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Journal Articles

Bone marrow failure unresponsive to bone marrow transplant is caused by mutations in thrombopoietin

Blood (2017) 130 (7): 875–880.

https://doi.org/10.1182/blood-2017-02-768036

Published: 2017

Includes: Supplemental data

Journal Articles

Mpl Gain-of-Function Mutations Can be Classified By Differential Subcellular Processing, Molecular Mechanisms, Mode of Inheritance and Clinical Impact

Clemens Stockklausner, MD, Christin Maria Duffert, Ziwei Zhou, Anne Christine Klotter, Isabelle Nadine Kuhlee, Andreas E Kulozik, MDPhD

Blood (2015) 126 (23): 1634.

https://doi.org/10.1182/blood.V126.23.1634.1634

Published: 2015

Journal Articles

The thrombopoietin receptor P106L mutation functionally separates receptor signaling activity from thrombopoietin homeostasis

Clemens Stockklausner, Anne-Christine Klotter, Nicole Dickemann, Isabelle N. Kuhlee, Christin M. Duffert, Carolin Kerber, Niels H. Gehring, Andreas E. Kulozik

Blood (2015) 125 (7): 1159–1169.

https://doi.org/10.1182/blood-2014-07-587170

Published: 2015

Includes: Supplemental data

Journal Articles

The Predominant Activation of Alternative Signaling Pathways Explains the Differential Propensity of Different c-Mpl Gain-of-Function Mutations to Promote Myeloproliferative Malignancies

Clemens Stockklausner, MD, Christin Maria Duffert, Nicole Dickemann, Anne Christine Klotter, Isabelle Nadine Kuhlee, Carolin Kerber, Andreas E. Kulozik, MD PhD

Blood (2014) 124 (21): 1578.

https://doi.org/10.1182/blood.V124.21.1578.1578

Published: 2014

Journal Articles

Hereditary Thrombocytosis: The Thrombopoietin Receptor c-MPL Can Activate Its Downstream Pathway without Full Glycosylation and Membrane Expression

Clemens Stockklausner, M.D., Nicole Echner, Anne-Christine Klotter, Isabelle Nadine Kuhlee, Andreas E Kulozik, MD, PhD

Blood (2011) 118 (21): 2374.

https://doi.org/10.1182/blood.V118.21.2374.2374

Published: 2011

Journal Articles

Autosomal Recessive Hereditary Thrombocytosis: Functional Analysis of a Novel C-Mpl Mutation Resulting in An Unsual Mode of Constitutive Receptor Activation

Clemens Stockklausner, Niels Gehring, Nicole Echner, Naima Ali Al Mulla, Andreas E. Kulozik

Blood (2008) 112 (11): 890.

https://doi.org/10.1182/blood.V112.11.890.890

Published: 2008

NARROW

Bone marrow failure unresponsive to bone marrow transplant is caused by mutations in thrombopoietin

Mpl Gain-of-Function Mutations Can be Classified By Differential Subcellular Processing, Molecular Mechanisms, Mode of Inheritance and Clinical Impact

The thrombopoietin receptor P106L mutation functionally separates receptor signaling activity from thrombopoietin homeostasis

The Predominant Activation of Alternative Signaling Pathways Explains the Differential Propensity of Different c-Mpl Gain-of-Function Mutations to Promote Myeloproliferative Malignancies

Hereditary Thrombocytosis: The Thrombopoietin Receptor c-MPL Can Activate Its Downstream Pathway without Full Glycosylation and Membrane Expression

Autosomal Recessive Hereditary Thrombocytosis: Functional Analysis of a Novel C-Mpl Mutation Resulting in An Unsual Mode of Constitutive Receptor Activation

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ASH Publications

American Society of Hematology

Bone marrow failure unresponsive to bone marrow transplant is caused by mutations in thrombopoietin

Mpl Gain-of-Function Mutations Can be Classified By Differential Subcellular Processing, Molecular Mechanisms, Mode of Inheritance and Clinical Impact

The thrombopoietin receptor P106L mutation functionally separates receptor signaling activity from thrombopoietin homeostasis

The Predominant Activation of Alternative Signaling Pathways Explains the Differential Propensity of Different c-Mpl Gain-of-Function Mutations to Promote Myeloproliferative Malignancies

Hereditary Thrombocytosis: The Thrombopoietin Receptor c-MPL Can Activate Its Downstream Pathway without Full Glycosylation and Membrane Expression

Autosomal Recessive Hereditary Thrombocytosis: Functional Analysis of a Novel C-Mpl Mutation Resulting in An Unsual Mode of Constitutive Receptor Activation

Email alerts

ASH Publications

American Society of Hematology

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