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Journal Articles

Coordinate Expression of Beta 2 Integrins and the Interleukin 23 Receptor Define a Pathogenic CD4+ T Cell Population That Induces Colonic Inflammation during Graft Versus Host Disease

Kimberle Agle, PhD, Vivian Zhou, PhD, Xiao Chen, MD PhD, Amy Beres, PhD, Richard Komorowski, MD, Carolyn Taylor, PhD, Dipica Haribhai, Calvin Williams, MD PhD, James Verbsky, MDPhD, Wendy Blumenschein, PhD, Svetlana Sadekova, PhD, Eddie Bowman, PhD, Casey Weaver, MD, Daniel Cua, PhD, William R. Drobyski, MD

Blood (2014) 124 (21): 537.

https://doi.org/10.1182/blood.V124.21.537.537

Published: 2014

Journal Articles

Emergence of Donor T Cells That Recognize Nonpolymorphic Antigens During Graft Versus Host Disease,

Hemalatha Rangarajan, MD, Maryam Yassai, Xiao Chen, MD, PhD, Richard Komorowski, MD, Jack Gorski, PhD, William R. Drobyski, MD

Blood (2011) 118 (21): 4013.

https://doi.org/10.1182/blood.V118.21.4013.4013

Published: 2011

Journal Articles

Stability of Foxp3 Expression Is a Critical Factor In the Ability of Regulatory T Cells to Mitigate Graft Versus Host Disease

Amy Beres, Richard Komorowski, MD, William R. Drobyski, MD

Blood (2010) 116 (21): 731.

https://doi.org/10.1182/blood.V116.21.731.731

Published: 2010

Journal Articles

Inhibition of Interleukin 17 Significantly Attenuates the Severity of Autoimmune-Mediated Pathological Damage in Chronic Graft Versus Host Disease.

Xiao Chen, MD, PhD, Jacques Van Snick, Catherine Uyttenhove, Richard Komorowski, MD, William R. Drobyski, MD

Blood (2007) 110 (11): 70.

https://doi.org/10.1182/blood.V110.11.70.70

Published: 2007

Journal Articles

Targeting NF-Kβ for the Prevention of Graft Versus Host Disease.

William R. Drobyski, MD, Sanja Vodanovic-Jankovic, MD, PhD, Parameswaran Hari, MD, Paulette Jacobs, MS, Richard Komorowski, MD

Blood (2005) 106 (11): 3109.

https://doi.org/10.1182/blood.V106.11.3109.3109

Published: 2005

Journal Articles

Phenyl N-Tert-Butylnitrone (PBN) Protects Syngeneic Marrow Transplant Recipients from the Lethal Cytokine Syndrome Occurring after Agonistic CD40 Antibody Administration.

William R. Drobyski, MD, Nadine Halligan, Richard Komorowski, MD, Brent Logan, William J. Murphy, PhD, Bruce Blazar, MD, Kirkwood Pritchard, PhD, Maria Gendelman

Blood (2004) 104 (11): 2128.

https://doi.org/10.1182/blood.V104.11.2128.2128

Published: 2004

Journal Articles

The Progression of Graft Versus Host Disease Is Characterized by the Breaking of Tolerance to Self Antigens and Is Dependent upon Donor Antigen Presenting Cells.

William R. Drobyski, MD, Richard Komorowski, MD, Elizabeth Tivol, PhD

Blood (2004) 104 (11): 598.

https://doi.org/10.1182/blood.V104.11.598.598

Published: 2004

NARROW

Coordinate Expression of Beta 2 Integrins and the Interleukin 23 Receptor Define a Pathogenic CD4+ T Cell Population That Induces Colonic Inflammation during Graft Versus Host Disease

Emergence of Donor T Cells That Recognize Nonpolymorphic Antigens During Graft Versus Host Disease,

Stability of Foxp3 Expression Is a Critical Factor In the Ability of Regulatory T Cells to Mitigate Graft Versus Host Disease

Inhibition of Interleukin 17 Significantly Attenuates the Severity of Autoimmune-Mediated Pathological Damage in Chronic Graft Versus Host Disease.

Targeting NF-Kβ for the Prevention of Graft Versus Host Disease.

Phenyl N-Tert-Butylnitrone (PBN) Protects Syngeneic Marrow Transplant Recipients from the Lethal Cytokine Syndrome Occurring after Agonistic CD40 Antibody Administration.

The Progression of Graft Versus Host Disease Is Characterized by the Breaking of Tolerance to Self Antigens and Is Dependent upon Donor Antigen Presenting Cells.

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ASH Publications

American Society of Hematology

Coordinate Expression of Beta 2 Integrins and the Interleukin 23 Receptor Define a Pathogenic CD4+ T Cell Population That Induces Colonic Inflammation during Graft Versus Host Disease

Emergence of Donor T Cells That Recognize Nonpolymorphic Antigens During Graft Versus Host Disease,

Stability of Foxp3 Expression Is a Critical Factor In the Ability of Regulatory T Cells to Mitigate Graft Versus Host Disease

Inhibition of Interleukin 17 Significantly Attenuates the Severity of Autoimmune-Mediated Pathological Damage in Chronic Graft Versus Host Disease.

Targeting NF-Kβ for the Prevention of Graft Versus Host Disease.

Phenyl N-Tert-Butylnitrone (PBN) Protects Syngeneic Marrow Transplant Recipients from the Lethal Cytokine Syndrome Occurring after Agonistic CD40 Antibody Administration.

The Progression of Graft Versus Host Disease Is Characterized by the Breaking of Tolerance to Self Antigens and Is Dependent upon Donor Antigen Presenting Cells.

Email alerts

ASH Publications

American Society of Hematology

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