Figure 3.
Figure 3. Myeloid leukocytosis. (A) Neutrophilia in a patient with a perforated tumor and infection. (B) G-CSF effect with immature granulocytes. (C) Neutrophils with toxic granulation and vacuoles are seen in a patient with a bacterial infection. (D) CML, BCR-ABL1+, chronic phase, shows many immature granulocytes and occasional blasts. (E) CMML with abnormal monocytes and a large hypogranular platelet (adapted with permission from Pereira et al,7 page 101). (F) Myeloid neoplasm with eosinophilia and PDGFRA shows 2 eosinophils with eosinophilic granules that do not completely fill the cytoplasm and occasional hypogranular platelets.

Myeloid leukocytosis. (A) Neutrophilia in a patient with a perforated tumor and infection. (B) G-CSF effect with immature granulocytes. (C) Neutrophils with toxic granulation and vacuoles are seen in a patient with a bacterial infection. (D) CML, BCR-ABL1+, chronic phase, shows many immature granulocytes and occasional blasts. (E) CMML with abnormal monocytes and a large hypogranular platelet (adapted with permission from Pereira et al, page 101). (F) Myeloid neoplasm with eosinophilia and PDGFRA shows 2 eosinophils with eosinophilic granules that do not completely fill the cytoplasm and occasional hypogranular platelets.

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