Page 1 | Search Results | Blood | American Society of Hematology

1-7 of 7

Sort by

Sort Order Select

Journal Articles

Class I Phosphatidylinositol Transfer Proteins (PITPs) Promote Platelet Activation by Functioning As Essential Cofactors for Phospholipid Kinases and Hydrolases

Liang Zhao, Ph.D., Lurong Lian, M.D., Aae Suzuki, Charles S. Abrams, MD

Blood (2012) 120 (21): 91.

https://doi.org/10.1182/blood.V120.21.91.91

Published: 2012

Journal Articles

Platelets Lacking Both Pleckstrin and Pleckstrin-2 Have a Marked Deficient Cell Spreading and Actin Assembly

Yanfeng Wang, M.D., Lurong Lian, M.D., John H. Hartwig, PhD, Charles S. Abrams, MD

Blood (2008) 112 (11): 253.

https://doi.org/10.1182/blood.V112.11.253.253

Published: 2008

Journal Articles

Loss of Individual PIP5KI Isoforms Demonstrate That Spatial PIP2 Synthesis Is Required for Platelet Second Messenger Formation & Integrity of the Actin Cytoskeleton

Yanfeng Wang, M.D., Lurong Lian, M.D., Aae Suzuki, Rustem I. Litvinov, M.D., Timothy J. Stalker, Ph.D., Alec A. Schmaier, Lawrence F. Brass, MD, PhD, John Weisel, Ph.D., Charles S. Abrams, MD

Blood (2008) 112 (11): 109.

https://doi.org/10.1182/blood.V112.11.109.109

Published: 2008

Journal Articles

Impaired Actin Assembly and Lamellipodia Formation in ATE1 Knockout Platelets.

Lurong Lian, M.D., Anna S. Kashina, Ph.D., Karen P. Fong, Ph.D., Yanfeng Wang, M.D., Charles S. Abrams, MD

Blood (2008) 112 (11): 1834.

https://doi.org/10.1182/blood.V112.11.1834.1834

Published: 2008

Journal Articles

Mice Lacking Pleckstrin or Pleckstrin-2 Each Have Unique Platelet Secretion Defects.

Yanfeng Wang, M.D., Lurong Lian, M.D., Matthew Flick, Ph.D., Jay Degen, Ph.D., Mark Lemmon, Ph.D., Charles S. Abrams, MD

Blood (2007) 110 (11): 134.

https://doi.org/10.1182/blood.V110.11.134.134

Published: 2007

Journal Articles

PIP5Kγ-Null Mutation Induces Cytoskeletal Changes within Megakaryocytes.

Yanfeng Wang, M.D., Lurong Lian, M.D., Tami L. Bach, M.D., Xinsheng Chen, Qing-Min Chen, Charles S. Abrams, M.D.

Blood (2004) 104 (11): 629.

https://doi.org/10.1182/blood.V104.11.629.629

Published: 2004

Journal Articles

Null Mutation of Pleckstrin2, a PI3K Effector, Impairs Platelet Secretion.

Yanfeng Wang, M.D., Lurong Lian, M.D., Charles S. Abrams, M.D.

Blood (2004) 104 (11): 3522.

https://doi.org/10.1182/blood.V104.11.3522.3522

Published: 2004

NARROW

Class I Phosphatidylinositol Transfer Proteins (PITPs) Promote Platelet Activation by Functioning As Essential Cofactors for Phospholipid Kinases and Hydrolases

Platelets Lacking Both Pleckstrin and Pleckstrin-2 Have a Marked Deficient Cell Spreading and Actin Assembly

Loss of Individual PIP5KI Isoforms Demonstrate That Spatial PIP2 Synthesis Is Required for Platelet Second Messenger Formation & Integrity of the Actin Cytoskeleton

Impaired Actin Assembly and Lamellipodia Formation in ATE1 Knockout Platelets.

Mice Lacking Pleckstrin or Pleckstrin-2 Each Have Unique Platelet Secretion Defects.

PIP5Kγ-Null Mutation Induces Cytoskeletal Changes within Megakaryocytes.

Null Mutation of Pleckstrin2, a PI3K Effector, Impairs Platelet Secretion.

Email alerts

ASH Publications

American Society of Hematology

Class I Phosphatidylinositol Transfer Proteins (PITPs) Promote Platelet Activation by Functioning As Essential Cofactors for Phospholipid Kinases and Hydrolases

Platelets Lacking Both Pleckstrin and Pleckstrin-2 Have a Marked Deficient Cell Spreading and Actin Assembly

Loss of Individual PIP5KI Isoforms Demonstrate That Spatial PIP2 Synthesis Is Required for Platelet Second Messenger Formation & Integrity of the Actin Cytoskeleton

Impaired Actin Assembly and Lamellipodia Formation in ATE1 Knockout Platelets.

Mice Lacking Pleckstrin or Pleckstrin-2 Each Have Unique Platelet Secretion Defects.

PIP5Kγ-Null Mutation Induces Cytoskeletal Changes within Megakaryocytes.

Null Mutation of Pleckstrin2, a PI3K Effector, Impairs Platelet Secretion.

Email alerts

ASH Publications

American Society of Hematology

This Feature Is Available To Subscribers Only