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Journal Articles

CXCR4 Signals Regulate Neutrophil Release from the Bone Marrow but Not Clearance from the Circulation.

Kyle J. Eash, BS, Jacquelyn M. Means, BS, Jill R. Woloszynek, BS, Fulu Liu, MD, Daniel C. Link, MD

Blood (2007) 110 (11): 3297.

https://doi.org/10.1182/blood.V110.11.3297.3297

Published: 2007

Journal Articles

G-CSF Receptor Mutations Found in Patients with Severe Congenital Neutropenia Confer a Strong Competitive Growth Advantage at the Hematopoietic Stem Cell Level That Is Mediated by STAT5 Activation.

Ghada M. Kunter, MD, Fulu Liu, MD, Maxwell Krem, MDPHD, Daniel Link, MD

Blood (2006) 108 (11): 632.

https://doi.org/10.1182/blood.V108.11.632.632

Published: 2006

Journal Articles

G-CSF Potently Suppresses Osteoblast Activity in the Bone Marrow.

Matthew J. Christopher, Fulu Liu, MD, Brenton Short, PhD, Paul J. Simmons, PhD, Ingrid Winkler, PhD, Jean-Pierre Levesque, PhD, Daniel C. Link, MD

Blood (2005) 106 (11): 591.

https://doi.org/10.1182/blood.V106.11.591.591

Published: 2005

Journal Articles

Evaluation of the Phenotype and GVHD-Inducing Potential of Splenic T Cells Isolated from G-CSF, AMD3100, or G-CSF and AMD3100 Pretreated Allogeneic Donors.

Michael Rettig, PhD, Matthew Holt, Fulu Liu, MD, Julie Ritchey, Daniel Link, MD, Gary Bridger, PhD, John DiPersio, MD/PhD

Blood (2005) 106 (11): 5224.

https://doi.org/10.1182/blood.V106.11.5224.5224

Published: 2005

Journal Articles

Disruption of SDF-1/CXCR4 Signaling during Flt-3 Ligand and Stem Cell Factor (SCF) Induced Hematopoietic Progenitor Mobilization.

Matthew J. Christopher, Fulu Liu, MD, Nathan Everly, Daniel C. Link, MD

Blood (2004) 104 (11): 4139.

https://doi.org/10.1182/blood.V104.11.4139.4139

Published: 2004

NARROW

CXCR4 Signals Regulate Neutrophil Release from the Bone Marrow but Not Clearance from the Circulation.

G-CSF Receptor Mutations Found in Patients with Severe Congenital Neutropenia Confer a Strong Competitive Growth Advantage at the Hematopoietic Stem Cell Level That Is Mediated by STAT5 Activation.

G-CSF Potently Suppresses Osteoblast Activity in the Bone Marrow.

Evaluation of the Phenotype and GVHD-Inducing Potential of Splenic T Cells Isolated from G-CSF, AMD3100, or G-CSF and AMD3100 Pretreated Allogeneic Donors.

Disruption of SDF-1/CXCR4 Signaling during Flt-3 Ligand and Stem Cell Factor (SCF) Induced Hematopoietic Progenitor Mobilization.

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ASH Publications

American Society of Hematology

CXCR4 Signals Regulate Neutrophil Release from the Bone Marrow but Not Clearance from the Circulation.

G-CSF Receptor Mutations Found in Patients with Severe Congenital Neutropenia Confer a Strong Competitive Growth Advantage at the Hematopoietic Stem Cell Level That Is Mediated by STAT5 Activation.

G-CSF Potently Suppresses Osteoblast Activity in the Bone Marrow.

Evaluation of the Phenotype and GVHD-Inducing Potential of Splenic T Cells Isolated from G-CSF, AMD3100, or G-CSF and AMD3100 Pretreated Allogeneic Donors.

Disruption of SDF-1/CXCR4 Signaling during Flt-3 Ligand and Stem Cell Factor (SCF) Induced Hematopoietic Progenitor Mobilization.

Email alerts

ASH Publications

American Society of Hematology

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