Fig. 3.
Fig. 3. Immunohistochemical analysis of Mig and IP-10 in lymphoproliferative disease tissues representative of LYG, nasal T/NK-cell lymphoma, and lymphoid hyperplasia. (A, B, and C) LYG, lung; (D, E, and F ) nasal T/NK-cell lymphoma, nasal septum (D, arrows indicate vessel with fibrinoid necrosis); (G, H, and I) reactive lymphoid hyperplasia, lymph node. Paraffin-embedded tissue sections were stained with Hematoxylin and Eosin (A, D, and G), with an anti-Mig heteroantiserum (B, E, and H), and with an anti–IP-10 heteroantiserum (C, F, and I). Primary antibodies were detected with biotinylated antimouse/rabbit IgG followed by streptavidin-peroxidase complexes. (Original magnification × 40.)

Immunohistochemical analysis of Mig and IP-10 in lymphoproliferative disease tissues representative of LYG, nasal T/NK-cell lymphoma, and lymphoid hyperplasia. (A, B, and C) LYG, lung; (D, E, and F ) nasal T/NK-cell lymphoma, nasal septum (D, arrows indicate vessel with fibrinoid necrosis); (G, H, and I) reactive lymphoid hyperplasia, lymph node. Paraffin-embedded tissue sections were stained with Hematoxylin and Eosin (A, D, and G), with an anti-Mig heteroantiserum (B, E, and H), and with an anti–IP-10 heteroantiserum (C, F, and I). Primary antibodies were detected with biotinylated antimouse/rabbit IgG followed by streptavidin-peroxidase complexes. (Original magnification × 40.)

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