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Journal Articles

Down-regulation of BOB.1/OBF.1 and Oct2 in classical Hodgkin disease but not in lymphocyte predominant Hodgkin disease correlates with immunoglobulin transcription

Harald Stein, Theresa Marafioti, Hans-Dieter Foss, Helmut Laumen, Michael Hummel, Ioannis Anagnostopoulos, Thomas Wirth, Gudrun Demel, Brunangelo Falini

Blood (2001) 97 (2): 496–501.

https://doi.org/10.1182/blood.V97.2.496

Published: 2001

Journal Articles

Hodgkin and Reed-Sternberg cells represent an expansion of a single clone originating from a germinal center B-cell with functional immunoglobulin gene rearrangements but defective immunoglobulin transcription

Theresa Marafioti, Michael Hummel, Hans-Dieter Foss, Helmut Laumen, Petra Korbjuhn, Ioannis Anagnostopoulos, Hetty Lammert, Gudrun Demel, Jan Theil, Thomas Wirth, Harald Stein

Blood (2000) 95 (4): 1443–1450.

https://doi.org/10.1182/blood.V95.4.1443.004k55_1443_1450

Published: 2000

Journal Articles

Frequent Expression of the B-Cell–Specific Activator Protein in Reed-Sternberg Cells of Classical Hodgkin’s Disease Provides Further Evidence for Its B-Cell Origin

Hans-Dieter Foss, Regina Reusch, Gudrun Demel, Georg Lenz, Ioannis Anagnostopoulos, Michael Hummel, Harald Stein

Blood (1999) 94 (9): 3108–3113.

https://doi.org/10.1182/blood.V94.9.3108

Published: 1999

Journal Articles

Overexpression of I Kappa B Alpha Without Inhibition of NF-κB Activity and Mutations in the I Kappa B Alpha Gene in Reed-Sternberg Cells

Florian Emmerich, Martina Meiser, Michael Hummel, Gudrun Demel, Hans-Dieter Foss, Franziska Jundt, Stephan Mathas, Daniel Krappmann, Claus Scheidereit, Harald Stein, Bernd Dörken

Blood (1999) 94 (9): 3129–3134.

https://doi.org/10.1182/blood.V94.9.3129

Published: 1999

Journal Articles

Tumor Necrosis Factor Receptor-Associated Factor 1 Is Overexpressed in Reed-Sternberg Cells of Hodgkin’s Disease and Epstein-Barr Virus-Transformed Lymphoid Cells

Horst Dürkop, Hans-Dieter Foss, Gudrun Demel, Heike Klotzbach, Corinna Hahn, Harald Stein

Blood (1999) 93 (2): 617–623.

https://doi.org/10.1182/blood.V93.2.617

Published: 1999

NARROW

Down-regulation of BOB.1/OBF.1 and Oct2 in classical Hodgkin disease but not in lymphocyte predominant Hodgkin disease correlates with immunoglobulin transcription

Hodgkin and Reed-Sternberg cells represent an expansion of a single clone originating from a germinal center B-cell with functional immunoglobulin gene rearrangements but defective immunoglobulin transcription

Frequent Expression of the B-Cell–Specific Activator Protein in Reed-Sternberg Cells of Classical Hodgkin’s Disease Provides Further Evidence for Its B-Cell Origin

Overexpression of I Kappa B Alpha Without Inhibition of NF-κB Activity and Mutations in the I Kappa B Alpha Gene in Reed-Sternberg Cells

Tumor Necrosis Factor Receptor-Associated Factor 1 Is Overexpressed in Reed-Sternberg Cells of Hodgkin’s Disease and Epstein-Barr Virus-Transformed Lymphoid Cells

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ASH Publications

American Society of Hematology

Down-regulation of BOB.1/OBF.1 and Oct2 in classical Hodgkin disease but not in lymphocyte predominant Hodgkin disease correlates with immunoglobulin transcription

Hodgkin and Reed-Sternberg cells represent an expansion of a single clone originating from a germinal center B-cell with functional immunoglobulin gene rearrangements but defective immunoglobulin transcription

Frequent Expression of the B-Cell–Specific Activator Protein in Reed-Sternberg Cells of Classical Hodgkin’s Disease Provides Further Evidence for Its B-Cell Origin

Overexpression of I Kappa B Alpha Without Inhibition of NF-κB Activity and Mutations in the I Kappa B Alpha Gene in Reed-Sternberg Cells

Tumor Necrosis Factor Receptor-Associated Factor 1 Is Overexpressed in Reed-Sternberg Cells of Hodgkin’s Disease and Epstein-Barr Virus-Transformed Lymphoid Cells

Email alerts

ASH Publications

American Society of Hematology

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