Figure 5
Figure 5. Granulocytes induced by Am80 coexpress CD66 and CD18 surface markers at a higher level than do those induced by G-CSF. (A-D) Flow cytometric analyses of G-CSF– versus Am80-induced granulocytes in coexpression of CD66a-CD18 and CD66a-CD11b surface markers (*GINs versus AINs. CD66a-CD18, P < .003; CD66a-CD11b, P < .028). (E-H) Increased induction of both CD66b-CD18 and CD66b-CD11b surface markers in Am80 versus G-CSF–induced granulocytes (*GINs versus AINs; CD66b-CD18, P < .047; CD66b-CD11b, P < .033). Corresponding isotypes and nonstaining (blank cells) served as controls.

Granulocytes induced by Am80 coexpress CD66 and CD18 surface markers at a higher level than do those induced by G-CSF. (A-D) Flow cytometric analyses of G-CSF– versus Am80-induced granulocytes in coexpression of CD66a-CD18 and CD66a-CD11b surface markers (*GINs versus AINs. CD66a-CD18, P < .003; CD66a-CD11b, P < .028). (E-H) Increased induction of both CD66b-CD18 and CD66b-CD11b surface markers in Am80 versus G-CSF–induced granulocytes (*GINs versus AINs; CD66b-CD18, P < .047; CD66b-CD11b, P < .033). Corresponding isotypes and nonstaining (blank cells) served as controls.

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