Abstract
In genome-wide analyses of DNA copy number abnormalities (CNAs) and loss of heterozygosity (LOH) using single nucleotide polymorphism (SNP) microarrays, we previously reported a high frequency of genetic alterations of regulators of B lymphoid development and cell cycle in B-progenitor acute lymphoblastic leukemia (B-ALL), and a near-obligate deletion of the transcription factor Ikaros (IKZF1) in BCR-ABL1 ALL (
Nature
2007
;446
:758
Nature
2008
;453
:110
Nature
2008
;453
:110
Disclosures: No relevant conflicts of interest to declare.
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Corresponding author
2008, The American Society of Hematology
2008