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Journal Articles

Inducible Expression of Mutant ELANE Correlates with Disruption of Differentiation Program, but Not the Unfolded Protein Response

Bhavuk Garg, PhD, Hrishikesh M Mehta, PhD, Ralph Kamel, Marshall S. Horwitz, MD PhD, Seth J. Corey, MD

Blood (2019) 134 (Supplement_1): 103.

https://doi.org/10.1182/blood-2019-127409

Published: 2019

Journal Articles

Effect of the Unfolded Protein Response and Oxidative Stress on Mutagenesis of CSF3R a Model of Evolution of Severe Congenital Neutropenia to Myelodysplastic Syndrome/Acute Myeloid Leukemia

Adya Sapra, MS, Roman Jaksik, PhD, Sara Biesiadny, Hrishikesh M Mehta, PhD, Marek Kimmel, PhD DSc, Seth J. Corey, MD

Blood (2019) 134 (Supplement_1): 3752.

https://doi.org/10.1182/blood-2019-127401

Published: 2019

Journal Articles

Molecular Spectrum of CSF3R variants Correlate with Specific Myeloid Malignancies and Secondary Mutations

Vera Adema, PhD, Cassandra M. Hirsch, Bartlomiej Przychodzen, Yasunobu Nagata, Tomas Radivoyevitch, Thomas Laframboise, PhD, Amy C Graham, BS, MS, Louis Williams, MD MBA, Hassan Awada, MD, Aziz Nazha, Hetty E. Carraway, MD MBA, Valeria Santini, Hrishikesh M Mehta, PhD, Francesc Sole, PhD, Mikkael A. Sekeres, MD MS, Valeria Visconte, PhD, Rabi Hanna, MD, Seth J. Corey, MD, Jaroslaw P. Maciejewski

Blood (2018) 132 (Supplement 1): 4389.

https://doi.org/10.1182/blood-2018-99-116554

Published: 2018

Journal Articles

Identification of Bone Marrow-Derived and Circulating Fibrocytes in Gata1low Mice with Myelofibrosis

Ana Marquez, BS, Jeffy George, PhD, Hrishikesh M Mehta, PhD, Regina M. Day, PhD, Seth Corey, MD MPH

Blood (2016) 128 (22): 3144.

https://doi.org/10.1182/blood.V128.22.3144.3144

Published: 2016

Journal Articles

Mutations in the Splicosome Lead to Alternative Splicing of CSF3R in Promoting Myelodysplastic Syndromes

Frances Austin, MD, Hrishikesh M Mehta, PhD, Seth J. Corey, MD MPH, Chonghui Cheng, MD PhD

Blood (2016) 128 (22): 3897.

https://doi.org/10.1182/blood.V128.22.3897.3897

Published: 2016

Journal Articles

Conditional Expression of Mutant ELANE Produces Unfolded Protein Response but Fails to Promote Cell Death or Differentiation Block: What Is the Mechanism for Severe Congenital Neutropenia?

Hrishikesh M Mehta, PhD, Arighno Das, BA, Ralph Kamel, MD, Marshall Horwitz, MD PhD, Seth Corey, MD MPH

Blood (2016) 128 (22): 3899.

https://doi.org/10.1182/blood.V128.22.3899.3899

Published: 2016

Journal Articles

Mutations in the Spliceosome Lead to Alternative Splicing of CSF3R in Promoting Myelodysplastic Syndromes

Frances Austin, MD, Hrishikesh M Mehta, PhD, Chonghui Cheng, MD PhD, Seth Corey, MD MPH

Blood (2016) 128 (22): 1978.

https://doi.org/10.1182/blood.V128.22.1978.1978

Published: 2016

NARROW

Inducible Expression of Mutant ELANE Correlates with Disruption of Differentiation Program, but Not the Unfolded Protein Response

Effect of the Unfolded Protein Response and Oxidative Stress on Mutagenesis of CSF3R a Model of Evolution of Severe Congenital Neutropenia to Myelodysplastic Syndrome/Acute Myeloid Leukemia

Molecular Spectrum of CSF3R variants Correlate with Specific Myeloid Malignancies and Secondary Mutations

Identification of Bone Marrow-Derived and Circulating Fibrocytes in Gata1low Mice with Myelofibrosis

Mutations in the Splicosome Lead to Alternative Splicing of CSF3R in Promoting Myelodysplastic Syndromes

Conditional Expression of Mutant ELANE Produces Unfolded Protein Response but Fails to Promote Cell Death or Differentiation Block: What Is the Mechanism for Severe Congenital Neutropenia?

Mutations in the Spliceosome Lead to Alternative Splicing of CSF3R in Promoting Myelodysplastic Syndromes

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ASH Publications

American Society of Hematology

Inducible Expression of Mutant ELANE Correlates with Disruption of Differentiation Program, but Not the Unfolded Protein Response

Effect of the Unfolded Protein Response and Oxidative Stress on Mutagenesis of CSF3R a Model of Evolution of Severe Congenital Neutropenia to Myelodysplastic Syndrome/Acute Myeloid Leukemia

Molecular Spectrum of CSF3R variants Correlate with Specific Myeloid Malignancies and Secondary Mutations

Identification of Bone Marrow-Derived and Circulating Fibrocytes in Gata1low Mice with Myelofibrosis

Mutations in the Splicosome Lead to Alternative Splicing of CSF3R in Promoting Myelodysplastic Syndromes

Conditional Expression of Mutant ELANE Produces Unfolded Protein Response but Fails to Promote Cell Death or Differentiation Block: What Is the Mechanism for Severe Congenital Neutropenia?

Mutations in the Spliceosome Lead to Alternative Splicing of CSF3R in Promoting Myelodysplastic Syndromes

Email alerts

ASH Publications

American Society of Hematology

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