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Journal Articles

A New Genetic Immunodeficiency Syndrome Caused By Autosomal Dominant Mutations in LCP1 Identified By Mosaic Tetraploidy and Severe Neutropenia in Unrelated Pedigrees

Thijs van Bergen, Anna Aalbers, Dennis Bosch, Onno Roovers, Akiko Shimamura, Marc H.G.P. Raaijmakers, Peter E. Newburger, Ivo P Touw

Blood (2022) 140 (Supplement 1): 5463–5464.

https://doi.org/10.1182/blood-2022-165112

Published: 2022

Journal Articles

PML Plays a Key Role in Severe Congenital Neutropenia with Mutant ELANE Causing Neutrophil Elastase Protein Misfolding

Patricia Olofsen, MSc, Paulette van Strien, BSc, Onno Roovers, BSc, Hans De Looper, BSc, Remco Hoogenboezem, MSc, Dennis Bosch, BSc, Mehrnaz Ghazvini, PhD, Eric Bindels, PhD, Emma De Pater, PhD, Ivo Touw, PhD

Blood (2019) 134 (Supplement_1): 213.

https://doi.org/10.1182/blood-2019-122423

Published: 2019

Journal Articles

A Leukemic Progression Model of Severe Congenital Neutropenia Uncovers a Novel Mechanism of AML Development Involving Elevated Inflammatory Responses, Mutation of CXXC4 and Decreased TET2 Levels

Patricia Olofsen, MSc, Szabolcs Fatrai, PhD, Paulette van Strien, BSc, Julia Obenauer, MSc, Remco Hoogenboezem, MSc, Claudia Erpelinck-Verschueren, B.Sc., Onno Roovers, BSc, Torsten Haferlach, MD, Peter Valk, PhD, Rebekka Schneider, MDPhD, Emma De Pater, PhD, Ivo Touw, PhD

Blood (2018) 132 (Supplement 1): 540.

https://doi.org/10.1182/blood-2018-99-115914

Published: 2018

Journal Articles

Targeted Therapy for ETV6-RUNX1-Driven B-Cell Precursor Acute Lymphoblastic Leukemia

Roel Polak, MD, Marc B. Bierings, MD PhD, Cindy S. van der Leije, MSc, Rosanna E.S. van den Dungen, Mathijs A. Sanders, MSc, Onno Roovers, Jan J. Cornelissen, MD PhD, João R.M. Marchante, MSc, Rob Pieters, MD PhD, Monique L. den Boer, PhD, Miranda Buitenhuis, PhD

Blood (2014) 124 (21): 62.

https://doi.org/10.1182/blood.V124.21.62.62

Published: 2014

Journal Articles

Characterization of a Juxtamembrane Lysine as a Determinant in Lysosomal Routing and Signal Downregulation of the Activated G-CSF Receptor (G-CSFR).

Mahban Irandoust, MSc, Albert Wolfler, MD, Judith Gits, Onno Roovers, Ivo P. Touw, PhD

Blood (2007) 110 (11): 2190.

https://doi.org/10.1182/blood.V110.11.2190.2190

Published: 2007

Journal Articles

G-CSF Receptor Lysine Residues Are Involved in Lysosomal Degradation and Attenuation of G-CSF Signaling Via Distinct Effector Mechanisms.

Lambertus H. Aarts, PhD, Mahban Irandoust, MSc, Onno Roovers, BSc, Marijke Valkhof, BSc, Stefan J. Erkeland, PhD, Ivo P. Touw, PhD

Blood (2005) 106 (11): 3155.

https://doi.org/10.1182/blood.V106.11.3155.3155

Published: 2005

Journal Articles

Receptor activation and 2 distinct COOH-terminal motifs control G-CSF receptor distribution and internalization kinetics

Lambertus H. J. Aarts, Onno Roovers, Alister C. Ward, Ivo P. Touw

Blood (2004) 103 (2): 571–579.

https://doi.org/10.1182/blood-2003-07-2250

Published: 2004

NARROW

A New Genetic Immunodeficiency Syndrome Caused By Autosomal Dominant Mutations in LCP1 Identified By Mosaic Tetraploidy and Severe Neutropenia in Unrelated Pedigrees

PML Plays a Key Role in Severe Congenital Neutropenia with Mutant ELANE Causing Neutrophil Elastase Protein Misfolding

A Leukemic Progression Model of Severe Congenital Neutropenia Uncovers a Novel Mechanism of AML Development Involving Elevated Inflammatory Responses, Mutation of CXXC4 and Decreased TET2 Levels

Targeted Therapy for ETV6-RUNX1-Driven B-Cell Precursor Acute Lymphoblastic Leukemia

Characterization of a Juxtamembrane Lysine as a Determinant in Lysosomal Routing and Signal Downregulation of the Activated G-CSF Receptor (G-CSFR).

G-CSF Receptor Lysine Residues Are Involved in Lysosomal Degradation and Attenuation of G-CSF Signaling Via Distinct Effector Mechanisms.

Receptor activation and 2 distinct COOH-terminal motifs control G-CSF receptor distribution and internalization kinetics

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ASH Publications

American Society of Hematology

A New Genetic Immunodeficiency Syndrome Caused By Autosomal Dominant Mutations in LCP1 Identified By Mosaic Tetraploidy and Severe Neutropenia in Unrelated Pedigrees

PML Plays a Key Role in Severe Congenital Neutropenia with Mutant ELANE Causing Neutrophil Elastase Protein Misfolding

A Leukemic Progression Model of Severe Congenital Neutropenia Uncovers a Novel Mechanism of AML Development Involving Elevated Inflammatory Responses, Mutation of CXXC4 and Decreased TET2 Levels

Targeted Therapy for ETV6-RUNX1-Driven B-Cell Precursor Acute Lymphoblastic Leukemia

Characterization of a Juxtamembrane Lysine as a Determinant in Lysosomal Routing and Signal Downregulation of the Activated G-CSF Receptor (G-CSFR).

G-CSF Receptor Lysine Residues Are Involved in Lysosomal Degradation and Attenuation of G-CSF Signaling Via Distinct Effector Mechanisms.

Receptor activation and 2 distinct COOH-terminal motifs control G-CSF receptor distribution and internalization kinetics

Email alerts

ASH Publications

American Society of Hematology

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