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Journal Articles

FLT3 and KIT Isoforms Activate PI3K/AKT Signaling Which Is Potently Blocked by the Novel Dual PI3K/Mtor Inhibitors NVP-BEZ235 and NVP-BGT226.

Kerstin M Kampa, MD, Sandra Mueller, PhD, Katharina-Henriette Rasp, Lothar Kanz, MD, Michael C Heinrich, MD, Marcus M Schittenhelm, MD

Blood (2009) 114 (22): 1740.

https://doi.org/10.1182/blood.V114.22.1740.1740

Published: 2009

Journal Articles

Insights Into the Role of ASPP2 (Apoptosis Stimulating Protein of p53-2) in Lymphomagenesis.

Kerstin M Kampa, MD, Sandra Mueller, PhD, Michael Bonin, PhD, Marcus M Schittenhelm, MD, Charles D Lopez, MD, PhD

Blood (2009) 114 (22): 1967.

https://doi.org/10.1182/blood.V114.22.1967.1967

Published: 2009

Journal Articles

KIT Inhibition Affects Heat Shock Protein (HSP) Activity: A Potential Rescue Mechanism towards KIT Tyrosine Kinase Inhibition and a Rationale for Dual KIT/HSP Inhibition.

Kerstin M Kampa, MD, Tanja Grandl, Sandra Mueller, Emmanuel Normant, PhD, Michael Walter, PhD, Wolfgang Schuetz, PhD, Lothar Kanz, MD, Michael C Heinrich, MD, Marcus M Schittenhelm, MD

Blood (2009) 114 (22): 2753.

https://doi.org/10.1182/blood.V114.22.2753.2753

Published: 2009

Journal Articles

Tyrosine Kinase Inhibitors Only Partially Inhibit AKT-Dependent Signaling in Cells Expressing Oncogenic KIT, FLT3 or ABL Kinases. A Potential Model for Leukemia Cell Resistance to TKIs.

Marcus M. Schittenhelm, MD, Kerstin M. Kampa, MD, Sharon Shiraga, Lothar Kanz, MD, PhD, Michael C. Heinrich, MD, PhD

Blood (2007) 110 (11): 2370.

https://doi.org/10.1182/blood.V110.11.2370.2370

Published: 2007

Journal Articles

Apoptosis Stimulating Protein of p53 (ASPP2) Haploinsufficiency Promotes Irradiation-Induced High Grade Lymphomas in a Mouse Model.

Kerstin M. Kampa, MD, Jared D. Akoba, MD, Dexi Chen, MD, PhD, Kelly Beemer, MS, Joel Gay, Zhiyi Zhu, MS, Emerson Padiernos, BS, Christopher Corless, MD, PhD, Alexis Baily, William H. Flemming, MD, PhD, Charles D. Lopez, MD, PhD.

Blood (2007) 110 (11): 2639.

https://doi.org/10.1182/blood.V110.11.2639.2639

Published: 2007

Journal Articles

Iv Vitro Modeling of the Use of FLT3 Inhibitors To Reduce Chemotherapy Doses for Elderly Patients with AML.

Marcus M. Schittenhelm, MD, Kerstin M. Kampa, MD, Kevin W.H. Yee, MD, Lothar Kanz, MD,PhD, Michael C. Heinrich, MD,PhD

Blood (2007) 110 (11): 4213.

https://doi.org/10.1182/blood.V110.11.4213.4213

Published: 2007

Journal Articles

ASPP2 Haploinsufficiency Promotes Tumor Formation in a Mouse Model.

Kerstin M. Kampa, MD, Jared D. Acoba, M.D., Dexi Chen, M.D.,Ph.D., Kelly Beemer, M.S., Joel Gay, Zhiyi Zhu, M.S., Emerson Padiernos, B.S., Alice Fan, M.D., Dean Felsher, M.D.,Ph.D., Christopher Corless, M.D.,Ph.D., Louie Naumovski, M.D.,Ph.D., Charles D. Lopez, M.D.,Ph.D.

Blood (2006) 108 (11): 4333.

https://doi.org/10.1182/blood.V108.11.4333.4333

Published: 2006

NARROW

FLT3 and KIT Isoforms Activate PI3K/AKT Signaling Which Is Potently Blocked by the Novel Dual PI3K/Mtor Inhibitors NVP-BEZ235 and NVP-BGT226.

Insights Into the Role of ASPP2 (Apoptosis Stimulating Protein of p53-2) in Lymphomagenesis.

KIT Inhibition Affects Heat Shock Protein (HSP) Activity: A Potential Rescue Mechanism towards KIT Tyrosine Kinase Inhibition and a Rationale for Dual KIT/HSP Inhibition.

Tyrosine Kinase Inhibitors Only Partially Inhibit AKT-Dependent Signaling in Cells Expressing Oncogenic KIT, FLT3 or ABL Kinases. A Potential Model for Leukemia Cell Resistance to TKIs.

Apoptosis Stimulating Protein of p53 (ASPP2) Haploinsufficiency Promotes Irradiation-Induced High Grade Lymphomas in a Mouse Model.

Iv Vitro Modeling of the Use of FLT3 Inhibitors To Reduce Chemotherapy Doses for Elderly Patients with AML.

ASPP2 Haploinsufficiency Promotes Tumor Formation in a Mouse Model.

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ASH Publications

American Society of Hematology

FLT3 and KIT Isoforms Activate PI3K/AKT Signaling Which Is Potently Blocked by the Novel Dual PI3K/Mtor Inhibitors NVP-BEZ235 and NVP-BGT226.

Insights Into the Role of ASPP2 (Apoptosis Stimulating Protein of p53-2) in Lymphomagenesis.

KIT Inhibition Affects Heat Shock Protein (HSP) Activity: A Potential Rescue Mechanism towards KIT Tyrosine Kinase Inhibition and a Rationale for Dual KIT/HSP Inhibition.

Tyrosine Kinase Inhibitors Only Partially Inhibit AKT-Dependent Signaling in Cells Expressing Oncogenic KIT, FLT3 or ABL Kinases. A Potential Model for Leukemia Cell Resistance to TKIs.

Apoptosis Stimulating Protein of p53 (ASPP2) Haploinsufficiency Promotes Irradiation-Induced High Grade Lymphomas in a Mouse Model.

Iv Vitro Modeling of the Use of FLT3 Inhibitors To Reduce Chemotherapy Doses for Elderly Patients with AML.

ASPP2 Haploinsufficiency Promotes Tumor Formation in a Mouse Model.

Email alerts

ASH Publications

American Society of Hematology

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