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Journal Articles

Mast cells and neutrophils proteolytically activate chemokine precursor CTAP-III and are subject to counterregulation by PF-4 through inhibition of chymase and cathepsin G

Florian Schiemann, Tobias Alexander Grimm, Josef Hoch, Roland Gross, Buko Lindner, Frank Petersen, Silvia Bulfone-Paus, Ernst Brandt

Blood (2006) 107 (6): 2234–2242.

https://doi.org/10.1182/blood-2005-06-2424

Published: 2006

Journal Articles

Neutrophil adhesion to endothelial cells induced by platelet factor 4 requires sequential activation of Ras, Syk, and JNK MAP kinases

Brigitte Kasper, Ernst Brandt, Martin Ernst, Frank Petersen

Blood (2006) 107 (5): 1768–1775.

https://doi.org/10.1182/blood-2005-06-2501

Published: 2006

Includes: Supplemental data

Journal Articles

Platelet factor 4 (PF-4)–induced neutrophil adhesion is controlled by src-kinases, whereas PF-4–mediated exocytosis requires the additional activation of p38 MAP kinase and phosphatidylinositol 3-kinase

Brigitte Kasper, Ernst Brandt, Silvia Bulfone-Paus, Frank Petersen

Blood (2004) 103 (5): 1602–1610.

https://doi.org/10.1182/blood-2003-08-2802

Published: 2004

Journal Articles

The CXC-chemokine platelet factor 4 promotes monocyte survival and induces monocyte differentiation into macrophages

Barbara Scheuerer, Martin Ernst, Iris Dürrbaum-Landmann, Jens Fleischer, Evelin Grage-Griebenow, Ernst Brandt, Hans-Dieter Flad, Frank Petersen

Blood (2000) 95 (4): 1158–1166.

https://doi.org/10.1182/blood.V95.4.1158.004k31_1158_1166

Published: 2000

Journal Articles

Platelet Factor 4-Induced Neutrophil-Endothelial Cell Interaction: Involvement of Mechanisms and Functional Consequences Different From Those Elicited by Interleukin-8

Frank Petersen, Lothar Bock, Hans-Dieter Flad, Ernst Brandt

Blood (1999) 94 (12): 4020–4028.

https://doi.org/10.1182/blood.V94.12.4020

Published: 1999

Journal Articles

The CXC-Chemokine Neutrophil-Activating Peptide-2 Induces Two Distinct Optima of Neutrophil Chemotaxis by Differential Interaction With Interleukin-8 Receptors CXCR-1 and CXCR-2

Andreas Ludwig, Frank Petersen, Stefan Zahn, Otto Götze, Jens-Michael Schröder, Hans-Dieter Flad, Ernst Brandt

Blood (1997) 90 (11): 4588–4597.

https://doi.org/10.1182/blood.V90.11.4588

Published: 1997

NARROW

Mast cells and neutrophils proteolytically activate chemokine precursor CTAP-III and are subject to counterregulation by PF-4 through inhibition of chymase and cathepsin G

Neutrophil adhesion to endothelial cells induced by platelet factor 4 requires sequential activation of Ras, Syk, and JNK MAP kinases

Platelet factor 4 (PF-4)–induced neutrophil adhesion is controlled by src-kinases, whereas PF-4–mediated exocytosis requires the additional activation of p38 MAP kinase and phosphatidylinositol 3-kinase

The CXC-chemokine platelet factor 4 promotes monocyte survival and induces monocyte differentiation into macrophages

Platelet Factor 4-Induced Neutrophil-Endothelial Cell Interaction: Involvement of Mechanisms and Functional Consequences Different From Those Elicited by Interleukin-8

The CXC-Chemokine Neutrophil-Activating Peptide-2 Induces Two Distinct Optima of Neutrophil Chemotaxis by Differential Interaction With Interleukin-8 Receptors CXCR-1 and CXCR-2

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ASH Publications

American Society of Hematology

Mast cells and neutrophils proteolytically activate chemokine precursor CTAP-III and are subject to counterregulation by PF-4 through inhibition of chymase and cathepsin G

Neutrophil adhesion to endothelial cells induced by platelet factor 4 requires sequential activation of Ras, Syk, and JNK MAP kinases

Platelet factor 4 (PF-4)–induced neutrophil adhesion is controlled by src-kinases, whereas PF-4–mediated exocytosis requires the additional activation of p38 MAP kinase and phosphatidylinositol 3-kinase

The CXC-chemokine platelet factor 4 promotes monocyte survival and induces monocyte differentiation into macrophages

Platelet Factor 4-Induced Neutrophil-Endothelial Cell Interaction: Involvement of Mechanisms and Functional Consequences Different From Those Elicited by Interleukin-8

The CXC-Chemokine Neutrophil-Activating Peptide-2 Induces Two Distinct Optima of Neutrophil Chemotaxis by Differential Interaction With Interleukin-8 Receptors CXCR-1 and CXCR-2

Email alerts

ASH Publications

American Society of Hematology

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