Page 6422: in the second paragraph on the right column, the figure reference after the sentence “Furthermore, the effects on the soluble coagulation cascade are addressed by experiments using heparin” should read “Figure 2G,” not “Figure 1G.”

Page 6423: the lowest curve in Figure 6J represents the wild-type (WT) group; this designation is missing from the figure key.

Figure 6.
C5aR1-induced inhibition of neovascularization by platelets depends on CXCL4 secretion and is distinct from platelet activation. (J-K) HLI was induced in C5aR1−/− mice or C5aR1−/−CXCL4−/− mice and a WT control group, which were generated as described in the “Methods.” Double knockout mice showed no increased revascularization in comparison to C5aR1−/− mice. Data are presented as the mean ± SEM (n = 5-7) and are displayed as a percentage of the perfusion in the contralateral control limb. n.s. = no significant difference could be observed compared with C5aR1−/− animals. (K) Shows representative LDI images of mouse hind limbs after femoral artery ligation and during revascularization over 14 days.
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C5aR1-induced inhibition of neovascularization by platelets depends on CXCL4 secretion and is distinct from platelet activation. (J-K) HLI was induced in C5aR1−/− mice or C5aR1−/−CXCL4−/− mice and a WT control group, which were generated as described in the “Methods.” Double knockout mice showed no increased revascularization in comparison to C5aR1−/− mice. Data are presented as the mean ± SEM (n = 5-7) and are displayed as a percentage of the perfusion in the contralateral control limb. n.s. = no significant difference could be observed compared with C5aR1−/− animals. (K) Shows representative LDI images of mouse hind limbs after femoral artery ligation and during revascularization over 14 days.

Figure 6.
C5aR1-induced inhibition of neovascularization by platelets depends on CXCL4 secretion and is distinct from platelet activation. (J-K) HLI was induced in C5aR1−/− mice or C5aR1−/−CXCL4−/− mice and a WT control group, which were generated as described in the “Methods.” Double knockout mice showed no increased revascularization in comparison to C5aR1−/− mice. Data are presented as the mean ± SEM (n = 5-7) and are displayed as a percentage of the perfusion in the contralateral control limb. n.s. = no significant difference could be observed compared with C5aR1−/− animals. (K) Shows representative LDI images of mouse hind limbs after femoral artery ligation and during revascularization over 14 days.
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C5aR1-induced inhibition of neovascularization by platelets depends on CXCL4 secretion and is distinct from platelet activation. (J-K) HLI was induced in C5aR1−/− mice or C5aR1−/−CXCL4−/− mice and a WT control group, which were generated as described in the “Methods.” Double knockout mice showed no increased revascularization in comparison to C5aR1−/− mice. Data are presented as the mean ± SEM (n = 5-7) and are displayed as a percentage of the perfusion in the contralateral control limb. n.s. = no significant difference could be observed compared with C5aR1−/− animals. (K) Shows representative LDI images of mouse hind limbs after femoral artery ligation and during revascularization over 14 days.

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In Figure 6K, due to an error in formatting, the right half of Figure 6K is a duplicate of the left side.

In the Figure 6 legend on page 6424, in the second to last line, “(M)” should read “(K).”

The corrected Figure 6J, 6K, and legend are shown below.

These errors do not affect the overall conclusions of the article.

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