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Journal Articles

BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistance

Mateusz Koptyra, Rafal Falinski, Michal O. Nowicki, Tomasz Stoklosa, Ireneusz Majsterek, Margaret Nieborowska-Skorska, Janusz Blasiak, Tomasz Skorski

Blood (2006) 108 (1): 319–327.

https://doi.org/10.1182/blood-2005-07-2815

Published: 2006

Includes: Supplemental data

Journal Articles

BCR/ABL Regulates the Expression and Interacts with Werner Syndrome Helicase/Exonuclease To Modulate Its Biochemical Properties.

Artur Slupianek, PhD, Stanislaw Jozwiakowski, Ewa Gurdek, Michal O. Nowicki, Tomasz Skorski, MD, PhD

Blood (2005) 106 (11): 2873.

https://doi.org/10.1182/blood.V106.11.2873.2873

Published: 2005

Journal Articles

BCR/ABL Modifies the Kinetics and Fidelity of DNA Double-Strand Breaks Repair in Leukemia Cells.

Artur Slupianek, PhD, Michal O. Nowicki, Tomasz Skorski, MD, PhD

Blood (2005) 106 (11): 1982.

https://doi.org/10.1182/blood.V106.11.1982.1982

Published: 2005

Journal Articles

BCR/ABL oncogenic kinase promotes unfaithful repair of the reactive oxygen species–dependent DNA double-strand breaks

Michal O. Nowicki, Rafal Falinski, Mateusz Koptyra, Artur Slupianek, Tomasz Stoklosa, Ewa Gloc, Margaret Nieborowska-Skorska, Janusz Blasiak, Tomasz Skorski

Blood (2004) 104 (12): 3746–3753.

https://doi.org/10.1182/blood-2004-05-1941

Published: 2004

Journal Articles

BCR/ABL Oncogenic Kinase Promotes Unfaithful Repair of the Reactive Oxygen Species - Dependent DNA Double-Strand Breaks.

Tomasz Skorski, MD, PhD, Michal O. Nowicki, MS, Rafal Falinski, MS, Mateusz Koptyra, MS, Artur Slupianek, PhD, Tomasz Stoklosa, MD, Ewa Gloc, Margaret Nieborowska-Skorska, MD, PhD, Janusz Blasiak, PhD

Blood (2004) 104 (11): 712.

https://doi.org/10.1182/blood.V104.11.712.712

Published: 2004

Journal Articles

BCR/ABL Oncogenic Kinase Stimulates DNA Oxidative Damage To Induce Self-Mutagenesis Causing Resistance to Imatinib Mesylate.

Mateusz Koptyra, MS, Rafal Falinski, MS, Michal O. Nowicki, MS, Janusz Blasiak, PhD, Tomasz Skorski, MD, PhD

Blood (2004) 104 (11): 714.

https://doi.org/10.1182/blood.V104.11.714.714

Published: 2004

NARROW

BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistance

BCR/ABL Regulates the Expression and Interacts with Werner Syndrome Helicase/Exonuclease To Modulate Its Biochemical Properties.

BCR/ABL Modifies the Kinetics and Fidelity of DNA Double-Strand Breaks Repair in Leukemia Cells.

BCR/ABL oncogenic kinase promotes unfaithful repair of the reactive oxygen species–dependent DNA double-strand breaks

BCR/ABL Oncogenic Kinase Promotes Unfaithful Repair of the Reactive Oxygen Species - Dependent DNA Double-Strand Breaks.

BCR/ABL Oncogenic Kinase Stimulates DNA Oxidative Damage To Induce Self-Mutagenesis Causing Resistance to Imatinib Mesylate.

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ASH Publications

American Society of Hematology

BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistance

BCR/ABL Regulates the Expression and Interacts with Werner Syndrome Helicase/Exonuclease To Modulate Its Biochemical Properties.

BCR/ABL Modifies the Kinetics and Fidelity of DNA Double-Strand Breaks Repair in Leukemia Cells.

BCR/ABL oncogenic kinase promotes unfaithful repair of the reactive oxygen species–dependent DNA double-strand breaks

BCR/ABL Oncogenic Kinase Promotes Unfaithful Repair of the Reactive Oxygen Species - Dependent DNA Double-Strand Breaks.

BCR/ABL Oncogenic Kinase Stimulates DNA Oxidative Damage To Induce Self-Mutagenesis Causing Resistance to Imatinib Mesylate.

Email alerts

ASH Publications

American Society of Hematology

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