheng Chen, M.D./Ph.D., Joan D Beckman, BS, Julia Ngyuen, BS, Clifford J Steer, M.D., Gregory M. Vercellotti, MD

Blood (2009) 114 (22): 1527.

https://doi.org/10.1182/blood.V114.22.1527.1527

Published: 2009

Journal Articles

The HDAC Inhibitors Trichostatin A (TSA) and Suberoylanilide Hydroxamic Acid (SAHA) Exhibit Multiple Modalities of Benefit for the Vascular Pathology of Sickle Disease.

Robert P Hebbel, MD, Gregory M. Vercellotti, MD, Betty Pace, MD, Anna Solovey, M.D., Rahn Kollander, Chine Abanonu, Julia Nguyen, BS, Julie V. Vineyard, BS, John D Belcher, Ph.D., Fuad Abdulla, Shade Osifuye, John W. Eaton, PhD, Robert Kelm, MD, Arne Slungaard, MD

Blood (2009) 114 (22): 2586.

https://doi.org/10.1182/blood.V114.22.2586.2586

Published: 2009

Journal Articles

Carbon Monoxide Decreases Leukocytosis in Murine Sickle Cell Disease Models Via Decreased Granulopoiesis.

Joan D Beckman, BS, Julie V. Vineyard, BS, Chunsheng Chen, PhD, Julia Ngyuen, BS, Michael O Nwaneri, M.D., Paul Marker, BS, John D. Belcher, Ph.D., Gregory M. Vercellotti, MD

Blood (2008) 112 (11): 1433.

https://doi.org/10.1182/blood.V112.11.1433.1433

Published: 2008

Journal Articles

Inhaled Carbon Monoxide: An Anti-Inflammatory Modulator in Transgenic Sickle Mice.

Joan D. Beckman, BS, Julie V. Vineyard, BS, Casey Yang, Thomas E. Schmidt, John D. Belcher, PhD, Gregory M. Vercellotti, MD

Blood (2007) 110 (11): 2268.

https://doi.org/10.1182/blood.V110.11.2268.2268

Published: 2007

NARROW

The HDAC inhibitors trichostatin A and suberoylanilide hydroxamic acid exhibit multiple modalities of benefit for the vascular pathobiology of sickle transgenic mice

Heme Oxygenase-1 Gene Therapy in a Murine Model of Sickle Cell Disease.

The HDAC Inhibitors Trichostatin A (TSA) and Suberoylanilide Hydroxamic Acid (SAHA) Exhibit Multiple Modalities of Benefit for the Vascular Pathology of Sickle Disease.

Carbon Monoxide Decreases Leukocytosis in Murine Sickle Cell Disease Models Via Decreased Granulopoiesis.

Inhaled Carbon Monoxide: An Anti-Inflammatory Modulator in Transgenic Sickle Mice.

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ASH Publications

American Society of Hematology

The HDAC inhibitors trichostatin A and suberoylanilide hydroxamic acid exhibit multiple modalities of benefit for the vascular pathobiology of sickle transgenic mice

Heme Oxygenase-1 Gene Therapy in a Murine Model of Sickle Cell Disease.

The HDAC Inhibitors Trichostatin A (TSA) and Suberoylanilide Hydroxamic Acid (SAHA) Exhibit Multiple Modalities of Benefit for the Vascular Pathology of Sickle Disease.

Carbon Monoxide Decreases Leukocytosis in Murine Sickle Cell Disease Models Via Decreased Granulopoiesis.

Inhaled Carbon Monoxide: An Anti-Inflammatory Modulator in Transgenic Sickle Mice.

Email alerts

ASH Publications

American Society of Hematology

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