Figure 2
Figure 2. Sodium dependence of L-glutamate uptake. Time courses for the uptake of L-[3H]-glutamate into arsenite-treated (A) and P falciparum–infected (B) human erythrocytes (RBCs) in the presence or absence of extracellular Na+. The cells were suspended in a solution containing 20mM sucrose and 10mM Tris-HCl (pH 7.4) together with either 150mM sodium chloride (●) or 150mM choline chloride (○). The uptake data are expressed in terms of a distribution ratio (ie, the estimated concentration of radiolabel inside the cell relative to that in the extracellular solution). Symbols show data averaged from 5 independent experiments, ± SEM.

Sodium dependence of L-glutamate uptake. Time courses for the uptake of L-[3H]-glutamate into arsenite-treated (A) and P falciparum–infected (B) human erythrocytes (RBCs) in the presence or absence of extracellular Na+. The cells were suspended in a solution containing 20mM sucrose and 10mM Tris-HCl (pH 7.4) together with either 150mM sodium chloride (●) or 150mM choline chloride (○). The uptake data are expressed in terms of a distribution ratio (ie, the estimated concentration of radiolabel inside the cell relative to that in the extracellular solution). Symbols show data averaged from 5 independent experiments, ± SEM.

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