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Journal Articles

The Major Phagocyte Peroxidase-Derived Oxidant, HOSCN (Hypothiocyanous Acid), Induces Proinflammatory and Cytoprotective Gene Expression in Endothelium: A Mechanism for Microlocalized Regulation of Inflammation.

Shawn A. Mahmud, BA, Jian-Guo Wang, Ph.D., Peng Wei, M.S., Molly S. Thomas, Ph.D., John D. Belcher, Ph.D., Gregory M. Vercellotti, M.D., Arne Slungaard, M.D.

Blood (2007) 110 (11): 3295.

https://doi.org/10.1182/blood.V110.11.3295.3295

Published: 2007

Journal Articles

“TF-kB”-Dependent Suppression of Agonist-Mediated Tissue Factor Induction by Histone Deacetylase Inhibitors in Human Endothelial Cells and Monocytes.

Jianguo Wang, PhD, Shawn A. Mahmud, B.A., Peter B. Bitterman, M.D., Yuqing Huo, M.D., Ph.D., Arne Slungaard, M.D.

Blood (2006) 108 (11): 1745.

https://doi.org/10.1182/blood.V108.11.1745.1745

Published: 2006

Journal Articles

Regulation of Tissue Factor by NF-kB Transcription Factor p50 Is Essential for the Pathogeneses of Deep Vein Thrombosis and Arterial Restenosis.

Jian-Guo Geng, MD, PhD, Jian-Guo Wang, Ph.D., Nigel Mackman, Ph.D., Arne Slungaard, M.D., Yuqing Huo, M.D., Ph.D., Nigel S. Key, M.D.

Blood (2006) 108 (11): 1458.

https://doi.org/10.1182/blood.V108.11.1458.1458

Published: 2006

Journal Articles

Thiocyanate-Dependent Inhibition of Spontaneous and Agonist-Induced Eosinophil Apoptosis by Eosinophil Peroxidase (EPO): A Potential Physiologic Role for Endogenously Generated HOSCN in Maintaining Eosinophil Viability.

Shawn A. Mahmud, BS, Jianguo Wang, Ph.D., Arne Slungaard, M.D.

Blood (2006) 108 (11): 1633.

https://doi.org/10.1182/blood.V108.11.1633.1633

Published: 2006

Journal Articles

The Principal Eosinophil Peroxidase Oxidant, HOSCN, Is a Uniquely Potent Phagocyte Oxidant Inducer of E-Selectin, ICAM-1 and VCAM-1 Expression on Endothelial Cells and Promotes Leukocyte-Endothelial Cell Adhesion. A Potential Mechanism Regulating Leukocyte Trafficking in Eosinophilic Inflammatory States.

Jianguo Wang, PhD, Rong Tang, PhD, Yuqing Huo, MD, PhD, Arne Slungaard, M.D

Blood (2005) 106 (11): 3085.

https://doi.org/10.1182/blood.V106.11.3085.3085

Published: 2005

Journal Articles

The Principal Eosinophil Peroxidase Oxidant, HOSCN, Potently Induces Endothelial Cell Tissue Factor Activity through Transcriptional Activation Dependent upon NF-kB and ERK1/2: Potential Mechanism for a Prothrombotic and Proinflammatory Endothelial Phenotype in the Hypereosinophilic Syndrome.

Jianguo Wang, Ph.D., Shawn A. Mahmud, B.S., Jake A. Thompson, B.S., Jianguo Geng, M.D./Ph.D., Nigel S. Key, M.D., Arne Slungaard, M.D.

Blood (2004) 104 (11): 804.

https://doi.org/10.1182/blood.V104.11.804.804

Published: 2004

NARROW

The Major Phagocyte Peroxidase-Derived Oxidant, HOSCN (Hypothiocyanous Acid), Induces Proinflammatory and Cytoprotective Gene Expression in Endothelium: A Mechanism for Microlocalized Regulation of Inflammation.

“TF-kB”-Dependent Suppression of Agonist-Mediated Tissue Factor Induction by Histone Deacetylase Inhibitors in Human Endothelial Cells and Monocytes.

Regulation of Tissue Factor by NF-kB Transcription Factor p50 Is Essential for the Pathogeneses of Deep Vein Thrombosis and Arterial Restenosis.

Thiocyanate-Dependent Inhibition of Spontaneous and Agonist-Induced Eosinophil Apoptosis by Eosinophil Peroxidase (EPO): A Potential Physiologic Role for Endogenously Generated HOSCN in Maintaining Eosinophil Viability.

The Principal Eosinophil Peroxidase Oxidant, HOSCN, Potently Induces Endothelial Cell Tissue Factor Activity through Transcriptional Activation Dependent upon NF-kB and ERK1/2: Potential Mechanism for a Prothrombotic and Proinflammatory Endothelial Phenotype in the Hypereosinophilic Syndrome.

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ASH Publications

American Society of Hematology

The Major Phagocyte Peroxidase-Derived Oxidant, HOSCN (Hypothiocyanous Acid), Induces Proinflammatory and Cytoprotective Gene Expression in Endothelium: A Mechanism for Microlocalized Regulation of Inflammation.

“TF-kB”-Dependent Suppression of Agonist-Mediated Tissue Factor Induction by Histone Deacetylase Inhibitors in Human Endothelial Cells and Monocytes.

Regulation of Tissue Factor by NF-kB Transcription Factor p50 Is Essential for the Pathogeneses of Deep Vein Thrombosis and Arterial Restenosis.

Thiocyanate-Dependent Inhibition of Spontaneous and Agonist-Induced Eosinophil Apoptosis by Eosinophil Peroxidase (EPO): A Potential Physiologic Role for Endogenously Generated HOSCN in Maintaining Eosinophil Viability.

The Principal Eosinophil Peroxidase Oxidant, HOSCN, Potently Induces Endothelial Cell Tissue Factor Activity through Transcriptional Activation Dependent upon NF-kB and ERK1/2: Potential Mechanism for a Prothrombotic and Proinflammatory Endothelial Phenotype in the Hypereosinophilic Syndrome.

Email alerts

ASH Publications

American Society of Hematology

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