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Journal Articles

No red blood cell damage and no hemolysis in G6PD-deficient subjects after ingestion of low vicine/convicine Vicia faba seeds

Valentina Gallo, Oleksii A. Skorokhod, Luigi Felice Simula, Tiziana Marrocco, Elisa Tambini, Evelin Schwarzer, Pascal Marget, Gérard Duc, Paolo Arese

Blood (2018) 131 (14): 1621–1625.

https://doi.org/10.1182/blood-2017-09-806364

Published: 2018

Journal Articles

Host fibrinogen stably bound to hemozoin rapidly activates monocytes via TLR-4 and CD11b/CD18-integrin: a new paradigm of hemozoin action

Valentina Barrera, Oleksii A. Skorokhod, Denisa Baci, Giuliana Gremo, Paolo Arese, Evelin Schwarzer

Blood (2011) 117 (21): 5674–5682.

https://doi.org/10.1182/blood-2010-10-312413

Published: 2011

Includes: Supplemental data

Journal Articles

Inhibition of erythropoiesis in malaria anemia: role of hemozoin and hemozoin-generated 4-hydroxynonenal

Oleksii A. Skorokhod, Luisa Caione, Tiziana Marrocco, Giorgia Migliardi, Valentina Barrera, Paolo Arese, Wanda Piacibello, Evelin Schwarzer

Blood (2010) 116 (20): 4328–4337.

https://doi.org/10.1182/blood-2010-03-272781

Published: 2010

Includes: Supplemental data

Journal Articles

Enhanced phagocytosis of ring-parasitized mutant erythrocytes: a common mechanism that may explain protection against falciparum malaria in sickle trait and beta-thalassemia trait

Kodjo Ayi, Franco Turrini, Antonio Piga, Paolo Arese

Blood (2004) 104 (10): 3364–3371.

https://doi.org/10.1182/blood-2003-11-3820

Published: 2004

Journal Articles

Malaria-parasitized erythrocytes and hemozoin nonenzymatically generate large amounts of hydroxy fatty acids that inhibit monocyte functions

Evelin Schwarzer, Hartmut Kühn, Elena Valente, Paolo Arese

Blood (2003) 101 (2): 722–728.

https://doi.org/10.1182/blood-2002-03-0979

Published: 2003

Journal Articles

Early Phagocytosis of Glucose-6-Phosphate Dehydrogenase (G6PD)-Deficient Erythrocytes Parasitized by Plasmodium falciparum May Explain Malaria Protection in G6PD Deficiency

Marina Cappadoro, Giuliana Giribaldi, Estella O'Brien, Franco Turrini, Franca Mannu, Daniela Ulliers, Gino Simula, Lucio Luzzatto, Paolo Arese

Blood (1998) 92 (7): 2527–2534.

https://doi.org/10.1182/blood.V92.7.2527

Published: 1998

NARROW

No red blood cell damage and no hemolysis in G6PD-deficient subjects after ingestion of low vicine/convicine Vicia faba seeds

Host fibrinogen stably bound to hemozoin rapidly activates monocytes via TLR-4 and CD11b/CD18-integrin: a new paradigm of hemozoin action

Inhibition of erythropoiesis in malaria anemia: role of hemozoin and hemozoin-generated 4-hydroxynonenal

Enhanced phagocytosis of ring-parasitized mutant erythrocytes: a common mechanism that may explain protection against falciparum malaria in sickle trait and beta-thalassemia trait

Malaria-parasitized erythrocytes and hemozoin nonenzymatically generate large amounts of hydroxy fatty acids that inhibit monocyte functions

Early Phagocytosis of Glucose-6-Phosphate Dehydrogenase (G6PD)-Deficient Erythrocytes Parasitized by Plasmodium falciparum May Explain Malaria Protection in G6PD Deficiency

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ASH Publications

American Society of Hematology

No red blood cell damage and no hemolysis in G6PD-deficient subjects after ingestion of low vicine/convicine Vicia faba seeds

Host fibrinogen stably bound to hemozoin rapidly activates monocytes via TLR-4 and CD11b/CD18-integrin: a new paradigm of hemozoin action

Inhibition of erythropoiesis in malaria anemia: role of hemozoin and hemozoin-generated 4-hydroxynonenal

Enhanced phagocytosis of ring-parasitized mutant erythrocytes: a common mechanism that may explain protection against falciparum malaria in sickle trait and beta-thalassemia trait

Malaria-parasitized erythrocytes and hemozoin nonenzymatically generate large amounts of hydroxy fatty acids that inhibit monocyte functions

Early Phagocytosis of Glucose-6-Phosphate Dehydrogenase (G6PD)-Deficient Erythrocytes Parasitized by Plasmodium falciparum May Explain Malaria Protection in G6PD Deficiency

Email alerts

ASH Publications

American Society of Hematology

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