Figure 7
Figure 7. Analysis of adhesion scores and peritoneal cytokine levels of EPCRδ/δ and PAR1−/− knockout mice. Adhesion scores for 10 EPCRδ/δ mice (supplemental Table 3) based on both Nair et al’s31 and Leach et al’s6 scales (A). The concentrations of IL-1β (B), IL-6 (C), TNF-α (D), TGF-β1 (E), IFN-γ (F), and tPA (G) for all EPCRδ/δ mice groups were measured by enzyme-linked immunosorbent assays. Adhesion scores for 10 PAR1−/− knockout mice (supplemental Table 4) based on both Nair et al’s31 and Leach et al’s6 scales (H). The concentrations of IL-1β (I), IL-6 (J), TNF-α (K), TGF-β1 (L), IFN-γ (M), and tPA (N) for all PAR1−/− knockout mice groups were measured by enzyme-linked immunosorbent assays. *P < .05 compared with saline-treated vehicle.

Analysis of adhesion scores and peritoneal cytokine levels of EPCRδ/δ and PAR1−/− knockout mice. Adhesion scores for 10 EPCRδ/δ mice (supplemental Table 3) based on both Nair et al’s31  and Leach et al’s scales (A). The concentrations of IL-1β (B), IL-6 (C), TNF-α (D), TGF-β1 (E), IFN-γ (F), and tPA (G) for all EPCRδ/δ mice groups were measured by enzyme-linked immunosorbent assays. Adhesion scores for 10 PAR1−/− knockout mice (supplemental Table 4) based on both Nair et al’s31  and Leach et al’s scales (H). The concentrations of IL-1β (I), IL-6 (J), TNF-α (K), TGF-β1 (L), IFN-γ (M), and tPA (N) for all PAR1−/− knockout mice groups were measured by enzyme-linked immunosorbent assays. *P < .05 compared with saline-treated vehicle.

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