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Shawn A. Mahmud
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Gene-Specific Effects of the Histone Deacetylase Inhibitor TSA on Activation of a Pro-Inflammatory Endothelial Cell Phenotype by the Major Phagocyte Peroxidase-Derived Oxidant HOSCN.

Teena Pasrija, Ph.D., Shawn A Mahmud, Ph.D., John D Belcher, Ph.D., Gregory M. Vercellotti, MD, Arne Slungaard, MD
Journal: Blood
Blood (2008) 112 (11): 1274.
https://doi.org/10.1182/blood.V112.11.1274.1274
Published: 2008
Journal Articles

The Major Phagocyte Peroxidase-Derived Oxidant, HOSCN (Hypothiocyanous Acid), Induces Proinflammatory and Cytoprotective Gene Expression in Endothelium: A Mechanism for Microlocalized Regulation of Inflammation.

Shawn A. Mahmud, BA, Jian-Guo Wang, Ph.D., Peng Wei, M.S., Molly S. Thomas, Ph.D., John D. Belcher, Ph.D., Gregory M. Vercellotti, M.D., Arne Slungaard, M.D.
Journal: Blood
Blood (2007) 110 (11): 3295.
https://doi.org/10.1182/blood.V110.11.3295.3295
Published: 2007
Journal Articles

Endogenous platelet factor 4 stimulates activated protein C generation in vivo and improves survival after thrombin or lipopolysaccharide challenge

Brief Report
M. Anna Kowalska, Shawn A. Mahmud, Michele P. Lambert, Mortimer Poncz, Arne Slungaard
Journal: Blood
Blood (2007) 110 (6): 1903–1905.
https://doi.org/10.1182/blood-2007-03-081901
Published: 2007
Journal Articles

“TF-kB”-Dependent Suppression of Agonist-Mediated Tissue Factor Induction by Histone Deacetylase Inhibitors in Human Endothelial Cells and Monocytes.

Jianguo Wang, PhD, Shawn A. Mahmud, B.A., Peter B. Bitterman, M.D., Yuqing Huo, M.D., Ph.D., Arne Slungaard, M.D.
Journal: Blood
Blood (2006) 108 (11): 1745.
https://doi.org/10.1182/blood.V108.11.1745.1745
Published: 2006
Journal Articles

Release of High Levels of Platelet Factor 4 (PF4) from Platelets Improves Survival after a Lethal Lipopolysaccharide (LPS) Challenge.

M. Anna Kowalska, PhD, Michele P. Lambert, MD, Lubica Rauova, MD, PhD, William J. Smith, BS, Shawn A. Mahmud, BS, Mortimer Poncz, MD, Arne Slungaard, MD
Journal: Blood
Blood (2006) 108 (11): 61.
https://doi.org/10.1182/blood.V108.11.61.61
Published: 2006
Journal Articles

Thiocyanate-Dependent Inhibition of Spontaneous and Agonist-Induced Eosinophil Apoptosis by Eosinophil Peroxidase (EPO): A Potential Physiologic Role for Endogenously Generated HOSCN in Maintaining Eosinophil Viability.

Shawn A. Mahmud, BS, Jianguo Wang, Ph.D., Arne Slungaard, M.D.
Journal: Blood
Blood (2006) 108 (11): 1633.
https://doi.org/10.1182/blood.V108.11.1633.1633
Published: 2006
Journal Articles

The principal eosinophil peroxidase product, HOSCN, is a uniquely potent phagocyte oxidant inducer of endothelial cell tissue factor activity: a potential mechanism for thrombosis in eosinophilic inflammatory states

Jian-Guo Wang, Shawn A. Mahmud, Jacob A. Thompson, Jian-Guo Geng, Nigel S. Key, Arne Slungaard
Journal: Blood
Blood (2006) 107 (2): 558–565.
https://doi.org/10.1182/blood-2005-05-2152
Published: 2006
Journal Articles

Endogenous Platelet PF4 Promotes In Vivo Activated Protein C (APC) Generation and Survival after Lethal Lipopolysaccharide Challenge in Mice: A Potential Physiologic Role for PF4.

M. Anna Kowalska, PhD, Shawn A. Mahmud, BA, Mortimer Poncz, MD, Arne Slungaard, MD
Journal: Blood
Blood (2005) 106 (11): 27.
https://doi.org/10.1182/blood.V106.11.27.27
Published: 2005
Journal Articles

The Principal Eosinophil Peroxidase Oxidant, HOSCN, Potently Induces Endothelial Cell Tissue Factor Activity through Transcriptional Activation Dependent upon NF-kB and ERK1/2: Potential Mechanism for a Prothrombotic and Proinflammatory Endothelial Phenotype in the Hypereosinophilic Syndrome.

Jianguo Wang, Ph.D., Shawn A. Mahmud, B.S., Jake A. Thompson, B.S., Jianguo Geng, M.D./Ph.D., Nigel S. Key, M.D., Arne Slungaard, M.D.
Journal: Blood
Blood (2004) 104 (11): 804.
https://doi.org/10.1182/blood.V104.11.804.804
Published: 2004