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Journal Articles

Endothelial JAK2 V617F Expression Drives Inflammation and Cellular Senescence; New Evidence for the Roles of Endothelial Cells in MPN-Related Clotting Abnormalities?

Megan E Cosgrove, PhD, Rakesh Suman, PhD, Helen J Harrison, RN, Gillian E Jackson, RN, Martin R Howard, MBChB, MD FRCP, FRCPath, Ian S Hitchcock, PhD

Blood (2016) 128 (22): 3134.

https://doi.org/10.1182/blood.V128.22.3134.3134

Published: 2016

Journal Articles

Lineage Restricted JAK2V617F Expression and Its Role in Thrombosis and Hemostasis; The Potential Involvement of Endothelial JAK2V617F in Clotting Abnormalities

Ian S Hitchcock, PhD, Michelle E Roh, BS, Veena Sangkhae, BS, Leah Etheridge, PhD, Rose Chen, BS, Norma E Fox, BS, Kenneth Kaushansky, MD

Blood (2011) 118 (21): 1737.

https://doi.org/10.1182/blood.V118.21.1737.1737

Published: 2011

Journal Articles

A Novel Activating JAK2 Mutation, JAK2R564Q, Causes Familial Essential Thrombocytosis (fET) Via Mechanisms Distinct From JAK2V617F

Leah Etheridge, PhD, Lana M Corbo, BS, Kenneth Kaushansky, MD, Edward Chan, MD, Ian S Hitchcock, PhD

Blood (2011) 118 (21): 123.

https://doi.org/10.1182/blood.V118.21.123.123

Published: 2011

Journal Articles

YRRL Motifs in the Cytoplasmic Domain of the Thrombopoietin Receptor Mpl Regulate Receptor Internalization and Degradation.

Ian S. Hitchcock, PhD, Maximus M. Chen, Jennifer R. King, Kenneth Kaushansky, M.D

Blood (2007) 110 (11): 90.

https://doi.org/10.1182/blood.V110.11.90.90

Published: 2007

Journal Articles

Thrombopoietin Promotes b1 Integrin-Mediated Adhesion in Hematopoietic Cells Via the Small GTPase Rap1.

Ian S. Hitchcock, PhD, Kenneth Kaushansky, MD

Blood (2006) 108 (11): 1393.

https://doi.org/10.1182/blood.V108.11.1393.1393

Published: 2006

Journal Articles

Focal Adhesion Kinase in Megakaryopoiesis: A Study of Megakaryocyte-Specific FAK Deletion.

Ian S. Hitchcock, PhD, Norma E. Fox, Katherine Sear, Ralph Tiedt, Radek Skoda, Kenneth Kaushansky

Blood (2006) 108 (11): 354.

https://doi.org/10.1182/blood.V108.11.354.354

Published: 2006

NARROW

Endothelial JAK2 V617F Expression Drives Inflammation and Cellular Senescence; New Evidence for the Roles of Endothelial Cells in MPN-Related Clotting Abnormalities?

Lineage Restricted JAK2V617F Expression and Its Role in Thrombosis and Hemostasis; The Potential Involvement of Endothelial JAK2V617F in Clotting Abnormalities

A Novel Activating JAK2 Mutation, JAK2R564Q, Causes Familial Essential Thrombocytosis (fET) Via Mechanisms Distinct From JAK2V617F

YRRL Motifs in the Cytoplasmic Domain of the Thrombopoietin Receptor Mpl Regulate Receptor Internalization and Degradation.

Thrombopoietin Promotes b1 Integrin-Mediated Adhesion in Hematopoietic Cells Via the Small GTPase Rap1.

Focal Adhesion Kinase in Megakaryopoiesis: A Study of Megakaryocyte-Specific FAK Deletion.

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ASH Publications

American Society of Hematology

Endothelial JAK2 V617F Expression Drives Inflammation and Cellular Senescence; New Evidence for the Roles of Endothelial Cells in MPN-Related Clotting Abnormalities?

Lineage Restricted JAK2V617F Expression and Its Role in Thrombosis and Hemostasis; The Potential Involvement of Endothelial JAK2V617F in Clotting Abnormalities

A Novel Activating JAK2 Mutation, JAK2R564Q, Causes Familial Essential Thrombocytosis (fET) Via Mechanisms Distinct From JAK2V617F

YRRL Motifs in the Cytoplasmic Domain of the Thrombopoietin Receptor Mpl Regulate Receptor Internalization and Degradation.

Thrombopoietin Promotes b1 Integrin-Mediated Adhesion in Hematopoietic Cells Via the Small GTPase Rap1.

Focal Adhesion Kinase in Megakaryopoiesis: A Study of Megakaryocyte-Specific FAK Deletion.

Email alerts

ASH Publications

American Society of Hematology

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