Page 1 | Search Results | Blood | American Society of Hematology

1-6 of 6

Sort by

Sort Order Select

Journal Articles

TGFβ1 Induces MLL -Rearranged Acute Lymphoblastic Leukemia into Chemoresistant Quiescence in a Coordinated Manner with FLT3 Ligand

Minori Tamai, MD,PhD, Yoshiyuki Furuichi, MD, PhD, Shin Kasai, MD, Norie Ando, MD, Kumiko Goi, MD,PhD, Takeshi Inukai, MD, PhD, Masako Abe, Keiko Kagami, Kanji Sugita, MD, PhD

Blood (2016) 128 (22): 3972.

https://doi.org/10.1182/blood.V128.22.3972.3972

Published: 2016

Journal Articles

Chemosensitivity Is Differentially Regulated by SDF-1/CXCR4 and SDF-1/CXCR7 Axes in Acute Lymphoblastic Leukemia.

Norie Ando, Yoshiyuki Furuichi, MD, PhD, Shin Kasai, Keiko Kagami, Kumiko Goi, MD, PhD, Takeshi Inukai, MD, PhD, Kanji Sugita, MD, PhD

Blood (2012) 120 (21): 2441.

https://doi.org/10.1182/blood.V120.21.2441.2441

Published: 2012

Journal Articles

Triggering of CD44 by Ultra-Low-Molecular-Weight Hyaluronan Induces Cell Death by Autophagy Via Endoplasmic Reticulum Stress in Acute Lymphoblastic Leukemia with MLL Gene Rearrangements

Shin Kasai, Yoshiyuki Furuichi, MD, PhD, Norie Ando, Keiko Kagami, Kumiko Goi, MD, PhD, Takeshi Inukai, MD, PhD, Kanji Sugita, MD, PhD

Blood (2012) 120 (21): 1361.

https://doi.org/10.1182/blood.V120.21.1361.1361

Published: 2012

Journal Articles

PKC412, a FLT3 Inhibitor, Effectively Induces Apoptosis of Leukemia Cells with 11q23 Translocation Particularly Having D835 Mutation.

Kazuya Takahashi, MD, Kumiko Goi, MD, Yoshiyuki Furuichi, MD, Takeshi Inukai, MD, Kanji Sugita, MD, Shinpei Nakazawa, MD

Blood (2004) 104 (11): 1886.

https://doi.org/10.1182/blood.V104.11.1886.1886

Published: 2004

Journal Articles

Ligand Activation of FLT3 Induces Cell Cycle Arrest in Acute Lymphoblastic Leukemia with 11q23 Translocation.

Yoshiyuki Furuichi, M.D., Kanji Sugita, M.D., Takeshi Inukai, M.D., Kumiko Goi, M.D., Kazuya Takahashi, M.D., Shinpei Nakazawa, M.D.

Blood (2004) 104 (11): 1888.

https://doi.org/10.1182/blood.V104.11.1888.1888

Published: 2004

Journal Articles

FLT3 mutations in the activation loop of tyrosine kinase domain are frequently found in infant ALL with MLL rearrangements and pediatric ALL with hyperdiploidy

Takeshi Taketani, Tomohiko Taki, Kanji Sugita, Yoshiyuki Furuichi, Eiichi Ishii, Ryoji Hanada, Masahiro Tsuchida, Kenichi Sugita, Kohmei Ida, Yasuhide Hayashi

Blood (2004) 103 (3): 1085–1088.

https://doi.org/10.1182/blood-2003-02-0418

Published: 2004

NARROW

TGFβ1 Induces MLL -Rearranged Acute Lymphoblastic Leukemia into Chemoresistant Quiescence in a Coordinated Manner with FLT3 Ligand

Chemosensitivity Is Differentially Regulated by SDF-1/CXCR4 and SDF-1/CXCR7 Axes in Acute Lymphoblastic Leukemia.

Triggering of CD44 by Ultra-Low-Molecular-Weight Hyaluronan Induces Cell Death by Autophagy Via Endoplasmic Reticulum Stress in Acute Lymphoblastic Leukemia with MLL Gene Rearrangements

PKC412, a FLT3 Inhibitor, Effectively Induces Apoptosis of Leukemia Cells with 11q23 Translocation Particularly Having D835 Mutation.

Ligand Activation of FLT3 Induces Cell Cycle Arrest in Acute Lymphoblastic Leukemia with 11q23 Translocation.

FLT3 mutations in the activation loop of tyrosine kinase domain are frequently found in infant ALL with MLL rearrangements and pediatric ALL with hyperdiploidy

Email alerts

ASH Publications

American Society of Hematology

TGFβ1 Induces MLL -Rearranged Acute Lymphoblastic Leukemia into Chemoresistant Quiescence in a Coordinated Manner with FLT3 Ligand

Chemosensitivity Is Differentially Regulated by SDF-1/CXCR4 and SDF-1/CXCR7 Axes in Acute Lymphoblastic Leukemia.

Triggering of CD44 by Ultra-Low-Molecular-Weight Hyaluronan Induces Cell Death by Autophagy Via Endoplasmic Reticulum Stress in Acute Lymphoblastic Leukemia with MLL Gene Rearrangements

PKC412, a FLT3 Inhibitor, Effectively Induces Apoptosis of Leukemia Cells with 11q23 Translocation Particularly Having D835 Mutation.

Ligand Activation of FLT3 Induces Cell Cycle Arrest in Acute Lymphoblastic Leukemia with 11q23 Translocation.

FLT3 mutations in the activation loop of tyrosine kinase domain are frequently found in infant ALL with MLL rearrangements and pediatric ALL with hyperdiploidy

Email alerts

ASH Publications

American Society of Hematology

This Feature Is Available To Subscribers Only