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Journal Articles

Nampt/SIRT2 Hyperactivation Lead to Activation of ß-Catenin: a Possible Mechanism of Leukomogenic Transformation In CN Patients.

Lan Dan, Ana Gigina, Karl Welte, MD, Julia Skokowa, Dr

Blood (2010) 116 (21): 1486.

https://doi.org/10.1182/blood.V116.21.1486.1486

Published: 2010

Journal Articles

New G-CSF-Dependent Signaling Pathway and Its Role In Patients with Severe Congenital Neutropenia and Acute Myeloid Leukemia

Julia Skokowa, Dr, Elena Chirvon, Lan Dan, Kshama Gupta, Ana Gigina, Marlene Reuter, Annette Müller Brechlin, Kais Hussein, Esteban Carrizosa, Janis Burkhardt, Hans Kreipe, Karl Welte, MD

Blood (2010) 116 (21): 385.

https://doi.org/10.1182/blood.V116.21.385.385

Published: 2010

Journal Articles

Neutrophil elastase is severely down-regulated in severe congenital neutropenia independent of ELA2 or HAX1 mutations but dependent on LEF-1

Julia Skokowa, John Paul Fobiwe, Lan Dan, Basant Kumar Thakur, Karl Welte

Blood (2009) 114 (14): 3044–3051.

https://doi.org/10.1182/blood-2008-11-188755

Published: 2009

Journal Articles

The in Vivo Response of Patients Suffering from Severe Congenital Neutropenia (CN) to G-CSF Is Due to “Emergency” Granulopoiesis Induced by C/EBPβ

Lan Dan, Basant Kumar Thakur, Julia Skokowa, Karl Welte

Blood (2008) 112 (11): 315.

https://doi.org/10.1182/blood.V112.11.315.315

Published: 2008

Journal Articles

LEF-1 Transcription Factor Regulates Proliferation and Differentiation of Myeloid Progenitors in Healthy Individuals and in Patients with Severe Congenital Neutropenia (CN).

Julia Skokowa, MD, PhD, Gunnar Cario, MD, Vesna Bucan, Lan Dan, Zheng Wang, PhD, Michaela Scherr, PhD, Axel Schambach, MD, Cornelia Zeidler, MD, Mathias Eder, MD, Christopher Baum, MD, Martin Stanulla, MD, Karl Welte, MD, PhD

Blood (2005) 106 (11): 390.

https://doi.org/10.1182/blood.V106.11.390.390

Published: 2005

Journal Articles

Pre-B-Cell Colony Enhancing Factor (PBEF) Is a New Cytokine Regulating Myeloid Differentiation in Healthy Individuals and Patients with Severe Congenital Neutropenia (CN).

Julia Skokowa, MD, PhD, Lan Dan, Gunnar Cario, MD, Axel Schambach, MD, Cornelia Zeidler, MD, Christopher Baum, MD, Martin Stanulla, MD, Karl Welte, MD, PhD

Blood (2005) 106 (11): 387.

https://doi.org/10.1182/blood.V106.11.387.387

Published: 2005

Journal Articles

Proto-Oncogenes β- and γ-Catenin in Leukemogenesis in Severe Congenital Neutropenia (CN).

Julia Skokowa, MD, PhD, Gunnar Cario, MD, Lan Dan, Cornelia Zeidler, MD, Vesna Bucan, Karl Welte, MD, PhD

Blood (2005) 106 (11): 94.

https://doi.org/10.1182/blood.V106.11.94.94

Published: 2005

NARROW

Nampt/SIRT2 Hyperactivation Lead to Activation of ß-Catenin: a Possible Mechanism of Leukomogenic Transformation In CN Patients.

New G-CSF-Dependent Signaling Pathway and Its Role In Patients with Severe Congenital Neutropenia and Acute Myeloid Leukemia

Neutrophil elastase is severely down-regulated in severe congenital neutropenia independent of ELA2 or HAX1 mutations but dependent on LEF-1

The in Vivo Response of Patients Suffering from Severe Congenital Neutropenia (CN) to G-CSF Is Due to “Emergency” Granulopoiesis Induced by C/EBPβ

LEF-1 Transcription Factor Regulates Proliferation and Differentiation of Myeloid Progenitors in Healthy Individuals and in Patients with Severe Congenital Neutropenia (CN).

Pre-B-Cell Colony Enhancing Factor (PBEF) Is a New Cytokine Regulating Myeloid Differentiation in Healthy Individuals and Patients with Severe Congenital Neutropenia (CN).

Proto-Oncogenes β- and γ-Catenin in Leukemogenesis in Severe Congenital Neutropenia (CN).

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American Society of Hematology

Nampt/SIRT2 Hyperactivation Lead to Activation of ß-Catenin: a Possible Mechanism of Leukomogenic Transformation In CN Patients.

New G-CSF-Dependent Signaling Pathway and Its Role In Patients with Severe Congenital Neutropenia and Acute Myeloid Leukemia

Neutrophil elastase is severely down-regulated in severe congenital neutropenia independent of ELA2 or HAX1 mutations but dependent on LEF-1

The in Vivo Response of Patients Suffering from Severe Congenital Neutropenia (CN) to G-CSF Is Due to “Emergency” Granulopoiesis Induced by C/EBPβ

LEF-1 Transcription Factor Regulates Proliferation and Differentiation of Myeloid Progenitors in Healthy Individuals and in Patients with Severe Congenital Neutropenia (CN).

Pre-B-Cell Colony Enhancing Factor (PBEF) Is a New Cytokine Regulating Myeloid Differentiation in Healthy Individuals and Patients with Severe Congenital Neutropenia (CN).

Proto-Oncogenes β- and γ-Catenin in Leukemogenesis in Severe Congenital Neutropenia (CN).

Email alerts

ASH Publications

American Society of Hematology

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