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Journal Articles

A Leukemic Progression Model of Severe Congenital Neutropenia Uncovers a Novel Mechanism of AML Development Involving Elevated Inflammatory Responses, Mutation of CXXC4 and Decreased TET2 Levels

Patricia Olofsen, MSc, Szabolcs Fatrai, PhD, Paulette van Strien, BSc, Julia Obenauer, MSc, Remco Hoogenboezem, MSc, Claudia Erpelinck-Verschueren, B.Sc., Onno Roovers, BSc, Torsten Haferlach, MD, Peter Valk, PhD, Rebekka Schneider, MDPhD, Emma De Pater, PhD, Ivo Touw, PhD

Blood (2018) 132 (Supplement 1): 540.

https://doi.org/10.1182/blood-2018-99-115914

Published: 2018

Journal Articles

Combinatorial CSF3R and RUNX1 Mutations Involved in the Leukemic Progression of Severe Congenital Neutropenia Confer a G-CSF-Dependent Premalignant State in Mice

Szabolcs Fatrai, PhD, Paulette van Strien, BSc, Julia Obenauer, MSc, BSc, M.M. Van den Heuvel-Eibrink, MD, Ivo Touw, PhD

Blood (2017) 130 (Supplement 1): 543.

https://doi.org/10.1182/blood.V130.Suppl_1.543.543

Published: 2017

Journal Articles

Identification of HIF2α as an important STAT5 target gene in human hematopoietic stem cells

Szabolcs Fatrai, Albertus T. J. Wierenga, Simon M. G. J. Daenen, Edo Vellenga, Jan Jacob Schuringa

Blood (2011) 117 (12): 3320–3330.

https://doi.org/10.1182/blood-2010-08-303669

Published: 2011

Includes: Supplemental data

Journal Articles

Identification of Human Hematopoietic Stem Cell-Specific STAT5 Target Genes Involved in Self-Renewal and Transformation.

Szabolcs Fatrai, Albertus T.J. Wierenga, Edo Vellenga, Simon M. G. J. Daenen, Jan Jacob Schuringa

Blood (2009) 114 (22): 568.

https://doi.org/10.1182/blood.V114.22.568.568

Published: 2009

Journal Articles

Overexpression of Oncogenic KRAS G12V in Human Stem and Progenitor Cells Enhances Proliferation and Initiates Monocytic Differentiation Via Intrinsic and Extrinsic Pathways.

Szabolcs Fatrai, Djoke van Gosliga, Lina Han, Simon M. G. J. Daenen, Edo Vellenga, Jan Jacob Schuringa

Blood (2009) 114 (22): 3975.

https://doi.org/10.1182/blood.V114.22.3975.3975

Published: 2009

Journal Articles

Hematopoietic Stem Cells, but Not Progenitors, Are the Exclusive Target for STAT5-Induced Long-Term Self-Renewal.

Szabolcs Fatrai, Albertus T.J. Wierenga, Edo Vellenga, Simon M. G. J. Daenen, Jan J. Schuringa

Blood (2008) 112 (11): 1347.

https://doi.org/10.1182/blood.V112.11.1347.1347

Published: 2008

Journal Articles

Muc1 Expression Identifies Human Self-Renewing Stem/Progenitor Populations and Is Elevated in AML CD34+ Cells.

Szabolcs Fatrai, Simon M.G.J. Daenen, Edo Vellenga, Jan J. Schuringa

Blood (2007) 110 (11): 4040.

https://doi.org/10.1182/blood.V110.11.4040.4040

Published: 2007

NARROW

A Leukemic Progression Model of Severe Congenital Neutropenia Uncovers a Novel Mechanism of AML Development Involving Elevated Inflammatory Responses, Mutation of CXXC4 and Decreased TET2 Levels

Combinatorial CSF3R and RUNX1 Mutations Involved in the Leukemic Progression of Severe Congenital Neutropenia Confer a G-CSF-Dependent Premalignant State in Mice

Identification of HIF2α as an important STAT5 target gene in human hematopoietic stem cells

Identification of Human Hematopoietic Stem Cell-Specific STAT5 Target Genes Involved in Self-Renewal and Transformation.

Overexpression of Oncogenic KRAS G12V in Human Stem and Progenitor Cells Enhances Proliferation and Initiates Monocytic Differentiation Via Intrinsic and Extrinsic Pathways.

Hematopoietic Stem Cells, but Not Progenitors, Are the Exclusive Target for STAT5-Induced Long-Term Self-Renewal.

Muc1 Expression Identifies Human Self-Renewing Stem/Progenitor Populations and Is Elevated in AML CD34+ Cells.

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ASH Publications

American Society of Hematology

A Leukemic Progression Model of Severe Congenital Neutropenia Uncovers a Novel Mechanism of AML Development Involving Elevated Inflammatory Responses, Mutation of CXXC4 and Decreased TET2 Levels

Combinatorial CSF3R and RUNX1 Mutations Involved in the Leukemic Progression of Severe Congenital Neutropenia Confer a G-CSF-Dependent Premalignant State in Mice

Identification of HIF2α as an important STAT5 target gene in human hematopoietic stem cells

Identification of Human Hematopoietic Stem Cell-Specific STAT5 Target Genes Involved in Self-Renewal and Transformation.

Overexpression of Oncogenic KRAS G12V in Human Stem and Progenitor Cells Enhances Proliferation and Initiates Monocytic Differentiation Via Intrinsic and Extrinsic Pathways.

Hematopoietic Stem Cells, but Not Progenitors, Are the Exclusive Target for STAT5-Induced Long-Term Self-Renewal.

Muc1 Expression Identifies Human Self-Renewing Stem/Progenitor Populations and Is Elevated in AML CD34+ Cells.

Email alerts

ASH Publications

American Society of Hematology

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