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Journal Articles

Extracellular Hemoglobin Increases the Binding of the Active Form of Von Willebrand Factor to Glycoprotein Ibα and Platelet Activation Under Shear Stress: Implications for Sickle Cell Disease and Other Hemolytic Disorders

Miguel A. Cruz, PhD,, Prasenjit Guchhait, PhD, Ryanne Ashley Brown

Blood (2011) 118 (21): 2095.

https://doi.org/10.1182/blood.V118.21.2095.2095

Published: 2011

Journal Articles

Hemoglobin Bound Von Willebrand Factor (HbVWF) Multimers Are Hyperactive and More Prevalent in Patients with Sickle Cell Disease.

Zhou Zhou, PhD, Hyojeong Han, Mark M. Udden, MD, Miguel A. Cruz, PhD, Jing-fei Dong, MD, PhD, Prasenjit Guchhait, PhD

Blood (2009) 114 (22): 3058.

https://doi.org/10.1182/blood.V114.22.3058.3058

Published: 2009

Journal Articles

Hemoglobin Blocks Von Willebrand Factor Proteolysis by ADAMTS-13: A Mechanism Associated with Acquired ADAMTS-13 Deficiency in Patients with Sickle Cell Disease

Zhou Zhou, Han Hyojeong, Miguel A. Cruz, PhD, Jose A. Lopez, MD, Jing-fei Dong, MD, PhD, Prasenjit Guchhait, PhD

Blood (2008) 112 (11): 3919.

https://doi.org/10.1182/blood.V112.11.3919.3919

Published: 2008

Journal Articles

Cell Membrane Sulfatide Promotes Sickle Cell Adhesion to Endothelium.

Prasenjit Guchhait, PhD, Perumal Thiagarajan, M.D., Jose A. Lopez, M.D.

Blood (2007) 110 (11): 1722.

https://doi.org/10.1182/blood.V110.11.1722.1722

Published: 2007

Journal Articles

Lactadherin Enhances the Adhesion of Sickle Cells to Vascular Endothelial Cells.

Prasenjit Guchhait, PhD, Swapan K. Dasgupta, Ph.D, Jose A. Lopez, MD, Perumal Thiagarajan, MD

Blood (2005) 106 (11): 2341.

https://doi.org/10.1182/blood.V106.11.2341.2341

Published: 2005

Journal Articles

Superoxide Inhibits ADAMTS-13 Activity and Regulates Sickle Cell Adhesion.

Prasenjit Guchhait, PhD, Miguel A. Cruz, PhD, Jing-Fei Dong, MD, PhD, José A. López, MD

Blood (2005) 106 (11): 2342.

https://doi.org/10.1182/blood.V106.11.2342.2342

Published: 2005

NARROW

Extracellular Hemoglobin Increases the Binding of the Active Form of Von Willebrand Factor to Glycoprotein Ibα and Platelet Activation Under Shear Stress: Implications for Sickle Cell Disease and Other Hemolytic Disorders

Hemoglobin Bound Von Willebrand Factor (HbVWF) Multimers Are Hyperactive and More Prevalent in Patients with Sickle Cell Disease.

Hemoglobin Blocks Von Willebrand Factor Proteolysis by ADAMTS-13: A Mechanism Associated with Acquired ADAMTS-13 Deficiency in Patients with Sickle Cell Disease

Cell Membrane Sulfatide Promotes Sickle Cell Adhesion to Endothelium.

Lactadherin Enhances the Adhesion of Sickle Cells to Vascular Endothelial Cells.

Superoxide Inhibits ADAMTS-13 Activity and Regulates Sickle Cell Adhesion.

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ASH Publications

American Society of Hematology

Extracellular Hemoglobin Increases the Binding of the Active Form of Von Willebrand Factor to Glycoprotein Ibα and Platelet Activation Under Shear Stress: Implications for Sickle Cell Disease and Other Hemolytic Disorders

Hemoglobin Bound Von Willebrand Factor (HbVWF) Multimers Are Hyperactive and More Prevalent in Patients with Sickle Cell Disease.

Hemoglobin Blocks Von Willebrand Factor Proteolysis by ADAMTS-13: A Mechanism Associated with Acquired ADAMTS-13 Deficiency in Patients with Sickle Cell Disease

Cell Membrane Sulfatide Promotes Sickle Cell Adhesion to Endothelium.

Lactadherin Enhances the Adhesion of Sickle Cells to Vascular Endothelial Cells.

Superoxide Inhibits ADAMTS-13 Activity and Regulates Sickle Cell Adhesion.

Email alerts

ASH Publications

American Society of Hematology

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