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Journal Articles

Adaptive secretion of granulocyte-macrophage colony-stimulating factor (GM-CSF) mediates imatinib and nilotinib resistance in BCR/ABL + progenitors via JAK-2/STAT-5 pathway activation

Ying Wang, Dali Cai, Cornelia Brendel, Christine Barett, Philipp Erben, Paul W. Manley, Andreas Hochhaus, Andreas Neubauer, Andreas Burchert

Blood (2006) 109 (5): 2147–2155.

https://doi.org/10.1182/blood-2006-08-040022

Published: 2006

Includes: Supplemental data

Journal Articles

Adaptive Autocrine Secretion of the Granulocyte Macrophage Colony Stimulating Factor (GM-CSF) Mediates Imatinib- and Nilotinib-Resistance in BCR/ABL-Positive Progenitors Via JAK-2/STAT-5 Pathway Activation.

Andreas Burchert, Ying Wang, Cornelia Brendel, Philipp Erben, Paul W. Manley, Andreas Hochhaus, Dali Cai, Andreas Neubauer

Blood (2006) 108 (11): 2187.

https://doi.org/10.1182/blood.V108.11.2187.2187

Published: 2006

Journal Articles

FLT3-ITD–, but not BCR/ABL-transformed cells require concurrent Akt/mTor blockage to undergo apoptosis after histone deacetylase inhibitor treatment

Dali Cai, Ying Wang, Oliver G. Ottmann, Peter J. Barth, Andreas Neubauer, Andreas Burchert

Blood (2006) 107 (5): 2094–2097.

https://doi.org/10.1182/blood-2005-08-3317

Published: 2006

Includes: Supplemental data

Journal Articles

Interferon consensus sequence binding protein (ICSBP; IRF-8) antagonizes BCR/ABL and down-regulates bcl-2

Andreas Burchert, Dali Cai, Lorenz C. Hofbauer, Magnus K. R. Samuelsson, Emily P. Slater, Justus Duyster, Markus Ritter, Andreas Hochhaus, Rolf Müller, Martin Eilers, Manuel Schmidt, Andreas Neubauer

Blood (2004) 103 (9): 3480–3489.

https://doi.org/10.1182/blood-2003-08-2970

Published: 2004

Journal Articles

Interferon-α, but not the ABL-kinase inhibitor imatinib (STI571), induces expression of myeloblastin and a specific T-cell response in chronic myeloid leukemia

Andreas Burchert, Stefan Wölfl, Manuel Schmidt, Cornelia Brendel, Barbara Denecke, Dali Cai, Larissa Odyvanova, Tanja Lahaye, Martin C. Müller, Thomas Berg, Harald Gschaidmeier, Burghardt Wittig, Rüdiger Hehlmann, Andreas Hochhaus, Andreas Neubauer

Blood (2003) 101 (1): 259–264.

https://doi.org/10.1182/blood-2002-02-0659

Published: 2003

NARROW

Adaptive secretion of granulocyte-macrophage colony-stimulating factor (GM-CSF) mediates imatinib and nilotinib resistance in BCR/ABL + progenitors via JAK-2/STAT-5 pathway activation

Adaptive Autocrine Secretion of the Granulocyte Macrophage Colony Stimulating Factor (GM-CSF) Mediates Imatinib- and Nilotinib-Resistance in BCR/ABL-Positive Progenitors Via JAK-2/STAT-5 Pathway Activation.

FLT3-ITD–, but not BCR/ABL-transformed cells require concurrent Akt/mTor blockage to undergo apoptosis after histone deacetylase inhibitor treatment

Interferon consensus sequence binding protein (ICSBP; IRF-8) antagonizes BCR/ABL and down-regulates bcl-2

Interferon-α, but not the ABL-kinase inhibitor imatinib (STI571), induces expression of myeloblastin and a specific T-cell response in chronic myeloid leukemia

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ASH Publications

American Society of Hematology

Adaptive secretion of granulocyte-macrophage colony-stimulating factor (GM-CSF) mediates imatinib and nilotinib resistance in BCR/ABL + progenitors via JAK-2/STAT-5 pathway activation

Adaptive Autocrine Secretion of the Granulocyte Macrophage Colony Stimulating Factor (GM-CSF) Mediates Imatinib- and Nilotinib-Resistance in BCR/ABL-Positive Progenitors Via JAK-2/STAT-5 Pathway Activation.

FLT3-ITD–, but not BCR/ABL-transformed cells require concurrent Akt/mTor blockage to undergo apoptosis after histone deacetylase inhibitor treatment

Interferon consensus sequence binding protein (ICSBP; IRF-8) antagonizes BCR/ABL and down-regulates bcl-2

Interferon-α, but not the ABL-kinase inhibitor imatinib (STI571), induces expression of myeloblastin and a specific T-cell response in chronic myeloid leukemia

Email alerts

ASH Publications

American Society of Hematology

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