Figure 6
Figure 6. Inhibition of Notch signal can partially rescue the decrease of HSCs in ncor2 morphants and vegfd-overexpressed embryos. (A) The expression pattern of runx1 in the control, ncor2 morphants, fos-overexpressed embryos, and vegfd-overexpressed embryos at 36 hpf, which were treated with DMSO or with a γ-secretase inhibitor DAPT from 24 to 36 hpf. (B) The expression pattern of cmyb in the control, ncor2 morphants, fos-overexpressed embryos, and vegfd-overexpressed embryos at 36 hpf, which were treated with DMSO or with DAPT from 24 to 36 hpf. (C) qPCR result showing the expression level of runx1 in the control, ncor2 morphants, fos-overexpressed embryos, and vegfd-overexpressed embryos at 36 hpf, which were treated with DMSO or with DAPT from 24 to 36 hpf (mean ± SD, n = 3, *P < .05).

Inhibition of Notch signal can partially rescue the decrease of HSCs in ncor2 morphants and vegfd-overexpressed embryos. (A) The expression pattern of runx1 in the control, ncor2 morphants, fos-overexpressed embryos, and vegfd-overexpressed embryos at 36 hpf, which were treated with DMSO or with a γ-secretase inhibitor DAPT from 24 to 36 hpf. (B) The expression pattern of cmyb in the control, ncor2 morphants, fos-overexpressed embryos, and vegfd-overexpressed embryos at 36 hpf, which were treated with DMSO or with DAPT from 24 to 36 hpf. (C) qPCR result showing the expression level of runx1 in the control, ncor2 morphants, fos-overexpressed embryos, and vegfd-overexpressed embryos at 36 hpf, which were treated with DMSO or with DAPT from 24 to 36 hpf (mean ± SD, n = 3, *P < .05).

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