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Journal Articles

Identification of key regulatory pathways of myeloid differentiation using an mESC-based karyotypically normal cell model

Dong Li, Hong Yang, Hong Nan, Peng Liu, Sulei Pang, Qian Zhao, Rotem Karni, Mark P. Kamps, Yuanfu Xu, Jiaxi Zhou, Therese Wiedmer, Peter J. Sims, Fei Wang

Blood (2012) 120 (24): 4712–4719.

https://doi.org/10.1182/blood-2012-03-414979

Published: 2012

Includes: Supplemental data

Journal Articles

Proteomics-based discovery of a novel, structurally unique, and developmentally regulated plasminogen receptor, Plg-R KT , a major regulator of cell surface plasminogen activation

Nicholas M. Andronicos, Emily I. Chen, Nagyung Baik, Hongdong Bai, Caitlin M. Parmer, William B. Kiosses, Mark P. Kamps, John R. Yates, III, Robert J. Parmer, Lindsey A. Miles

Blood (2010) 115 (7): 1319–1330.

https://doi.org/10.1182/blood-2008-11-188938

Published: 2010

Includes: Supplemental data

Journal Articles

Survival signaling in HoxA9/Meis1 AML

Gang G. Wang, Mark P. Kamps

Blood (2007) 109 (9): 3619–3620.

https://doi.org/10.1182/blood-2007-02-072785

Published: 2007

Journal Articles

Fusion to Vp16 Converts MEIS1 into an Oncoprotein That Immortalizes Progenitors and Causes AML in the Absence of Coexpressed Hox Genes: Hoxa7 and Hoxa9 Induced Further Stem Cell Gene Transcription in Vp16MEIS1 Progenitors.

Gang G. Wang, MS, Martina P. Pasillas, BS, Mark P. Kamps, PhD

Blood (2005) 106 (11): 662.

https://doi.org/10.1182/blood.V106.11.662.662

Published: 2005

Journal Articles

Meis1 programs transcription of FLT3 and cancer stem cell character, using a mechanism that requires interaction with Pbx and a novel function of the Meis1 C -terminus

Gang G. Wang, Martina P. Pasillas, Mark P. Kamps

Blood (2005) 106 (1): 254–264.

https://doi.org/10.1182/blood-2004-12-4664

Published: 2005

Includes: Supplemental data

Journal Articles

Estrogen-dependent E2a/Pbx1 myeloid cell lines exhibit conditional differentiation that can be arrested by other leukemic oncoproteins

David B. Sykes, Mark P. Kamps

Blood (2001) 98 (8): 2308–2318.

https://doi.org/10.1182/blood.V98.8.2308

Published: 2001

NARROW

Identification of key regulatory pathways of myeloid differentiation using an mESC-based karyotypically normal cell model

Proteomics-based discovery of a novel, structurally unique, and developmentally regulated plasminogen receptor, Plg-R KT , a major regulator of cell surface plasminogen activation

Survival signaling in HoxA9/Meis1 AML

Fusion to Vp16 Converts MEIS1 into an Oncoprotein That Immortalizes Progenitors and Causes AML in the Absence of Coexpressed Hox Genes: Hoxa7 and Hoxa9 Induced Further Stem Cell Gene Transcription in Vp16MEIS1 Progenitors.

Meis1 programs transcription of FLT3 and cancer stem cell character, using a mechanism that requires interaction with Pbx and a novel function of the Meis1 C -terminus

Estrogen-dependent E2a/Pbx1 myeloid cell lines exhibit conditional differentiation that can be arrested by other leukemic oncoproteins

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ASH Publications

American Society of Hematology

Identification of key regulatory pathways of myeloid differentiation using an mESC-based karyotypically normal cell model

Proteomics-based discovery of a novel, structurally unique, and developmentally regulated plasminogen receptor, Plg-R KT , a major regulator of cell surface plasminogen activation

Survival signaling in HoxA9/Meis1 AML

Fusion to Vp16 Converts MEIS1 into an Oncoprotein That Immortalizes Progenitors and Causes AML in the Absence of Coexpressed Hox Genes: Hoxa7 and Hoxa9 Induced Further Stem Cell Gene Transcription in Vp16MEIS1 Progenitors.

Meis1 programs transcription of FLT3 and cancer stem cell character, using a mechanism that requires interaction with Pbx and a novel function of the Meis1 C -terminus

Estrogen-dependent E2a/Pbx1 myeloid cell lines exhibit conditional differentiation that can be arrested by other leukemic oncoproteins

Email alerts

ASH Publications

American Society of Hematology

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