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Journal Articles

Analysis of clonotypic switch junctions reveals multiple myeloma originates from a single class switch event with ongoing mutation in the isotype-switched progeny

Brian J. Taylor, Jitra Kriangkum, Julie A. Pittman, Michael J. Mant, Tony Reiman, Andrew R. Belch, Linda M. Pilarski

Blood (2008) 112 (5): 1894–1903.

https://doi.org/10.1182/blood-2008-01-129221

Published: 2008

Journal Articles

Genetic Variations in Genomic HAS1 Direct Aberrant Pre-mRNA Splicing That Generates the Prognostically Significant HAS1Vb Intronic Splice Variant.

Jitra Kriangkum, PhD, Anirban Ghosh, PhD, Brian J. Taylor, PhD, Linda M. Pilarski, PhD

Blood (2007) 110 (11): 2497.

https://doi.org/10.1182/blood.V110.11.2497.2497

Published: 2007

Journal Articles

Molecular Characterization of Clonotypic VDJ Sequences in Waldenstrom’s Macroglobulinemia Suggests Independent Transformation Events in the Development of Biclonal B Cells.

Jitra Kriangkum, PhD, Brian J. Taylor, PhD, Steven P. Treon, MD, PhD, Michael J. Mant, MD, Tony Reiman, MD, Andrew R. Belch, MD, Linda M. Pilarski, PhD

Blood (2006) 108 (11): 4624.

https://doi.org/10.1182/blood.V108.11.4624.4624

Published: 2006

Journal Articles

Genomic Instability in Multiple Myeloma: Inducible Transfectants Provide a Model To Define the Role of RHAMM and Centrosome/Mitotic Spindle Stability in Myelomagenesis.

Brian J. Taylor, PhD, Eva Baigorri, MSc, Mary Hay, PhD, Tony Reiman, MD, Andrew R. Belch, MD, Linda M. Pilarski, PhD

Blood (2006) 108 (11): 5037.

https://doi.org/10.1182/blood.V108.11.5037.5037

Published: 2006

Journal Articles

Impaired class switch recombination (CSR) in Waldenström macroglobulinemia (WM) despite apparently normal CSR machinery

Jitra Kriangkum, Brian J. Taylor, Erin Strachan, Michael J. Mant, Tony Reiman, Andrew R. Belch, Linda M. Pilarski

Blood (2006) 107 (7): 2920–2927.

https://doi.org/10.1182/blood-2005-09-3613

Published: 2006

Journal Articles

Analysis of Clonotypic Switch Junctions Reveals Multiple Myeloma Originates from a Single Class Switch Event with Ongoing Mutation in the Isotype Switched Progeny.

Brian J. Taylor, PhD, Jitra Kriangkum, PhD, Jonathan J. Keats, PhD, Julie Pittman, BSc, Tony Reiman, MD, Andrew R. Belch, MD, Linda M. Pilarski, PhD

Blood (2005) 106 (11): 360.

https://doi.org/10.1182/blood.V106.11.360.360

Published: 2005

Journal Articles

Microfluidic Devices for Molecular Monitoring of Clonotypic IgH VDJ Signatures and the T(4;14) Translocation in Multiple Myeloma.

Linda M. Pilarski, PhD, Govindkrishna V. Kaigala, Jaron VanDjiken, Alexey Atrazhev, PhD, Brian J. Taylor, PhD, Andrew R. Belch, MD, Christopher J. Backhouse, PhD

Blood (2005) 106 (11): 1536.

https://doi.org/10.1182/blood.V106.11.1536.1536

Published: 2005

Journal Articles

Clonotypic IgG B Cells in Waldenstrom Macroglobulinemia Cells Are Derived from Multiple Post-Transformed B Cells That Have Undergone Isotype Switch Recombination.

Jitra Kriangkum, PhD, Brian J. Taylor, PhD, Erin Strachan, BSc, Tony Reiman, MD, Andrew R. Belch, MD, Michael J. Mant, MD, Linda M. Pilarski, PhD

Blood (2005) 106 (11): 1002.

https://doi.org/10.1182/blood.V106.11.1002.1002

Published: 2005

Journal Articles

Analysis of Validated Clonotypic Igh VDJ Sequences from a Large Cohort of MM Patients.

Brian J. Taylor, PhD, Jonathan J. Keats, PhD, Erin R. Strachan, B.Sc, Tony Reiman, MD, Andrew R. Belch, MD, Linda M. Pilarski, PhD

Blood (2005) 106 (11): 1543.

https://doi.org/10.1182/blood.V106.11.1543.1543

Published: 2005

Journal Articles

Overexpression of transcripts originating from the MMSET locus characterizes all t(4;14)(p16;q32)-positive multiple myeloma patients

Jonathan J. Keats, Christopher A. Maxwell, Brian J. Taylor, Michael J. Hendzel, Marta Chesi, P. Leif Bergsagel, Loree M. Larratt, Michael J. Mant, Tony Reiman, Andrew R. Belch, Linda M. Pilarski

Blood (2005) 105 (10): 4060–4069.

https://doi.org/10.1182/blood-2004-09-3704

Published: 2005

Journal Articles

Mutator Genes UDG and AID Appear To Be Normal in Class-Switch Deficient and Clonally Homogeneous Waldenstrom’s Macroglobulinemias Having Either Germline or Hypermutated Clonotypic IgH VDJ.

Jitra Kriangkum, PhD, Brian J. Taylor, PhD, Erin R. Strachan, BSc, Steven P. Treon, MD, PhD, Michael J. Mant, MD, Tony Reiman, MD, Andrew R. Belch, MD, Linda M. Pilarski, PhD

Blood (2004) 104 (11): 1359.

https://doi.org/10.1182/blood.V104.11.1359.1359

Published: 2004

Journal Articles

VDJ-Switch Region Analysis in Multiple Myeloma Patients Reveals Homogeneity and Long-Term Stability of Switch Junctions, and Ongoing Mutation Upstream of Switch Mu.

Brian J. Taylor, Ph.D., Julie A. Pittman, B. Sc., Andrew R. Belch, M.D., Linda M. Pilarski, Ph.D.

Blood (2004) 104 (11): 1414.

https://doi.org/10.1182/blood.V104.11.1414.1414

Published: 2004

Journal Articles

Clonal Analysis of IgM Cells in Multiple Myeloma Patients Suggests the Co-Existence of Normal and Malignant Arms of the Myeloma Clone.

Brian J. Taylor, Ph. D., Ming Ye, B.Sc., Erin R. Strachan, B. Sc., Tara M. Tiffinger, Andrew R. Belch, M.D., Linda M. Pilarski, Ph. D.

Blood (2004) 104 (11): 1415.

https://doi.org/10.1182/blood.V104.11.1415.1415

Published: 2004

Journal Articles

Altered Kinetics and Localization of MMSET Variants Suggests That RE-IIBP Overexpression Is a Unifying Event in t(4;14)(p16;q32) Multiple Myeloma.

Jonathan J. Keats, Christopher A. Maxwell, PhD, Tony Reiman, MD, Brian J. Taylor, PhD, Michael J. Mant, MD, Andrew J. Belch, MD, Linda M. Pilarski, PhD

Blood (2004) 104 (11): 75.

https://doi.org/10.1182/blood.V104.11.75.75

Published: 2004

Journal Articles

Clonotypic IgM V/D/J sequence analysis in Waldenstrom macroglobulinemia suggests an unusual B-cell origin and an expansion of polyclonal B cells in peripheral blood

Jitra Kriangkum, Brian J. Taylor, Steven P. Treon, Michael J. Mant, Andrew R. Belch, Linda M. Pilarski

Blood (2004) 104 (7): 2134–2142.

https://doi.org/10.1182/blood-2003-11-4024

Published: 2004

Journal Articles

In multiple myeloma, t(4;14)(p16;q32) is an adverse prognostic factor irrespective of FGFR3 expression

Jonathan J. Keats, Tony Reiman, Christopher A. Maxwell, Brian J. Taylor, Loree M. Larratt, Michael J. Mant, Andrew R. Belch, Linda M. Pilarski

Blood (2003) 101 (4): 1520–1529.

https://doi.org/10.1182/blood-2002-06-1675

Published: 2003

Journal Articles

Persistent preswitch clonotypic myeloma cells correlate with decreased survival: evidence for isotype switching within the myeloma clone

Tony Reiman, Karen Seeberger, Brian J. Taylor, Agnieszka J. Szczepek, John Hanson, Michael J. Mant, Robert W. Coupland, Andrew R. Belch, Linda M. Pilarski

Blood (2001) 98 (9): 2791–2799.

https://doi.org/10.1182/blood.V98.9.2791

Published: 2001

NARROW

Analysis of clonotypic switch junctions reveals multiple myeloma originates from a single class switch event with ongoing mutation in the isotype-switched progeny

Genetic Variations in Genomic HAS1 Direct Aberrant Pre-mRNA Splicing That Generates the Prognostically Significant HAS1Vb Intronic Splice Variant.

Molecular Characterization of Clonotypic VDJ Sequences in Waldenstrom’s Macroglobulinemia Suggests Independent Transformation Events in the Development of Biclonal B Cells.

Genomic Instability in Multiple Myeloma: Inducible Transfectants Provide a Model To Define the Role of RHAMM and Centrosome/Mitotic Spindle Stability in Myelomagenesis.

Impaired class switch recombination (CSR) in Waldenström macroglobulinemia (WM) despite apparently normal CSR machinery

Analysis of Clonotypic Switch Junctions Reveals Multiple Myeloma Originates from a Single Class Switch Event with Ongoing Mutation in the Isotype Switched Progeny.

Microfluidic Devices for Molecular Monitoring of Clonotypic IgH VDJ Signatures and the T(4;14) Translocation in Multiple Myeloma.

Clonotypic IgG B Cells in Waldenstrom Macroglobulinemia Cells Are Derived from Multiple Post-Transformed B Cells That Have Undergone Isotype Switch Recombination.

Analysis of Validated Clonotypic Igh VDJ Sequences from a Large Cohort of MM Patients.

Overexpression of transcripts originating from the MMSET locus characterizes all t(4;14)(p16;q32)-positive multiple myeloma patients

Mutator Genes UDG and AID Appear To Be Normal in Class-Switch Deficient and Clonally Homogeneous Waldenstrom’s Macroglobulinemias Having Either Germline or Hypermutated Clonotypic IgH VDJ.

VDJ-Switch Region Analysis in Multiple Myeloma Patients Reveals Homogeneity and Long-Term Stability of Switch Junctions, and Ongoing Mutation Upstream of Switch Mu.

Clonal Analysis of IgM Cells in Multiple Myeloma Patients Suggests the Co-Existence of Normal and Malignant Arms of the Myeloma Clone.

Altered Kinetics and Localization of MMSET Variants Suggests That RE-IIBP Overexpression Is a Unifying Event in t(4;14)(p16;q32) Multiple Myeloma.

Clonotypic IgM V/D/J sequence analysis in Waldenstrom macroglobulinemia suggests an unusual B-cell origin and an expansion of polyclonal B cells in peripheral blood

In multiple myeloma, t(4;14)(p16;q32) is an adverse prognostic factor irrespective of FGFR3 expression

Persistent preswitch clonotypic myeloma cells correlate with decreased survival: evidence for isotype switching within the myeloma clone

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American Society of Hematology

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ASH Publications

American Society of Hematology

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