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Journal Articles

Genetic Deletion or Pharmacologic Inhibition of PTPN11 Impedes the Development and Progression of Myeloproliferative Neoplasms Induced By JAK2V617F and MPLW515L Mutants

Golam Mohi, Yue Yang, Mohammad A. Sayem, Bao T. Le, Fahim Ather, Avik Dutta, Salar Abbas

Blood (2023) 142 (Supplement 1): 739.

https://doi.org/10.1182/blood-2023-190216

Published: 2023

Journal Articles

PTPN1 Deficiency Accelerates the Progression to Myelofibrosis in JAK2V617F-Driven Myeloproliferative Neoplasm

Bao T. Le, Yue Yang, Golam Mohi

Blood (2022) 140 (Supplement 1): 3855.

https://doi.org/10.1182/blood-2022-170621

Published: 2022

Journal Articles

IL-1 Signaling Promotes Clonal Expansion and Progression of Bone Marrow Fibrosis in JAK2V617F-Induced Myeloproliferative Neoplasm

Mohammed Ferdous Ur Rahman, Yue Yang, Bao T. Le, Avik Dutta, Patrick Faughnan, Golam Mohi

Blood (2021) 138 (Supplement 1): 2540.

https://doi.org/10.1182/blood-2021-147517

Published: 2021

Journal Articles

Inhibition of SHP2 Ameliorates JAK2V617F-Induced Myeloproliferative Neoplasms

Yue Yang, Bao T. Le, Avik Dutta, Golam Mohi

Blood (2021) 138 (Supplement 1): 1465.

https://doi.org/10.1182/blood-2021-152174

Published: 2021

Journal Articles

The RNA Splicing Factor U2AF1 Controls Hematopoietic Stem Cell Survival and Function

Avik Dutta, PhD, Yue Yang, PhD, Bao Le, Golam Mohi, PhD

Blood (2020) 136 (Supplement 1): 9.

https://doi.org/10.1182/blood-2020-143163

Published: 2020

Journal Articles

A Phase 1 Study of TP-3654, an Orally-Delivered PIM Kinase Inhibitor, in Patients with Intermediate-2 or High-Risk Primary or Secondary Myelofibrosis

Claudia Lebedinsky, MD, Stephen P. Anthony, DO, Golam Mohi, PhD, Huyuan Yang, PhD, Jian Mei, PharmD, Edgar Braendle, MD

Blood (2020) 136 (Supplement 1): 3–4.

https://doi.org/10.1182/blood-2020-134039

Published: 2020

Journal Articles

Critical Requirement of U2AF1 in the Maintenance and Function of Hematopoietic Stem Cells

Avik Dutta, PhD, Yue Yang, PhD, Bao Le, Golam Mohi, PhD

Blood (2019) 134 (Supplement_1): 1195.

https://doi.org/10.1182/blood-2019-132007

Published: 2019

Journal Articles

The CDK6 Inhibitor Palbociclib in Combination with Ruxolitinib Remarkably Improves Myelofibrosis in Murine Models

Golam Mohi, PhD, Avik Dutta, PhD, Dipmoy Nath, PhD, Yue Yang, PhD

Blood (2019) 134 (Supplement_1): 4200.

https://doi.org/10.1182/blood-2019-132038

Published: 2019

Journal Articles

The NF-KB Inhibitor DMAPT in Combination with Ruxolitinib Displays Efficacy in Jak2V617F Knock-in Mouse Model of Myeloproliferative Neoplasms

Yue Yang, BS, Dipmoy Nath, PhD, Avik Dutta, MSc, Peter A Crooks, PhD, Golam Mohi, PhD

Blood (2018) 132 (Supplement 1): 1783.

https://doi.org/10.1182/blood-2018-99-120195

Published: 2018

Journal Articles

The PIM Kinase Inhibitor TP-3654 in Combination with Ruxolitinib Exhibits Marked Improvement of Myelofibrosis in Murine Models

Dipmoy Nath, PhD, Avik Dutta, MSc, Yue Yang, BS, Clifford Whatcott, PhD, Steven L. Warner, PhD, Golam Mohi, PhD

Blood (2018) 132 (Supplement 1): 54.

https://doi.org/10.1182/blood-2018-99-119421

Published: 2018

Journal Articles

Hmga2 promotes the development of myelofibrosis in Jak2 V617F knockin mice by enhancing TGF-β1 and Cxcl12 pathways

Avik Dutta, Robert E. Hutchison, Golam Mohi

Blood (2017) 130 (7): 920–932.

https://doi.org/10.1182/blood-2016-12-757344

Published: 2017

Includes: Supplemental data

Journal Articles

Deletion of PTPN1 Promotes the Development of Myelofibrosis

Fatoumata Jobe, PhD, Bhumika Patel, MD, Hideki Makishima, MD PhD, Bartlomiej P Przychodzen, MSc, Robert E Hutchison, MD, Kendra K Bence, PhD, Jaroslaw P Maciejewski, MD PhD FACP, Golam Mohi, PhD

Blood (2016) 128 (22): 795.

https://doi.org/10.1182/blood.V128.22.795.795

Published: 2016

Journal Articles

Expression of HMGA2 Cooperates with Jak2V617F in the Development of Myelofibrosis

Avik Dutta, MSc, Robert E Hutchison, MD, Golam Mohi, PhD

Blood (2016) 128 (22): 797.

https://doi.org/10.1182/blood.V128.22.797.797

Published: 2016

Journal Articles

Loss of Ezh2 cooperates with Jak2V617F in the development of myelofibrosis in a mouse model of myeloproliferative neoplasm

Yue Yang, Hajime Akada, Dipmoy Nath, Robert E. Hutchison, Golam Mohi

Blood (2016) 127 (26): 3410–3423.

https://doi.org/10.1182/blood-2015-11-679431

Published: 2016

Includes: Supplemental data

Journal Articles

Distinct GAB2 signaling pathways are essential for myeloid and lymphoid transformation and leukemogenesis by BCR-ABL1

Shengqing Gu, Wayne W. Chan, Golam Mohi, Joel Rosenbaum, Azin Sayad, Zhibin Lu, Carl Virtanen, Shaoguang Li, Benjamin G. Neel, Richard A. Van Etten

Blood (2016) 127 (14): 1803–1813.

https://doi.org/10.1182/blood-2015-06-653006

Published: 2016

Includes: Supplemental data

Journal Articles

Critical Role of SHP2 in the Initiation and Maintenance of Myeloproliferative Neoplasms Evoked By JAK2V617F

Golam Mohi, PhD, Dongqing Yan, PhD, Fatoumata Jobe, Robert E Hutchison, MD

Blood (2015) 126 (23): 483.

https://doi.org/10.1182/blood.V126.23.483.483

Published: 2015

Journal Articles

Loss of EZH2 Inhibits Erythropoiesis and Accelerates the Development of Myelofibrosis in Jak2V617F Knock-in Mice

Yue Yang, Hajime Akada, PhD, Dipmoy Nath, Robert E Hutchison, MD, Golam Mohi, PhD

Blood (2014) 124 (21): 159.

https://doi.org/10.1182/blood.V124.21.159.159

Published: 2014

Journal Articles

Stat3 Negatively Regulates Myeloproliferative Neoplasm Induced By Jak2V617F

Dongqing Yan, Ph.D., Golam Mohi, PhD

Blood (2013) 122 (21): 111.

https://doi.org/10.1182/blood.V122.21.111.111

Published: 2013

Journal Articles

Critical Role Of Jak2 In The Maintenance and Function Of Adult Hematopoietic Stem Cells

Hajime Akada, Saeko Akada, Golam Mohi, PhD

Blood (2013) 122 (21): 1180.

https://doi.org/10.1182/blood.V122.21.1180.1180

Published: 2013

Journal Articles

Loss of Wild-Type Jak2 Allele Enhances Myeloid Cell Expansion and Accelerates Myelofibrosis in Jak2V617F Knock-in Mice

Hajime Akada, Saeko Akada, Dongqing Yan, Robert Hutchison, MD, Golam Mohi, PhD

Blood (2012) 120 (21): 809.

https://doi.org/10.1182/blood.V120.21.809.809

Published: 2012

NARROW

Genetic Deletion or Pharmacologic Inhibition of PTPN11 Impedes the Development and Progression of Myeloproliferative Neoplasms Induced By JAK2V617F and MPLW515L Mutants

PTPN1 Deficiency Accelerates the Progression to Myelofibrosis in JAK2V617F-Driven Myeloproliferative Neoplasm

IL-1 Signaling Promotes Clonal Expansion and Progression of Bone Marrow Fibrosis in JAK2V617F-Induced Myeloproliferative Neoplasm

Inhibition of SHP2 Ameliorates JAK2V617F-Induced Myeloproliferative Neoplasms

The RNA Splicing Factor U2AF1 Controls Hematopoietic Stem Cell Survival and Function

A Phase 1 Study of TP-3654, an Orally-Delivered PIM Kinase Inhibitor, in Patients with Intermediate-2 or High-Risk Primary or Secondary Myelofibrosis

Critical Requirement of U2AF1 in the Maintenance and Function of Hematopoietic Stem Cells

The CDK6 Inhibitor Palbociclib in Combination with Ruxolitinib Remarkably Improves Myelofibrosis in Murine Models

The NF-KB Inhibitor DMAPT in Combination with Ruxolitinib Displays Efficacy in Jak2V617F Knock-in Mouse Model of Myeloproliferative Neoplasms

The PIM Kinase Inhibitor TP-3654 in Combination with Ruxolitinib Exhibits Marked Improvement of Myelofibrosis in Murine Models

Hmga2 promotes the development of myelofibrosis in Jak2 V617F knockin mice by enhancing TGF-β1 and Cxcl12 pathways

Deletion of PTPN1 Promotes the Development of Myelofibrosis

Expression of HMGA2 Cooperates with Jak2V617F in the Development of Myelofibrosis

Loss of Ezh2 cooperates with Jak2V617F in the development of myelofibrosis in a mouse model of myeloproliferative neoplasm

Distinct GAB2 signaling pathways are essential for myeloid and lymphoid transformation and leukemogenesis by BCR-ABL1

Critical Role of SHP2 in the Initiation and Maintenance of Myeloproliferative Neoplasms Evoked By JAK2V617F

Loss of EZH2 Inhibits Erythropoiesis and Accelerates the Development of Myelofibrosis in Jak2V617F Knock-in Mice

Stat3 Negatively Regulates Myeloproliferative Neoplasm Induced By Jak2V617F

Critical Role Of Jak2 In The Maintenance and Function Of Adult Hematopoietic Stem Cells

Loss of Wild-Type Jak2 Allele Enhances Myeloid Cell Expansion and Accelerates Myelofibrosis in Jak2V617F Knock-in Mice

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American Society of Hematology

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American Society of Hematology

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