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Journal Articles

Tissue-Resident TSP1-CD47 Signaling Promotes Sickle Cell-Associated Arterial Vasculopathy and Pulmonary Hypertension

Enrico M Novelli, MD MS, Lynda L Ihrig, Heather E Knupp, MSc, Natasha M Rogers, Mingyi Yao, PhD, Xiaojun Huang, Jeffrey Baust, Daniel Meijles, PhD, Claudette M St Croix, PhD, Mark A Ross, Patrick J Pagano, PhD, Hunter C Champion, MD PhD, George Miles, MD PhD, Karin P Potoka, MD, Mark T Gladwin, MD, Jeffrey S Isenberg

Blood (2017) 130 (Supplement 1): 957.

https://doi.org/10.1182/blood.V130.Suppl_1.957.957

Published: 2017

Journal Articles

Chronic Phosphodiesterase Type 5 Inhibitor Therapy Reduces Priapic Activity in Transgenic Sickle Cell Mice: Restoring Nitric Oxide/Redox Balance in the Penis.

Arthur L. Burnett, M.D., M.B.A., Biljana Musicki, Ph.D., Hunter C. Champion, MD, PhD, Trinity J. Bivalacqua, M.D., Ph.D.

Blood (2009) 114 (22): 576.

https://doi.org/10.1182/blood.V114.22.576.576

Published: 2009

Journal Articles

Sickle Cell Pulmonary Hypertension and Dysregulated NO Axis in a Mouse Model Are Modulated by Apolipoprotein a-1 Availability

Lewi s L. Hsu, MD, PhD, Hazim El-Haddad, MD, Marcelo Amar, MD, Gregory J. Kato, MD, Alan T Remaley, MD, PhD, Hunter C. Champion, MD, PhD

Blood (2008) 112 (11): 2499.

https://doi.org/10.1182/blood.V112.11.2499.2499

Published: 2008

Journal Articles

Hemolysis in Sickle Cell Mice Causes Pulmonary Hypertension Due to Global Impairment in Nitric Oxide Bioavailability.

Lewis L. Hsu, MD, PhD, Hunter C. Champion, MD, PhD, Sally A. Campbell-Lee, MD, Trinity J. Bivalacqua, MD, PhD, Elizabeth A. Manci, MD, Bhalchandra A. Diwan, PhD, Daniel M. Schimel, Audrey E. Cochard, Xunde Wang, Alan N. Schechter, MD, Constance Tom Noguchi, PhD, Mark T. Gladwin, MD

Blood (2006) 108 (11): 785.

https://doi.org/10.1182/blood.V108.11.785.785

Published: 2006

Journal Articles

Spontaneous Pulmonary Hypertension in Transgenic Sickle Cell Mice - Hemodynamics and Histology Suggest Abnormal Nitric Oxide Production and Scavenging.

Lewis L. Hsu, MD, PhD, Hunter C. Champion, MD, PhD, Elizabeth Manci, MD, Bhalchandra Diwan, PhD, Daniel Schimel, RLAT, Audrey Cochard, BA, Xunde Wang, MD, J. D. Holtzclaw, PhD, Alan Schechter, MD, Constance T. Noguchi, PhD, Mark Gladwin, MD

Blood (2005) 106 (11): 208.

https://doi.org/10.1182/blood.V106.11.208.208

Published: 2005

NARROW

Tissue-Resident TSP1-CD47 Signaling Promotes Sickle Cell-Associated Arterial Vasculopathy and Pulmonary Hypertension

Chronic Phosphodiesterase Type 5 Inhibitor Therapy Reduces Priapic Activity in Transgenic Sickle Cell Mice: Restoring Nitric Oxide/Redox Balance in the Penis.

Sickle Cell Pulmonary Hypertension and Dysregulated NO Axis in a Mouse Model Are Modulated by Apolipoprotein a-1 Availability

Hemolysis in Sickle Cell Mice Causes Pulmonary Hypertension Due to Global Impairment in Nitric Oxide Bioavailability.

Spontaneous Pulmonary Hypertension in Transgenic Sickle Cell Mice - Hemodynamics and Histology Suggest Abnormal Nitric Oxide Production and Scavenging.

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ASH Publications

American Society of Hematology

Tissue-Resident TSP1-CD47 Signaling Promotes Sickle Cell-Associated Arterial Vasculopathy and Pulmonary Hypertension

Chronic Phosphodiesterase Type 5 Inhibitor Therapy Reduces Priapic Activity in Transgenic Sickle Cell Mice: Restoring Nitric Oxide/Redox Balance in the Penis.

Sickle Cell Pulmonary Hypertension and Dysregulated NO Axis in a Mouse Model Are Modulated by Apolipoprotein a-1 Availability

Hemolysis in Sickle Cell Mice Causes Pulmonary Hypertension Due to Global Impairment in Nitric Oxide Bioavailability.

Spontaneous Pulmonary Hypertension in Transgenic Sickle Cell Mice - Hemodynamics and Histology Suggest Abnormal Nitric Oxide Production and Scavenging.

Email alerts

ASH Publications

American Society of Hematology

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