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Journal Articles

Gene Expression Profiling of Day 7 Erythroblasts from RARS Before and after Treatment with G-CSF.

Maryam Nikpour, MSc, Andrea Pellagatti, PhD, Martin Jädersten, MD, Ann-Mari Forsblom, James S. Wainscoat, MD, Jacqueline Boultwood, PhD, Eva Hellström-Lindberg, MD, PhD

Blood (2007) 110 (11): 2427.

https://doi.org/10.1182/blood.V110.11.2427.2427

Published: 2007

Journal Articles

Lenalidomide Up-Regulates SPARC and Inhibits In Vitro Growth of the Malignant Clone in Myelodysplastic Syndrome Patients with 5q Deletion.

Andrea Pellagatti, Martin Jädersten, Ann-Mari Forsblom, Helen Cattan, Birger Christensson, Emma K. Emanuelsson, Mats Merup, Lars Nilsson, Jan Samuelsson, Birgitta Sander, James S. Wainscoat, Jacqueline Boultwood, Eva Hellström-Lindberg

Blood (2006) 108 (11): 855.

https://doi.org/10.1182/blood.V108.11.855.855

Published: 2006

Journal Articles

Lenalidomide Selectively Inhibits In Vitro Growth of the Malignant Clone in Myelodysplastic Syndrome (MDS) Patients with 5q Deletion.

Martin Jädersten, MD, Andrea Pellagatti, Ann-Mari Forsblom, Emma K. Emanuelsson, Mats Merup, MD, Jan Samuelsson, MD, James S. Wainscoat, Jacqueline Boultwood, Eva Hellström-Lindberg, MD

Blood (2005) 106 (11): 3438.

https://doi.org/10.1182/blood.V106.11.3438.3438

Published: 2005

Journal Articles

Aberrant mitochondrial iron distribution and maturation arrest characterize early erythroid precursors in low-risk myelodysplastic syndromes

Ramin Tehranchi, Rosangela Invernizzi, Alf Grandien, Boris Zhivotovsky, Bengt Fadeel, Ann-Mari Forsblom, Erica Travaglino, Jan Samuelsson, Robert Hast, Lars Nilsson, Mario Cazzola, Rolf Wibom, Eva Hellström-Lindberg

Blood (2005) 106 (1): 247–253.

https://doi.org/10.1182/blood-2004-12-4649

Published: 2005

Journal Articles

Erythropoiesis Is Highly Stimulated in CD34 + Cells in Low-Risk Myelodysplastic Syndromes (MDS) with an Improper Mitochondrial Function.

Ramin Tehranchi, MD, Rosangela Invernizzi, MD, PhD, Boris Zhivotovsky, PhD, Bengt Fadeel, MD, PhD, Ann-Mari Forsblom, Erica Travaglino, Jan Samuelsson, MD, Robert Hast, MD, PhD, Lars Nilsson, MD, PhD, Rolf Wibom, PhD, Alf Grandien, PhD, Mario Cazzola, MD, PhD, Eva Hellstrom-Lindberg, MD, PhD

Blood (2004) 104 (11): 473.

https://doi.org/10.1182/blood.V104.11.473.473

Published: 2004

Journal Articles

Granulocyte colony-stimulating factor inhibits spontaneous cytochrome c release and mitochondria-dependent apoptosis of myelodysplastic syndrome hematopoietic progenitors

Ramin Tehranchi, Bengt Fadeel, Ann-Mari Forsblom, Birger Christensson, Jan Samuelsson, Boris Zhivotovsky, Eva Hellstrom-Lindberg

Blood (2003) 101 (3): 1080–1086.

https://doi.org/10.1182/blood-2002-06-1774

Published: 2003

NARROW

Gene Expression Profiling of Day 7 Erythroblasts from RARS Before and after Treatment with G-CSF.

Lenalidomide Up-Regulates SPARC and Inhibits In Vitro Growth of the Malignant Clone in Myelodysplastic Syndrome Patients with 5q Deletion.

Lenalidomide Selectively Inhibits In Vitro Growth of the Malignant Clone in Myelodysplastic Syndrome (MDS) Patients with 5q Deletion.

Aberrant mitochondrial iron distribution and maturation arrest characterize early erythroid precursors in low-risk myelodysplastic syndromes

Erythropoiesis Is Highly Stimulated in CD34 + Cells in Low-Risk Myelodysplastic Syndromes (MDS) with an Improper Mitochondrial Function.

Granulocyte colony-stimulating factor inhibits spontaneous cytochrome c release and mitochondria-dependent apoptosis of myelodysplastic syndrome hematopoietic progenitors

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ASH Publications

American Society of Hematology

Gene Expression Profiling of Day 7 Erythroblasts from RARS Before and after Treatment with G-CSF.

Lenalidomide Up-Regulates SPARC and Inhibits In Vitro Growth of the Malignant Clone in Myelodysplastic Syndrome Patients with 5q Deletion.

Lenalidomide Selectively Inhibits In Vitro Growth of the Malignant Clone in Myelodysplastic Syndrome (MDS) Patients with 5q Deletion.

Aberrant mitochondrial iron distribution and maturation arrest characterize early erythroid precursors in low-risk myelodysplastic syndromes

Erythropoiesis Is Highly Stimulated in CD34 + Cells in Low-Risk Myelodysplastic Syndromes (MDS) with an Improper Mitochondrial Function.

Granulocyte colony-stimulating factor inhibits spontaneous cytochrome c release and mitochondria-dependent apoptosis of myelodysplastic syndrome hematopoietic progenitors

Email alerts

ASH Publications

American Society of Hematology

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